ERK1/2 and p38α/β signaling in tumor cell quiescence: opportunities to control dormant residual disease. - Wikidata - wikimedia - TriplyDB

ERK1/2 and p38α/β signaling in tumor cell quiescence: opportunities to control dormant residual disease.

ERK1/2 and p38α/β signaling in tumor cell quiescence: opportunities to control dormant residual disease.

http://www.wikidata.org/entity/Q35579435

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Tumor dormancy: potential therapeutic target in tumor recurrence and metastasis prevention Glioblastoma Circulating Cells: Reality, Trap or Illusion?Molecular interactions in the development of brain metastases Metastatic mechanisms in follicular cell-derived thyroid cancer Targeting tumor-stromal interactions in bone metastasis Mechanisms of disseminated cancer cell dormancy: an awakening field EMT, CTCs and CSCs in tumor relapse and drug-resistance Ubiquitin-conjugating enzyme Ubc13 controls breast cancer metastasis through a TAK1-p38 MAP kinase cascade.NR2F1 controls tumour cell dormancy via SOX9- and RARβ-driven quiescence programmes.Overexpression of chemokine receptor CXCR2 and ligand CXCL7 in liver metastases from colon cancer is correlated to shorter disease-free and overall survival.Fluoro-Sorafenib (Regorafenib) effects on hepatoma cells: growth inhibition, quiescence, and recovery Variations in genes involved in dormancy associated with outcome in patients with resected colorectal liver metastases Systems biology of tumor dormancy: linking biology and mathematics on multiple scales to improve cancer therapy Increased growth of colorectal liver metastasis following partial hepatectomy Tumor cell dissemination: emerging biological insights from animal models and cancer patients.BMP/Coco antagonism as a deterministic factor of metastasis dormancy in lung.Angiopoietin-2 promotes ER+ breast cancer cell survival in bone marrow niche.A hypoxic signature marks tumors formed by disseminated tumor cells in the BALB-neuT mammary cancer model.TES inhibits colorectal cancer progression through activation of p38.Targeting signal transduction pathways of cancer stem cells for therapeutic opportunities of metastasis.Metastatic stem cells: sources, niches, and vital pathways.Effects of p38α/β inhibition on acute lymphoblastic leukemia proliferation and survival in vivo Biology and significance of circulating and disseminated tumour cells in colorectal cancer.Insights into the biology and prevention of tumor metastasis provided by the Nm23 metastasis suppressor gene.Using MKK4's metastasis suppressor function to identify and dissect cancer cell-microenvironment interactions during metastatic colonization.The dormancy dilemma: quiescence versus balanced proliferation.Tumor metastasis: moving new biological insights into the clinic.The role of the microenvironment-dormant prostate disseminated tumor cells in the bone marrow.Normal vs cancer thyroid stem cells: the road to transformation.Immunogenic, cellular, and angiogenic drivers of tumor dormancy--a melanoma view.Downregulation of estrogen receptor and modulation of growth of breast cancer cell lines mediated by paracrine stromal cell signals.Suppression of pancreatic cancer growth and metastasis by HMP19 identified through genome-wide shRNA screen.Insulin-like growth factor 1 receptor and p38 mitogen-activated protein kinase signals inversely regulate signal transducer and activator of transcription 3 activity to control human dental pulp stem cell quiescence, propagation, and differentiation BMP9-Induced Survival Effect in Liver Tumor Cells Requires p38MAPK Activation.Urokinase is a negative modulator of Egf-dependent proliferation and motility in the two breast cancer cell lines MCF-7 and MDA-MB-231.The Relationship Between Dormant Cancer Cells and Their Microenvironment.Metastasis: an early event in cancer progression.Means to the ends: The role of telomeres and telomere processing machinery in metastasis.Investigational serine/threonine kinase inhibitors against prostate cancer metastases.Emerging Biological Principles of Metastasis.

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P2860

ERK1/2 and p38α/β signaling in tumor cell quiescence: opportunities to control dormant residual disease.

http://www.wikidata.org/entity/Q35579435

description

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Alvaro Avivar-Valderas

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Huei-Chi Wen

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Julio A Aguirre-Ghiso

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Maria Soledad Sosa

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Paloma Bragado

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P2860

A Mutant-p53/Smad complex opposes p63 to empower TGFbeta-induced metastasis

The genomic landscapes of human breast and colorectal cancers

Amplification of PPM1D in human tumors abrogates p53 tumor-suppressor activity

BIM regulates apoptosis during mammary ductal morphogenesis, and its absence reveals alternative cell death mechanisms

Microenvironmental regulation of metastasis

Metastasis: from dissemination to organ-specific colonization

Systemic spread is an early step in breast cancer

Genomic alterations indicate tumor origin and varied metastatic potential of disseminated cells from prostate cancer patients

Mirk/Dyrk1B maintains the viability of quiescent pancreatic cancer cells by reducing levels of reactive oxygen species.

Discovery of a novel unfolded protein response phenotype of cancer stem/progenitor cells from the bone marrow of breast cancer patients.

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5850-5857

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10.1158/1078-0432.CCR-10-2574

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18

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