Dissecting autoimmune diabetes through genetic manipulation of non-obese diabetic mice.
about
Changes in liver cell DNA methylation status in diabetic mice affect its FT-IR characteristicsPD-1 deficiency reveals various tissue-specific autoimmunity by H-2b and dose-dependent requirement of H-2g7 for diabetes in NOD mice.Genes within the Idd5 and Idd9/11 diabetes susceptibility loci affect the pathogenic activity of B cells in nonobese diabetic mice.Establishment of NOD-Pdcd1-/- mice as an efficient animal model of type I diabetes.Lentivectors encoding immunosuppressive proteins genetically engineer pancreatic beta-cells to correct diabetes in allogeneic mice.The use of animal models in diabetes research.The good turned ugly: immunopathogenic basis for diabetogenic CD8+ T cells in NOD mice.Toll-like receptor engagement converts T-cell autoreactivity into overt autoimmune disease.Innate stimulation of B1a cells enhances the autoreactive IgM repertoire in the NOD mouse: implications for type 1 diabetes.
P2860
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P2860
Dissecting autoimmune diabetes through genetic manipulation of non-obese diabetic mice.
description
2003 nî lūn-bûn
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2003年の論文
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2003年論文
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2003年論文
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2003年論文
@zh-hk
2003年論文
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2003年論文
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2003年论文
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2003年论文
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2003年论文
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name
Dissecting autoimmune diabetes through genetic manipulation of non-obese diabetic mice.
@ast
Dissecting autoimmune diabetes through genetic manipulation of non-obese diabetic mice.
@en
type
label
Dissecting autoimmune diabetes through genetic manipulation of non-obese diabetic mice.
@ast
Dissecting autoimmune diabetes through genetic manipulation of non-obese diabetic mice.
@en
prefLabel
Dissecting autoimmune diabetes through genetic manipulation of non-obese diabetic mice.
@ast
Dissecting autoimmune diabetes through genetic manipulation of non-obese diabetic mice.
@en
P1433
P1476
Dissecting autoimmune diabetes through genetic manipulation of non-obese diabetic mice.
@en
P2093
P2888
P304
P356
10.1007/S00125-003-1218-1
P577
2003-10-28T00:00:00Z