Proteasome malfunction activates macroautophagy in the heart.
about
New insights into the role of mitochondrial dynamics and autophagy during oxidative stress and aging in the heartp62 Stages an interplay between the ubiquitin-proteasome system and autophagy in the heart of defense against proteotoxic stressThe ubiquitin-proteasome system and cardiovascular diseaseAutophagy and cardiovascular aging: lesson learned from rapamycin.The ubiquitin proteasome system and myocardial ischemiaProteasomal and lysosomal protein degradation and heart disease.Autophagic-lysosomal inhibition compromises ubiquitin-proteasome system performance in a p62 dependent manner in cardiomyocytesThe ubiquitin proteasome system in human cardiomyopathies and heart failure.Heat shock response and autophagy--cooperation and control.The interplay between autophagy and the ubiquitin-proteasome system in cardiac proteotoxicitybFGF regulates autophagy and ubiquitinated protein accumulation induced by myocardial ischemia/reperfusion via the activation of the PI3K/Akt/mTOR pathway.Proteasome functional insufficiency in cardiac pathogenesis.Partial inhibition of the ubiquitin-proteasome system ameliorates cardiac dysfunction following ischemia-reperfusion in the presence of high glucoseBreaking down protein degradation mechanisms in cardiac muscle.Primary proteasome inhibition results in cardiac dysfunctionNerve growth factor-induced Akt/mTOR activation protects the ischemic heart via restoring autophagic flux and attenuating ubiquitinated protein accumulation.Selective autophagy: talking with the UPS.Autophagy and ubiquitination in cardiovascular diseases.Proteotoxicity and cardiac dysfunction.Nuclear factor erythroid-2 related factor 2 (Nrf2)-mediated protein quality control in cardiomyocytes.Autophagy modulation: a potential therapeutic approach in cardiac hypertrophy.The GST-BHMT assay reveals a distinct mechanism underlying proteasome inhibition-induced macroautophagy in mammalian cells.Tau phosphorylation affects its axonal transport and degradation.Systemic inhibition of neddylation by 3-day MLN4924 treatment regime does not impair autophagic flux in mouse hearts and brains.
P2860
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P2860
Proteasome malfunction activates macroautophagy in the heart.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Proteasome malfunction activates macroautophagy in the heart.
@ast
Proteasome malfunction activates macroautophagy in the heart.
@en
type
label
Proteasome malfunction activates macroautophagy in the heart.
@ast
Proteasome malfunction activates macroautophagy in the heart.
@en
prefLabel
Proteasome malfunction activates macroautophagy in the heart.
@ast
Proteasome malfunction activates macroautophagy in the heart.
@en
P2093
P2860
P1476
Proteasome malfunction activates macroautophagy in the heart.
@en
P2093
Qingwen Zheng
Xuejun Wang
Zongwen Tian
P2860
P304
P577
2011-07-28T00:00:00Z