Coordinate loss of a microRNA and protein-coding gene cooperate in the pathogenesis of 5q- syndrome. - Wikidata - wikimedia - TriplyDB

Coordinate loss of a microRNA and protein-coding gene cooperate in the pathogenesis of 5q- syndrome.

Coordinate loss of a microRNA and protein-coding gene cooperate in the pathogenesis of 5q- syndrome.

http://www.wikidata.org/entity/Q35525418

about

Role of microRNA-146a in normal and malignant hematopoietic stem cell function PP2A: The Achilles Heal in MDS with 5q Deletion Advances in megakaryocytopoiesis and thrombopoiesis: from bench to bedside.The genetics of myelodysplastic syndrome: from clonal haematopoiesis to secondary leukaemia Aberrant overexpression of CD14 on granulocytes sensitizes the innate immune response in mDia1 heterozygous del(5q) MDS.Current therapy of myelodysplastic syndromes Platelets and cancer: a casual or causal relationship: revisited.Role of casein kinase 1A1 in the biology and targeted therapy of del(5q) MDS.Genetic deletions in AML and MDS.Overexpression of the toll-like receptor (TLR) signaling adaptor MYD88, but lack of genetic mutation, in myelodysplastic syndromes.MicroRNA expression differences in human hematopoietic cell lineages enable regulated transgene expression.Small RNA Sequencing Uncovers New miRNAs and moRNAs Differentially Expressed in Normal and Primary Myelofibrosis CD34+ Cells.Long non-coding RNAs and cancer: a new frontier of translational research?Alterations in the ribosomal machinery in cancer and hematologic disorders.The novel mechanism of lenalidomide activity.Important genes in the pathogenesis of 5q- syndrome and their connection with ribosomal stress and the innate immune system pathway.Molecular pathophysiology of myelodysplastic syndromes.α-1,6-Fucosyltransferase (FUT8) inhibits hemoglobin production during differentiation of murine and K562 human erythroleukemia cells.MicroRNA deregulation in polycythemia vera and essential thrombocythemia patients.MicroRNAs in myeloproliferative neoplasms.miR-99a/100~125b tricistrons regulate hematopoietic stem and progenitor cell homeostasis by shifting the balance between TGFβ and Wnt signaling.Molecular dissection of the 5q deletion in myelodysplastic syndrome.MicroRNAs in erythroid and megakaryocytic differentiation and megakaryocyte-erythroid progenitor lineage commitment Myelodysplastic syndrome hematopoietic stem cell.The genetic basis of phenotypic heterogeneity in myelodysplastic syndromes.Thalidomide-analogue biology: immunological, molecular and epigenetic targets in cancer therapy.Molecular mechanism of microRNA involvement in genesis of myelodysplastic syndrome and its transformation to acute myeloid leukemia.MicroRNAs in platelet production and activation.Human diseases caused by germline and somatic abnormalities in microRNA and microRNA-related genes.Myelodysplastic syndromes with 5q deletion: pathophysiology and role of lenalidomide.The molecular pathogenesis of the myelodysplastic syndromes.Recent Advances in the 5q- Syndrome.Therapy-related myeloid neoplasms: when genetics and environment collide.Deregulated microRNA expression and its pathogenetic implications for myelodysplastic syndromes.MicroRNAs in Control of Stem Cells in Normal and Malignant Hematopoiesis.Specific Depletion of Leukemic Stem Cells: Can MicroRNAs Make the Difference?Correlation of clinical response and response duration with miR-145 induction by lenalidomide in CD34(+) cells from patients with del(5q) myelodysplastic syndrome.Biological activity of lenalidomide in myelodysplastic syndromes with del5q: results of gene expression profiling from a multicenter phase II study.Transcription factors Fli1 and EKLF in the differentiation of megakaryocytic and erythroid progenitor in 5q- syndrome and in Diamond-Blackfan anemia.The role of p53 in myelodysplastic syndromes and acute myeloid leukemia: molecular aspects and clinical implications.

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P2860

Coordinate loss of a microRNA and protein-coding gene cooperate in the pathogenesis of 5q- syndrome.

http://www.wikidata.org/entity/Q35525418

description

2011 nî lūn-bûn

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2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի օգոստոսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

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2011年論文

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2011年论文

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name

Coordinate loss of a microRNA ...... pathogenesis of 5q- syndrome.

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Coordinate loss of a microRNA ...... pathogenesis of 5q- syndrome.

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type

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Coordinate loss of a microRNA ...... pathogenesis of 5q- syndrome.

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Coordinate loss of a microRNA ...... pathogenesis of 5q- syndrome.

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prefLabel

Coordinate loss of a microRNA ...... pathogenesis of 5q- syndrome.

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Coordinate loss of a microRNA ...... pathogenesis of 5q- syndrome.

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Coordinate loss of a microRNA ...... pathogenesis of 5q- syndrome.

@en

P2093

Ann Mullally

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Anupama Narla

http://www.w3.org/2001/XMLSchema#string

Atsushi Nonami

http://www.w3.org/2001/XMLSchema#string

Azra Raza

http://www.w3.org/2001/XMLSchema#string

Benjamin L Ebert

http://www.w3.org/2001/XMLSchema#string

Brian Ball

http://www.w3.org/2001/XMLSchema#string

D Gary Gilliland

http://www.w3.org/2001/XMLSchema#string

Eyal Attar

http://www.w3.org/2001/XMLSchema#string

J Randall McAuley

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Jeffrey L Kutok

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P2860

Discovery of microvascular miRNAs using public gene expression data: miR-145 is expressed in pericytes and is a regulator of Fli1

Identification of RPS14 as a 5q- syndrome gene by RNA interference screen

miR-145 and miR-143 regulate smooth muscle cell fate and plasticity

MicroRNA-145 regulates OCT4, SOX2, and KLF4 and represses pluripotency in human embryonic stem cells

FLI1 monoallelic expression combined with its hemizygous loss underlies Paris-Trousseau/Jacobsen thrombopenia

p53 represses c-Myc through induction of the tumor suppressor miR-145

Targeted deletion reveals essential and overlapping functions of the miR-17 through 92 family of miRNA clusters

MicroRNAs miR-143 and miR-145 modulate cytoskeletal dynamics and responsiveness of smooth muscle cells to injury

The impact of microRNAs on protein output

Most mammalian mRNAs are conserved targets of microRNAs

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