Hepatitis C virus induces interferon-λ and interferon-stimulated genes in primary liver cultures. - Wikidata - wikimedia - TriplyDB

Hepatitis C virus induces interferon-λ and interferon-stimulated genes in primary liver cultures.

Hepatitis C virus induces interferon-λ and interferon-stimulated genes in primary liver cultures.

http://www.wikidata.org/entity/Q35560734

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IFNL3 mRNA structure is remodeled by a functional non-coding polymorphism associated with hepatitis C virus clearance A variant upstream of IFNL3 (IL28B) creating a new interferon gene IFNL4 is associated with impaired clearance of hepatitis C virus Interferon (IFN) and Cellular Immune Response Evoked in RNA-Pattern Sensing During Infection with Hepatitis C Virus (HCV)Interferon Response in Hepatitis C Virus (HCV) Infection: Lessons from Cell Culture Systems of HCV Infection Animal models for the study of HCV A New Twist to a Chronic HCV Infection: Occult Hepatitis C Interferon-λ: Immune Functions at Barrier Surfaces and Beyond Regulation of hepatic innate immunity by hepatitis C virus Innate immune responses in hepatitis C virus infection Leukocyte-derived IFN-α/β and epithelial IFN-λ constitute a compartmentalized mucosal defense system that restricts enteric virus infections Targeted induction of interferon-λ in humanized chimeric mouse liver abrogates hepatotropic virus infection (-)-Epigallocatechin-3-Gallate Enhances Hepatitis C Virus Double-Stranded RNA Intermediates-Triggered Innate Immune Responses in Hepatocytes.Hepatitis E virus persists in the presence of a type III interferon response.Transcriptional regulation of IFN-λ genes in hepatitis C virus-infected hepatocytes via IRF-3·IRF-7·NF-κB complex Multiple interferon stimulated genes synergize with the zinc finger antiviral protein to mediate anti-alphavirus activity.Protective immunity against hepatitis C: many shades of gray.Interferon lambda alleles predict innate antiviral immune responses and hepatitis C virus permissiveness Diverse intracellular pathogens activate type III interferon expression from peroxisomes An interferon response gene signature is associated with the therapeutic response of hepatitis C patients IFN-λ exerts opposing effects on T cell responses depending on the chronicity of the virus infection.Visualizing hepatitis C virus infection in humanized mice.Immune control and failure in HCV infection--tipping the balance.Productive hepatitis C virus infection of stem cell-derived hepatocytes reveals a critical transition to viral permissiveness during differentiation.IL-29 is the dominant type III interferon produced by hepatocytes during acute hepatitis C virus infection Barriers of hepatitis C virus interspecies transmission Interferon lambda: opportunities, risks, and uncertainties in the fight against HCV.Type III interferons, IL-28 and IL-29, are increased in chronic HCV infection and induce myeloid dendritic cell-mediated FoxP3+ regulatory T cells Dendritic cells in hepatitis C virus infection: key players in the IFNL3-genotype response.Albumin fusion of interleukin-28B: production and characterization of its biological activities and protein stability.Permissivity of primary human hepatocytes and different hepatoma cell lines to cell culture adapted hepatitis C virus.Plasma interferon-gamma-inducible protein-10 levels are associated with early, but not sustained virological response during treatment of acute or early chronic HCV infection Suppression of interferon lambda signaling by SOCS-1 results in their excessive production during influenza virus infection IFNL4 ss469415590 variant shows similar performance to rs12979860 as predictor of response to treatment against Hepatitis C Virus genotype 1 or 4 in Caucasians Identification, molecular cloning, and analysis of full-length hepatitis C virus transmitted/founder genotypes 1, 3, and 4 Control of hepatitis C virus replication in mouse liver-derived cells by MAVS-dependent production of type I and type III interferons.Impairment of type I but not type III IFN signaling by hepatitis C virus infection influences antiviral responses in primary human hepatocytes Hepatitis C Virus. Strategies to Evade Antiviral Responses Activation and evasion of antiviral innate immunity by hepatitis C virus.IFN-λ: A New Class of Interferon with Distinct Functions-Implications for Hepatitis C Virus Research.Infection of Human Liver Myofibroblasts by Hepatitis C Virus: A Direct Mechanism of Liver Fibrosis in Hepatitis C

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Hepatitis C virus induces interferon-λ and interferon-stimulated genes in primary liver cultures.

http://www.wikidata.org/entity/Q35560734

description

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Alexander Ploss

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Charles M Rice

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Christopher T Jones

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Edgar D Charles

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Linda Andrus

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Svetlana Marukian

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Timothy P Sheahan

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P2860

Hepatitis C virus protease NS3/4A cleaves mitochondrial antiviral signaling protein off the mitochondria to evade innate immunity

Cardif is an adaptor protein in the RIG-I antiviral pathway and is targeted by hepatitis C virus

Viral and therapeutic control of IFN-beta promoter stimulator 1 during hepatitis C virus infection

Regulating intracellular antiviral defense and permissiveness to hepatitis C virus RNA replication through a cellular RNA helicase, RIG-I

Genetic variation in IL28B and spontaneous clearance of hepatitis C virus

A diverse range of gene products are effectors of the type I interferon antiviral response

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Lambda interferon inhibits hepatitis B and C virus replication.

DNA microarray analysis of chimpanzee liver during acute resolving hepatitis C virus infection.

Robust hepatitis C virus infection in vitro.

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1913-1923

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10.1002/HEP.24580

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6

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54

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21800339

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3219820

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