TNFalpha-induced MMP-9 promotes macrophage recruitment into injured peripheral nerve.
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Spinal Gap Junction Channels in Neuropathic PainWallerian degeneration: the innate-immune response to traumatic nerve injuryIn vivo nanoneurotoxicity screening using oxidative stress and neuroinflammation paradigmsMagnetic nanoparticles for theragnosticsLDL receptor-related protein-1: a regulator of inflammation in atherosclerosis, cancer, and injury to the nervous systemRepeated social stress enhances the innate immune response to a primary HSV-1 infection in the cornea and trigeminal ganglia of Balb/c miceHigh matrix metalloproteinase production correlates with immune activation and leukocyte migration in leprosy reactional lesions.MMPs initiate Schwann cell-mediated MBP degradation and mechanical nociception after nerve damage.Cytokine regulation of MMP-9 in peripheral glia: implications for pathological processes and pain in injured nerve.Matrix metalloproteinase inhibition enhances the rate of nerve regeneration in vivo by promoting dedifferentiation and mitosis of supporting schwann cellsIn vivo gene knockdown in rat dorsal root ganglia mediated by self-complementary adeno-associated virus serotype 5 following intrathecal delivery.The MMP-9/TIMP-1 axis controls the status of differentiation and function of myelin-forming Schwann cells in nerve regeneration.Targeting anti-inflammatory treatment can ameliorate injury-induced neuropathic pain.Effect of low-level laser therapy (LLLT) on acute neural recovery and inflammation-related gene expression after crush injury in rat sciatic nerve.Interactions between the immune and nervous systems in pain.Gene expression profiling of the rat sciatic nerve in early Wallerian degeneration after injuryMatrix metalloproteinase-14 both sheds cell surface neuronal glial antigen 2 (NG2) proteoglycan on macrophages and governs the response to peripheral nerve injuryTNF-alpha signaling in glaucomatous neurodegenerationMatrix metalloproteinase-9 controls proliferation of NG2+ progenitor cells immediately after spinal cord injury.Wallerian degeneration: gaining perspective on inflammatory events after peripheral nerve injury.The unfolded protein response is a major mechanism by which LRP1 regulates Schwann cell survival after injury.The calcium-binding proteins S100A8 and S100A9 initiate the early inflammatory program in injured peripheral nerves.Chronic constriction injury-induced nociception is relieved by nanomedicine-mediated decrease of rat hippocampal tumor necrosis factorSpinal Glia Division Contributes to Conditioning Lesion-Induced Axon Regeneration Into the Injured Spinal Cord: Potential Role of Cyclic AMP-Induced Tissue Inhibitor of Metalloproteinase-1Distribution and tumor necrosis factor-alpha isoform binding specificity of locally administered etanercept into injured and uninjured rat sciatic nerve.The alternatively spliced fibronectin CS1 isoform regulates IL-17A levels and mechanical allodynia after peripheral nerve injury.Immunodominant fragments of myelin basic protein initiate T cell-dependent pain.miR-182 inhibits Schwann cell proliferation and migration by targeting FGF9 and NTM, respectively at an early stage following sciatic nerve injury.Discovering cytokines as targets for chemotherapy-induced painful peripheral neuropathy.c-Jun is a negative regulator of myelination.Antinociception occurs with a reversal in alpha 2-adrenoceptor regulation of TNF production by peripheral monocytes/macrophages from pro- to anti-inflammatoryMMP-9 controls Schwann cell proliferation and phenotypic remodeling via IGF-1 and ErbB receptor-mediated activation of MEK/ERK pathway.Epithelial cell-derived microvesicles activate macrophages and promote inflammation via microvesicle-containing microRNAs.Augmenting nerve regeneration with electrical stimulation.Therapeutic potential of matrix metalloprotease inhibitors in neuropathic pain.Dual peripheral actions of immune cells in neuropathic pain.The ology of neuropathy: an integrative review of the role of neuroinflammation and TNF-α axonal transport in neuropathic pain.Tumor necrosis factor-alpha-converting enzyme activities and tumor-associated macrophages in breast cancer.The transition from acute to chronic pain: understanding how different biological systems interact.Molecules involved in the crosstalk between immune- and peripheral nerve Schwann cells.
P2860
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P2860
TNFalpha-induced MMP-9 promotes macrophage recruitment into injured peripheral nerve.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
TNFalpha-induced MMP-9 promotes macrophage recruitment into injured peripheral nerve.
@ast
TNFalpha-induced MMP-9 promotes macrophage recruitment into injured peripheral nerve.
@en
type
label
TNFalpha-induced MMP-9 promotes macrophage recruitment into injured peripheral nerve.
@ast
TNFalpha-induced MMP-9 promotes macrophage recruitment into injured peripheral nerve.
@en
prefLabel
TNFalpha-induced MMP-9 promotes macrophage recruitment into injured peripheral nerve.
@ast
TNFalpha-induced MMP-9 promotes macrophage recruitment into injured peripheral nerve.
@en
P2093
P2860
P1476
TNFalpha-induced MMP-9 promotes macrophage recruitment into injured peripheral nerve.
@en
P2093
Jennifer Dolkas
Kai Palenscar
Mila Angert
Robert R Myers
Veronica I Shubayev
W Marie Campana
P2860
P304
P356
10.1016/J.MCN.2005.10.011
P577
2005-11-16T00:00:00Z