Activation of human monocytic cells by Treponema pallidum and Borrelia burgdorferi lipoproteins and synthetic lipopeptides proceeds via a pathway distinct from that of lipopolysaccharide but involves the transcriptional activator NF-kappa B.
about
Immune evasion and recognition of the syphilis spirochete in blood and skin of secondary syphilis patients: two immunologically distinct compartmentsCytokines and chemokines at the crossroads of neuroinflammation, neurodegeneration, and neuropathic painThe apoptotic signaling pathway activated by Toll-like receptor-2Syphilis: review with emphasis on clinical, epidemiologic, and some biologic featuresThe pathogenesis of lyme neuroborreliosis: from infection to inflammationp38 mitogen-activated protein kinase controls NF-kappaB transcriptional activation and tumor necrosis factor alpha production through RelA phosphorylation mediated by mitogen- and stress-activated protein kinase 1 in response to Borrelia burgdorferiLipoproteins of Gram-Positive Bacteria: Key Players in the Immune Response and VirulenceMicroglia are mediators of Borrelia burgdorferi-induced apoptosis in SH-SY5Y neuronal cellsStructural and Biochemical Basis for Polyamine Binding to the Tp0655 Lipoprotein of Treponema pallidum: Putative Role for Tp0655 (TpPotD) as a Polyamine ReceptorImmunostimulation by the synthetic lipopeptide P3CSK4: TLR4-independent activation of the ERK1/2 signal transduction pathway in macrophagesStructure and specific activity of macrophage-stimulating lipopeptides from Mycoplasma hyorhinisActivation of human monocytes by live Borrelia burgdorferi generates TLR2-dependent and -independent responses which include induction of IFN-beta.Borrelia burgdorferi induces the production and release of proinflammatory cytokines in canine synovial explant cultures.Lipoprotein release by bacteria: potential factor in bacterial pathogenesis.Staphylococcus aureus deficient in lipidation of prelipoproteins is attenuated in growth and immune activation.Lipoprotein-dependent and -independent immune responses to spirochetal infection.Induction of pro- and anti-inflammatory cytokines by Borrelia burgdorferi lipoproteins in monocytes is mediated by CD14.Dual role of interleukin-10 in murine Lyme disease: regulation of arthritis severity and host defenseInterleukin-10 modulates proinflammatory cytokines in the human monocytic cell line THP-1 stimulated with Borrelia burgdorferi lipoproteins.Early induction of gamma interferon and interleukin-10 production in draining lymph nodes from mice infected with Borrelia burgdorferi.Dendritic cells phagocytose and are activated by Treponema pallidumAutocrine and exocrine regulation of interleukin-10 production in THP-1 cells stimulated with Borrelia burgdorferi lipoproteins.Biological basis for syphilis.Different patterns of expression and of IL-10 modulation of inflammatory mediators from macrophages of Lyme disease-resistant and -susceptible mice.Murine Lyme arthritis development mediated by p38 mitogen-activated protein kinase activity.Nuclear receptor signaling inhibits HIV-1 replication in macrophages through multiple trans-repression mechanismsThe effects of early syphilis on CD4 counts and HIV-1 RNA viral loads in blood and semenProduction of interleukin-8 (IL-8) by cultured endothelial cells in response to Borrelia burgdorferi occurs independently of secreted [corrected] IL-1 and tumor necrosis factor alpha and is required for subsequent transendothelial migration of neutAnti-Ehrlichia chaffeensis antibody complexed with E. chaffeensis induces potent proinflammatory cytokine mRNA expression in human monocytes through sustained reduction of IkappaB-alpha and activation of NF-kappaB.Spiralin, a mycoplasmal membrane lipoprotein, induces T-cell-independent B-cell blastogenesis and secretion of proinflammatory cytokinesInterleukin-10 alters effector functions of multiple genes induced by Borrelia burgdorferi in macrophages to regulate Lyme disease inflammationCD14 mediates cross talk between mononuclear cells and fibroblasts for upregulation of matrix metalloproteinase 9 by Borrelia burgdorferi.Treponema pallidum elicits innate and adaptive cellular immune responses in skin and blood during secondary syphilis: a flow-cytometric analysis.Viable Borrelia burgdorferi enhances interleukin-10 production and suppresses activation of murine macrophages.Persistence of Borrelia burgdorferi following antibiotic treatment in miceGene expression profiling provides insights into the pathways involved in inflammatory arthritis development: murine model of Lyme disease.The antibiotics doxycycline and minocycline inhibit the inflammatory responses to the Lyme disease spirochete Borrelia burgdorferi.Human TLR8 is activated upon recognition of Borrelia burgdorferi RNA in the phagosome of human monocytesSpirochetal Lipoproteins and Immune Evasion.The Cross-Talk between Spirochetal Lipoproteins and Immunity.
P2860
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P2860
Activation of human monocytic cells by Treponema pallidum and Borrelia burgdorferi lipoproteins and synthetic lipopeptides proceeds via a pathway distinct from that of lipopolysaccharide but involves the transcriptional activator NF-kappa B.
description
1996 nî lūn-bûn
@nan
1996 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
1996 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
name
Activation of human monocytic ...... iptional activator NF-kappa B.
@ast
Activation of human monocytic ...... iptional activator NF-kappa B.
@en
type
label
Activation of human monocytic ...... iptional activator NF-kappa B.
@ast
Activation of human monocytic ...... iptional activator NF-kappa B.
@en
prefLabel
Activation of human monocytic ...... iptional activator NF-kappa B.
@ast
Activation of human monocytic ...... iptional activator NF-kappa B.
@en
P2093
P2860
P1476
Activation of human monocytic ...... iptional activator NF-kappa B.
@en
P2093
D A Harrich
J D Radolf
L L Curetty
M V Norgard
P2860
P304
P407
P577
1996-09-01T00:00:00Z