Teriparatide as a chondroregenerative therapy for injury-induced osteoarthritis. - Wikidata - wikimedia - TriplyDB

Teriparatide as a chondroregenerative therapy for injury-induced osteoarthritis.

Teriparatide as a chondroregenerative therapy for injury-induced osteoarthritis.

http://www.wikidata.org/entity/Q35525309

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Current research on pharmacologic and regenerative therapies for osteoarthritis Wolff's law in action: a mechanism for early knee osteoarthritis Articular Cartilage: Structural and Developmental Intricacies and Questions RBP-Jκ-dependent Notch signaling is required for murine articular cartilage and joint maintenance.Endogenous adenosine maintains cartilage homeostasis and exogenous adenosine inhibits osteoarthritis progression.Targeting TGFβ signaling in subchondral bone and articular cartilage homeostasis PTH [1-34] induced differentiation and mineralization of mandibular condylar cartilage Distribution and alteration of lymphatic vessels in knee joints of normal and osteoarthritic mice.The identification of CD163 expressing phagocytic chondrocytes in joint cartilage and its novel scavenger role in cartilage degradation.Daily oral consumption of hydrolyzed type 1 collagen is chondroprotective and anti-inflammatory in murine posttraumatic osteoarthritis.Parathyroid hormone 1-34 enhances extracellular matrix deposition and organization during flexor tendon repair.Runx1 is critical for PTH-induced onset of mesenchymal progenitor cell chondrogenic differentiation.Synovial joints: from development to homeostasis.A high-throughput model of post-traumatic osteoarthritis using engineered cartilage tissue analogs Subchondral insufficiency fracture of the knee: a non-traumatic injury with prolonged recovery time Emerging targets in osteoarthritis therapy.Orthopedic tissue regeneration: cells, scaffolds, and small molecules.Teriparatide - Indications beyond osteoporosis.Chondrogenesis, chondrocyte differentiation, and articular cartilage metabolism in health and osteoarthritis.Effect of antiresorptive and anabolic bone therapy on development of osteoarthritis in a posttraumatic rat model of OA Do mouse models reflect the diversity of osteoarthritis in humans?Impact of proteoglycan-4 and parathyroid hormone on articular cartilage.Use of a whole-slide imaging system to assess the presence and alteration of lymphatic vessels in joint sections of arthritic mice.Suppressive Effects of Insulin on Tumor Necrosis Factor-Dependent Early Osteoarthritic Changes Associated With Obesity and Type 2 Diabetes Mellitus.Mechanical motion promotes expression of Prg4 in articular cartilage via multiple CREB-dependent, fluid flow shear stress-induced signaling pathways.Osteoarthritis and osteoporosis: what is the overlap?New and emerging treatments for osteoarthritis management: will the dream come true with personalized medicine?The coupling of bone and cartilage turnover in osteoarthritis: opportunities for bone antiresorptives and anabolics as potential treatments?Using mouse models to investigate the pathophysiology, treatment, and prevention of post-traumatic osteoarthritis.Building and maintaining joints by exquisite local control of cell fate.Articular cartilage endurance and resistance to osteoarthritic changes require transcription factor Erg.A homeostatic function of CXCR2 signalling in articular cartilage.Wnt/β-catenin Signaling in Osteoarthritis and in Other Forms of Arthritis.Effect of parathyroid hormone on early chondrogenic differentiation from mesenchymal stem cells.PPARγ/mTOR signalling: striking the right balance in cartilage homeostasis.The association between parathyroid hormone 1‑34 and the Wnt/β‑catenin signaling pathway in a rat model of osteoarthritis.Reduction in disease progression by inhibition of transforming growth factor α-CCL2 signaling in experimental posttraumatic osteoarthritis.Subchondral bone fragility with meniscal tear accelerates and parathyroid hormone decelerates articular cartilage degeneration in rat osteoarthritis model.Loading-Induced Reduction in Sclerostin as a Mechanism of Subchondral Bone Plate Sclerosis in Mouse Knee Joints During Late-Stage Osteoarthritis.Transforming growth factor-β in stem cells and tissue homeostasis.

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Teriparatide as a chondroregenerative therapy for injury-induced osteoarthritis.

http://www.wikidata.org/entity/Q35525309

description

2011 nî lūn-bûn

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2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Teriparatide as a chondroregenerative therapy for injury-induced osteoarthritis.

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Teriparatide as a chondroregenerative therapy for injury-induced osteoarthritis.

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Teriparatide as a chondroregenerative therapy for injury-induced osteoarthritis.

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Teriparatide as a chondroregenerative therapy for injury-induced osteoarthritis.

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Teriparatide as a chondroregenerative therapy for injury-induced osteoarthritis.

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Teriparatide as a chondroregenerative therapy for injury-induced osteoarthritis.

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SCITRANSLMED.3002214

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Science Translational Medicine

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Teriparatide as a chondroregenerative therapy for injury-induced osteoarthritis

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Di Chen

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Edward M Schwarz

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Erik R Sampson

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J Edward Puzas

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Randy N Rosier

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Regis J O'Keefe

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Robert A Mooney

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Susan V Bukata

http://www.w3.org/2001/XMLSchema#string

Ye Tian

http://www.w3.org/2001/XMLSchema#string

P2860

The secreted glycoprotein lubricin protects cartilage surfaces and inhibits synovial cell overgrowth

Genetic manipulation of hedgehog signaling in the endochondral skeleton reveals a direct role in the regulation of chondrocyte proliferation

Knee osteoarthritis after meniscectomy: prevalence of radiographic changes after twenty-one years, compared with matched controls

PTH/PTHrP receptor in early development and Indian hedgehog-regulated bone growth

Lethal skeletal dysplasia from targeted disruption of the parathyroid hormone-related peptide gene

Enhanced chondrogenesis and Wnt signaling in PTH-treated fractures.

Lack of bone neoplasms and persistence of bone efficacy in cynomolgus macaques after long-term treatment with teriparatide [rhPTH(1-34)].

OARSI recommendations for the management of hip and knee osteoarthritis: part III: Changes in evidence following systematic cumulative update of research published through January 2009.

Matrix metalloproteinase 13-deficient mice are resistant to osteoarthritic cartilage erosion but not chondrocyte hypertrophy or osteophyte development.

Targeted expression of constitutively active receptors for parathyroid hormone and parathyroid hormone-related peptide delays endochondral bone formation and rescues mice that lack parathyroid hormone-related peptide.

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Matthew J Hilton

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