A critical role for macrophages in promotion of urethane-induced lung carcinogenesis - Wikidata - wikimedia - TriplyDB

A critical role for macrophages in promotion of urethane-induced lung carcinogenesis

A critical role for macrophages in promotion of urethane-induced lung carcinogenesis

http://www.wikidata.org/entity/Q35566757

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Lung tumor promotion by chromium-containing welding particulate matter in a mouse model Spatial, Temporal, and Functional Aspects of Macrophages during "The Good, the Bad, and the Ugly" Phases of Inflammation Functional Relationship between Tumor-Associated Macrophages and Macrophage Colony-Stimulating Factor as Contributors to Cancer Progression Mechanisms driving macrophage diversity and specialization in distinct tumor microenvironments and parallelisms with other tissues Effect of angiotensin II type 2 receptor-interacting protein on adipose tissue function via modulation of macrophage polarization Macrophage plasticity and polarization: in vivo veritas Tumor cell-activated CARD9 signaling contributes to metastasis-associated macrophage polarization.M2 polarization of macrophages facilitates arsenic-induced cell transformation of lung epithelial cells Innate immune regulation by STAT-mediated transcriptional mechanisms.Tissue-infiltrating neutrophils constitute the major in vivo source of angiogenesis-inducing MMP-9 in the tumor microenvironment Depletion of tumor-associated macrophages slows the growth of chemically induced mouse lung adenocarcinomas Epithelial β1 integrin is required for lung branching morphogenesis and alveolarization.Sweets for a bitter end: lung cancer cell-surface protein glycosylation mediates metastatic colonization.Immune and Inflammatory Cell Composition of Human Lung Cancer Stroma.Differential macrophage programming in the tumor microenvironment.Macrophages promote benzopyrene-induced tumor transformation of human bronchial epithelial cells by activation of NF-κB and STAT3 signaling in a bionic airway chip culture and in animal models.Opposite Effects of M1 and M2 Macrophage Subtypes on Lung Cancer Progression.Sodium Valproate Enhances the Urethane-Induced Lung Adenomas and Suppresses Malignization of Adenomas in Ovariectomized Female Mice Deletion of TGF-β signaling in myeloid cells enhances their anti-tumorigenic properties.Microenvironmental effects limit efficacy of thymoquinone treatment in a mouse model of ovarian cancer.Lipopolysaccharide enhances mouse lung tumorigenesis: a model for inflammation-driven lung cancer.Lack of MMP10 exacerbates experimental colitis and promotes development of inflammation-associated colonic dysplasia.Expression Profiling of Macrophages Reveals Multiple Populations with Distinct Biological Roles in an Immunocompetent Orthotopic Model of Lung Cancer The Metabolic Prospective and Redox Regulation of Macrophage Polarization.Chronic NF-κB activation links COPD and lung cancer through generation of an immunosuppressive microenvironment in the lungs.Manipulating the NF-κB pathway in macrophages using mannosylated, siRNA-delivering nanoparticles can induce immunostimulatory and tumor cytotoxic functions.Neutrophil-Derived IL-1β Impairs the Efficacy of NF-κB Inhibitors against Lung Cancer.Murine mesothelioma induces locally-proliferating IL-10(+)TNF-α(+)CD206(-)CX3CR1(+) M3 macrophages that can be selectively depleted by chemotherapy or immunotherapy.Identification of myeloid cell subsets in murine lungs using flow cytometry.Investigating palygorskite's role in the development of mesothelioma in southern Nevada: Insights into fiber-induced carcinogenicity Tumor angiogenesis: MMP-mediated induction of intravasation- and metastasis-sustaining neovasculature.Autocrine HBEGF expression promotes breast cancer intravasation, metastasis and macrophage-independent invasion in vivo.The mechanism of the anticancer function of M1 macrophages and their use in the clinic.Non-small-cell lung cancers: a heterogeneous set of diseases.Tumor-associated macrophages as major players in the tumor microenvironment.Plasmacytoid dendritic cells, a role in neoplastic prevention and progression.Role of pulmonary macrophages in initiation of lung metastasis in anaplastic thyroid cancer.Macrophage and cancer cell cross-talk via CCR2 and CX3CR1 is a fundamental mechanism driving lung cancer.Biocompatible mannosylated endosomal-escape nanoparticles enhance selective delivery of short nucleotide sequences to tumor associated macrophages.Myeloid cell RelA/p65 promotes lung cancer proliferation through Wnt/β-catenin signaling in murine and human tumor cells.

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A critical role for macrophages in promotion of urethane-induced lung carcinogenesis

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A critical role for macrophages in promotion of urethane-induced lung carcinogenesis

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A critical role for macrophages in promotion of urethane-induced lung carcinogenesis

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A critical role for macrophages in promotion of urethane-induced lung carcinogenesis

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A critical role for macrophages in promotion of urethane-induced lung carcinogenesis

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A critical role for macrophages in promotion of urethane-induced lung carcinogenesis

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A critical role for macrophages in promotion of urethane-induced lung carcinogenesis

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Journal of Immunology

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A critical role for macrophages in promotion of urethane-induced lung carcinogenesis

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Allyson G McLoed

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Amber L Degryse

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Fiona E Yull

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Frank B McMahon

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Georgios T Stathopoulos

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Jamie A Ausborn

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Linda A Gleaves

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Rinat Zaynagetdinov

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Taylor P Sherrill

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Timothy S Blackwell

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P2860

The Hallmarks of Cancer

Inflammation and cancer

Cancer statistics, 2010

Alternative activation of macrophages

Lung cancer and chronic obstructive pulmonary disease: needs and opportunities for integrated research

Macrophages: Obligate Partners for Tumor Cell Migration, Invasion, and Metastasis

Macrophage polarization: tumor-associated macrophages as a paradigm for polarized M2 mononuclear phagocytes

Tumour-educated macrophages promote tumour progression and metastasis

Macrophage plasticity and interaction with lymphocyte subsets: cancer as a paradigm

Macrophages, innate immunity and cancer: balance, tolerance, and diversity

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