Inhibition of caspase-1 activation in Gram-negative sepsis and experimental endotoxemia
about
TXNIP deficiency exacerbates endotoxic shock via the induction of excessive nitric oxide synthesisC10X polymorphism in the CARD8 gene is associated with bacteraemia.Revisiting caspases in sepsis.Measurement of monocyte apoptosis, plasma IL-1β and PR3 activity as an approach to evaluate the immunological status in sepsisImmunity, inflammation and sepsis: new insights and persistent questions.Compartmentalized Cytokine Responses in Hidradenitis SuppurativaInflammasome and caspase-1 inhibition caused by Bcl-2 and Bcl-XL may influence cytokine responses of lipopolysaccharide-stimulated peripheral blood mononuclear cells from septic patientsHuman caspase-4 and caspase-5 regulate the one-step non-canonical inflammasome activation in monocytes.Inflammasome priming by lipopolysaccharide is dependent upon ERK signaling and proteasome function.The immune response to severe bacterial infections: consequences for therapy.The interplay between inflammasome activation and antifungal host defense.Inflammasome expression and cytomegalovirus viremia in critically ill patients with sepsis.Sevoflurane combined with ATP activates caspase-1 and triggers caspase-1-dependent pyroptosis in murine J774 macrophages.Systemic CD14 inhibition attenuates organ inflammation in porcine Escherichia coli sepsisIdentification of key genes in Gram‑positive and Gram‑negative sepsis using stochastic perturbation.Change of annexin binding of monocytes as an expression of cellular response to Candida albicans: down-regulation in severe sepsis.Effect of clarithromycin in inflammatory markers of patients with ventilator-associated pneumonia and sepsis caused by Gram-negative bacteria: results from a randomized clinical studyWhat is the role for the inflammasome in burn injury and sepsis?The functional role of natural killer cells early in clinical sepsis.Deficient Candida-specific T-helper 17 response during sepsis.A hot-water extract of Sanguisorba officinalis ameliorates endotoxin-induced septic shock by inhibiting inflammasome activation.Increase of circulating endocan over sepsis follow-up is associated with progression into organ dysfunction.Multiple inflammasomes may regulate the interleukin-1-driven inflammation in protracted bacterial bronchitis.
P2860
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P2860
Inhibition of caspase-1 activation in Gram-negative sepsis and experimental endotoxemia
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Inhibition of caspase-1 activation in Gram-negative sepsis and experimental endotoxemia
@ast
Inhibition of caspase-1 activation in Gram-negative sepsis and experimental endotoxemia
@en
type
label
Inhibition of caspase-1 activation in Gram-negative sepsis and experimental endotoxemia
@ast
Inhibition of caspase-1 activation in Gram-negative sepsis and experimental endotoxemia
@en
prefLabel
Inhibition of caspase-1 activation in Gram-negative sepsis and experimental endotoxemia
@ast
Inhibition of caspase-1 activation in Gram-negative sepsis and experimental endotoxemia
@en
P2093
P2860
P356
P1433
P1476
Inhibition of caspase-1 activation in Gram-negative sepsis and experimental endotoxemia
@en
P2093
Anastasia Antonopoulou
Athina Savva
Frank L van de Veerdonk
Jos W M van der Meer
Leo A B Joosten
Maria Mouktaroudi
Maria Raftogiannis
Marianna Georgitsi
Mihai G Netea
Peter Pickkers
P2860
P2888
P356
10.1186/CC9974
P577
2011-01-18T00:00:00Z
P5875
P6179
1052188521