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Type III protein secretion systems in bacterial pathogens of animals and plantsThe Salmonella invasin SipB induces macrophage apoptosis by binding to caspase-1Pseudomonas aeruginosa induces type-III-secretion-mediated apoptosis of macrophages and epithelial cells.Tripeptidyl peptidase II promotes maturation of caspase-1 in Shigella flexneri-induced macrophage apoptosisVirulence factors associated with pediatric shigellosis in Brazilian Amazon.Intracellular Staphylococcus aureus escapes the endosome and induces apoptosis in epithelial cells.Role of adenylate cyclase-hemolysin in alveolar macrophage apoptosis during Bordetella pertussis infection in vivo.The pathogenesis of Shigella flexneri infection: lessons from in vitro and in vivo studies.Streptococcal pyrogenic exotoxin B induces apoptosis and reduces phagocytic activity in U937 cells.Effect of Chlamydia trachomatis infection and subsequent tumor necrosis factor alpha secretion on apoptosis in the murine genital tractProinflammatory and proapoptotic activities associated with Bordetella pertussis filamentous hemagglutininApoptotic signaling pathway activated by Helicobacter pylori infection and increase of apoptosis-inducing activity under serum-starved conditions.Decreased apoptosis in the ileum and ileal Peyer's patches: a feature after infection with rabbit enteropathogenic Escherichia coli O103.Salmonella-induced cell death is not required for enteritis in calves.Roles of Vibrio fischeri and nonsymbiotic bacteria in the dynamics of mucus secretion during symbiont colonization of the Euprymna scolopes light organ.Antigen-specific CD8(+) T cells fail to respond to Shigella flexneri.Analysis of virulence and inflammatory potential of Shigella flexneri purine biosynthesis mutantsThe interleukin 1beta-converting enzyme, caspase 1, is activated during Shigella flexneri-induced apoptosis in human monocyte-derived macrophagesSalmonella exploits caspase-1 to colonize Peyer's patches in a murine typhoid modelPreventing acute gut wall damage in infectious diarrhoeas with glycosylated dendrimers.Molecular pathogenesis of Shigella spp.: controlling host cell signaling, invasion, and death by type III secretion.Impact of simulated microgravity on the normal developmental time line of an animal-bacteria symbiosis.Perspectives series: host/pathogen interactions. Apoptosis in bacterial pathogenesisSpa15 of Shigella flexneri is secreted through the type III secretion system and prevents staurosporine-induced apoptosis.Streptococcal pyrogenic exotoxin B-induced apoptosis in a549 cells is mediated by a receptor- and mitochondrion-dependent pathway.Macrophage Apoptosis Triggered by IpaD from Shigella flexneri.Influence of the Paracoccidioides brasiliensis 14-3-3 and gp43 proteins on the induction of apoptosis in A549 epithelial cells.Interleukin-8 controls bacterial transepithelial translocation at the cost of epithelial destruction in experimental shigellosis.Thymic independence of adaptive immunity to the intracellular pathogen Shigella flexneri serotype 2aIncreased interleukin-1 (IL-1) and imbalance between IL-1 and IL-1 receptor antagonist during acute inflammation in experimental Shigellosis.Induction of necrosis in human neutrophils by Shigella flexneri requires type III secretion, IpaB and IpaC invasins, and actin polymerizationEnterohemorrhagic Escherichia coli induces apoptosis which augments bacterial binding and phosphatidylethanolamine exposure on the plasma membrane outer leaflet.Shigella flexneri IpaH(7.8) facilitates escape of virulent bacteria from the endocytic vacuoles of mouse and human macrophages.Release of Toll-like receptor-2-activating bacterial lipoproteins in Shigella flexneri culture supernatantsInduction of epithelial cell death including apoptosis by enteropathogenic Escherichia coli expressing bundle-forming pili.Expression of the virulence plasmid-carried apyrase gene (apy) of enteroinvasive Escherichia coli and Shigella flexneri is under the control of H-NS and the VirF and VirB regulatory cascade.Host cell death due to enteropathogenic Escherichia coli has features of apoptosis.Apoptosis in acute shigellosis is associated with increased production of Fas/Fas ligand, perforin, caspase-1, and caspase-3 but reduced production of Bcl-2 and interleukin-2.Human monocytes kill Shigella flexneri but then die by apoptosis associated with suppression of proinflammatory cytokine production.Lactoferrin protects rabbits from Shigella flexneri-induced inflammatory enteritis.
P2860
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P2860
description
1996 nî lūn-bûn
@nan
1996 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1996 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
name
In vivo apoptosis in Shigella flexneri infections
@ast
In vivo apoptosis in Shigella flexneri infections
@en
type
label
In vivo apoptosis in Shigella flexneri infections
@ast
In vivo apoptosis in Shigella flexneri infections
@en
prefLabel
In vivo apoptosis in Shigella flexneri infections
@ast
In vivo apoptosis in Shigella flexneri infections
@en
P2093
P2860
P1476
In vivo apoptosis in Shigella flexneri infections
@en
P2093
A O Aliprantis
A Zychlinsky
J R Cantey
K Thirumalai
P J Sansonetti
P2860
P304
P407
P577
1996-12-01T00:00:00Z