Acute inflammation causes epithelial invasion and mucosal destruction in experimental shigellosis - Wikidata - wikimedia - TriplyDB

Acute inflammation causes epithelial invasion and mucosal destruction in experimental shigellosis

Acute inflammation causes epithelial invasion and mucosal destruction in experimental shigellosis

http://www.wikidata.org/entity/Q36363758

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Type III protein secretion systems in bacterial pathogens of animals and plants p62 and NDP52 proteins target intracytosolic Shigella and Listeria to different autophagy pathways Common themes in microbial pathogenicity revisited Cellular Aspects of Shigella Pathogenesis: Focus on the Manipulation of Host Cell Processes Shigella IpaH Family Effectors as a Versatile Model for Studying Pathogenic Bacteria The zebrafish as a new model for the in vivo study of Shigella flexneri interaction with phagocytes and bacterial autophagy Neutrophil elastase targets virulence factors of enterobacteria A bacterial invasin induces macrophage apoptosis by binding directly to ICE Shigella impairs T lymphocyte dynamics in vivo.Shigella reroutes host cell central metabolism to obtain high-flux nutrient supply for vigorous intracellular growth.Lawsonia intracellularis infection in the large intestines of pigs.Characterisation of early mucosal and neuronal lesions following Shigella flexneri infection in human colon Comprehensive study of the intestinal stage of listeriosis in a rat ligated ileal loop system.Interactions of the invasive pathogens Salmonella typhimurium, Listeria monocytogenes, and Shigella flexneri with M cells and murine Peyer's patches.Vaccination against shigellosis with attenuated Shigella flexneri 2a strain SC602 A role for IL-1 alpha in inducing pathologic inflammation during bacterial infection.The pathogenesis of Shigella flexneri infection: lessons from in vitro and in vivo studies.Structure-function analysis of the Shigella virulence factor IpaB.Parameters underlying successful protection with live attenuated mutants in experimental shigellosis Mycobacterium avium invades the intestinal mucosa primarily by interacting with enterocytes The Shigella flexneri effector OspG interferes with innate immune responses by targeting ubiquitin-conjugating enzymes Unsolved mysteries of intestinal M cells.Up-regulation of MUC2 and IL-1β expression in human colonic epithelial cells by Shigella and its interaction with mucins.Microbes and microbial toxins: paradigms for microbial-mucosal interactions III. Shigellosis: from symptoms to molecular pathogenesis.Quantitative RT-PCR profiling of the rabbit immune response: assessment of acute Shigella flexneri infection.Recent advances in understanding enteric pathogenic Escherichia coli.Local entrapment of interferon gamma in the recovery from Shigella dysenteriae type 1 infection.The regulatory protein PhoP controls susceptibility to the host inflammatory response in Shigella flexneri.Host-pathogen interactions: the seduction of molecular cross talk.Toll-like receptor 4 contributes to colitis development but not to host defense during Citrobacter rodentium infection in mice.RNA-seq analysis of the influence of anaerobiosis and FNR on Shigella flexneri.Peripheral blood neutrophil responses in children with shigellosis.Down-regulation of gamma interferon, tumor necrosis factor type I, interleukin 1 (IL-1) type I, IL-3, IL-4, and transforming growth factor beta type I receptors at the local site during the acute phase of Shigella infection Induction of adherence and degranulation of polymorphonuclear leukocytes: a new expression of the invasive phenotype of Shigella flexneri Induction of a local anti-IpaC antibody response in mice by use of a Shigella flexneri 2a vaccine candidate: implications for use of IpaC as a protein carrier.Infection of rabbit Peyer's patches by Shigella flexneri: effect of adhesive or invasive bacterial phenotypes on follicle-associated epithelium In vivo apoptosis in Shigella flexneri infections Shigella flexneri is trapped in polymorphonuclear leukocyte vacuoles and efficiently killed Human monocyte-derived macrophages infected with virulent Shigella flexneri in vitro undergo a rapid cytolytic event similar to oncosis but not apoptosis The interleukin 1beta-converting enzyme, caspase 1, is activated during Shigella flexneri-induced apoptosis in human monocyte-derived macrophages

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Acute inflammation causes epithelial invasion and mucosal destruction in experimental shigellosis

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1994 nî lūn-bûn

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1994年学术文章

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1994年学术文章

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1994年学术文章

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1994年学术文章

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1994年学术文章

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1994年學術文章

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1994年學術文章

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Acute inflammation causes epit ...... on in experimental shigellosis

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Acute inflammation causes epit ...... on in experimental shigellosis

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Cavaillon JM

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Gounon P

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Huerre M

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Ohayon H

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Perdomo OJ

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Sansonetti PJ

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P2860

Shigella flexneri enters human colonic Caco-2 epithelial cells through the basolateral pole

Death in shigellosis: incidence and risk factors in hospitalized patients.

Interleukin 1 induces a shock-like state in rabbits. Synergism with tumor necrosis factor and the effect of cyclooxygenase inhibition

Identification of icsA, a plasmid locus of Shigella flexneri that governs bacterial intra- and intercellular spread through interaction with F-actin.

Polymorphonuclear leukocyte transmigration promotes invasion of colonic epithelial monolayer by Shigella flexneri

Interleukin 1 is released by murine macrophages during apoptosis induced by Shigella flexneri.

Neutrophil migration across a cultured intestinal epithelium. Dependence on a CD11b/CD18-mediated event and enhanced efficiency in physiological direction

In vivo biologic and immunohistochemical analysis of interleukin-1 alpha, beta and tumor necrosis factor during experimental endotoxemia. Kinetics, Kupffer cell expression, and glucocorticoid effects.

Penetration of Salmonella through a polarized Madin-Darby canine kidney epithelial cell monolayer

Neutrophil migration across cultured intestinal epithelial monolayers is modulated by epithelial exposure to IFN-gamma in a highly polarized fashion

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1307-1319

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7931064

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