Inflammasome mRNA expression in human monocytes during early septic shock. - Wikidata - wikimedia - TriplyDB

Inflammasome mRNA expression in human monocytes during early septic shock.

Inflammasome mRNA expression in human monocytes during early septic shock.

http://www.wikidata.org/entity/Q36614655

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Host innate immune responses to sepsis The immunopathology of sepsis: pathogen recognition, systemic inflammation, the compensatory anti-inflammatory response, and regulatory T cells Depletion of the ubiquitin-binding adaptor molecule SQSTM1/p62 from macrophages harboring cftr ΔF508 mutation improves the delivery of Burkholderia cenocepacia to the autophagic machinery Autophagy proteins regulate innate immune responses by inhibiting the release of mitochondrial DNA mediated by the NALP3 inflammasome Confronting the next influenza pandemic with anti-inflammatory and immunomodulatory agents: why they are needed and how they might work.Activation of the pyrin inflammasome by intracellular Burkholderia cenocepacia Monocyte derived microvesicles deliver a cell death message via encapsulated caspase-1.C10X polymorphism in the CARD8 gene is associated with bacteraemia.Apoptosis-associated speck-like protein (ASC) controls Legionella pneumophila infection in human monocytes.Microvesicular caspase-1 mediates lymphocyte apoptosis in sepsis.Receptor interacting protein-2 plays a critical role in human lung epithelial cells survival in response to Fas-induced cell-death.The association of polymorphisms of TLR4 and CD14 genes with susceptibility to sepsis in a Chinese population.Measurement of monocyte apoptosis, plasma IL-1β and PR3 activity as an approach to evaluate the immunological status in sepsis Inhibition of caspase-1 activation in Gram-negative sepsis and experimental endotoxemia Mononuclear Phagocyte-Derived Microparticulate Caspase-1 Induces Pulmonary Vascular Endothelial Cell Injury.Autophagy stimulation by rapamycin suppresses lung inflammation and infection by Burkholderia cenocepacia in a model of cystic fibrosis.Inflammasome-regulated cytokines are critical mediators of acute lung injury Clinical relevance of single nucleotide polymorphisms within the entire NLRP3 gene in patients with major blunt trauma.Innate immunity gene expression changes in critically ill patients with sepsis and disease-related malnutrition.The emerging importance of autophagy in pulmonary diseases.Inflammasome and toll-like receptor signaling in human monocytes after successful cardiopulmonary resuscitation.Sepsis: links between pathogen sensing and organ damage Therapeutic Administration of a Monoclonal Anti-Il-1β Antibody Protects Against Experimental Melioidosis.Pyrin critical to macrophage IL-1beta response to Francisella challenge Inflammasome priming by lipopolysaccharide is dependent upon ERK signaling and proteasome function.Innate immune recognition in infectious and noninfectious diseases of the lung.Update in pediatric lung disease 2009.Monocytes and macrophages regulate immunity through dynamic networks of survival and cell death.Inflammasome gene profile is modulated in septic patients, with a greater magnitude in non-survivors.Inflammasome expression and cytomegalovirus viremia in critically ill patients with sepsis.Expression and regulation of the NALP3 inflammasome complex in periodontal diseases.Immunosuppressive effects of red blood cells on monocytes are related to both storage time and storage solution.Immunohistochemical localization of NALP3 inflammasome in experimental periapical lesions.Nlrp1 inflammasome is downregulated in trauma patients.Reduced NLRP3 Gene Expression Limits the IL-1β Cleavage via Inflammasome in Monocytes from Severely Injured Trauma Patients.Inflammasomes in Tissue Damages and Immune Disorders After Trauma

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Inflammasome mRNA expression in human monocytes during early septic shock.

http://www.wikidata.org/entity/Q36614655

description

2008 nî lūn-bûn

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2008年の論文

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2008年论文

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2008年论文

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name

Inflammasome mRNA expression in human monocytes during early septic shock.

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Inflammasome mRNA expression in human monocytes during early septic shock.

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type

label

Inflammasome mRNA expression in human monocytes during early septic shock.

@ast

Inflammasome mRNA expression in human monocytes during early septic shock.

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prefLabel

Inflammasome mRNA expression in human monocytes during early septic shock.

@ast

Inflammasome mRNA expression in human monocytes during early septic shock.

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RCCM.200703-418OC

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American Journal of Respiratory and Critical Care Medicine

P1476

Inflammasome mRNA expression in human monocytes during early septic shock.

@en

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Anasuya Sarkar

http://www.w3.org/2001/XMLSchema#string

Beth Y Besecker

http://www.w3.org/2001/XMLSchema#string

Haikady N Nagaraja

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Jennifer L Hollyfield

http://www.w3.org/2001/XMLSchema#string

Mark D Wewers

http://www.w3.org/2001/XMLSchema#string

Mark Hall

http://www.w3.org/2001/XMLSchema#string

Matthew C Exline

http://www.w3.org/2001/XMLSchema#string

Michelle D Duncan

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Mikhail A Gavrilin

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Nina L Knatz

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CATERPILLER: a novel gene family important in immunity, cell death, and diseases

The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta

NLRs join TLRs as innate sensors of pathogens

ASC directs NF-kappaB activation by regulating receptor interacting protein-2 (RIP2) caspase-1 interactions

Pathogen recognition and innate immunity

NALPs: a novel protein family involved in inflammation

Enhanced bacterial clearance and sepsis resistance in caspase-12-deficient mice

Caspase-1 regulates Escherichia coli sepsis and splenic B cell apoptosis independently of interleukin-1beta and interleukin-18

Differential modulation of endotoxin responsiveness by human caspase-12 polymorphisms

Inflammatory caspases: linking an intracellular innate immune system to autoinflammatory diseases

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983-988

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10.1164/RCCM.200703-418OC

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9

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177

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2008-02-08T00:00:00Z

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18263805

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2361424

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