Gender-Specific Mechanism of Synaptic Impairment and Its Prevention by GCSF in a Mouse Model of ALS. - Wikidata - wikimedia - TriplyDB

Gender-Specific Mechanism of Synaptic Impairment and Its Prevention by GCSF in a Mouse Model of ALS.

Gender-Specific Mechanism of Synaptic Impairment and Its Prevention by GCSF in a Mouse Model of ALS.

http://www.wikidata.org/entity/Q35613992

about

The Granulocyte-colony stimulating factor has a dual role in neuronal and vascular plasticity Generation of Functional Neuromuscular Junctions from Human Pluripotent Stem Cell Lines.Modulation of astrocytic mitochondrial function by dichloroacetate improves survival and motor performance in inherited amyotrophic lateral sclerosis The role of oxidative stress in degeneration of the neuromuscular junction in amyotrophic lateral sclerosis The lack of CuZnSOD leads to impaired neurotransmitter release, neuromuscular junction destabilization and reduced muscle strength in mice.Adenosine A2A receptors activation facilitates neuromuscular transmission in the pre-symptomatic phase of the SOD1(G93A) ALS mice, but not in the symptomatic phase.Early changes of neuromuscular transmission in the SOD1(G93A) mice model of ALS start long before motor symptoms onset Gene expression changes in spinal motoneurons of the SOD1(G93A) transgenic model for ALS after treatment with G-CSF.Homocysteine aggravates ROS-induced depression of transmitter release from motor nerve terminals: potential mechanism of peripheral impairment in motor neuron diseases associated with hyperhomocysteinemia.MicroNeurotrophins Improve Survival in Motor Neuron-Astrocyte Co-Cultures but Do Not Improve Disease Phenotypes in a Mutant SOD1 Mouse Model of Amyotrophic Lateral Sclerosis.Expression of a Mutant SEMA3A Protein with Diminished Signalling Capacity Does Not Alter ALS-Related Motor Decline, or Confer Changes in NMJ Plasticity after BotoxA-Induced Paralysis of Male Gastrocnemic Muscle Morphological and functional changes in innervation of a fast forelimb muscle in SOD1-G85R mice.Pyrrolidine dithiocarbamate activates the Nrf2 pathway in astrocytes.Gacyclidine improves the survival and reduces motor deficits in a mouse model of amyotrophic lateral sclerosis.Loss and gain of FUS function impair neuromuscular synaptic transmission in a genetic model of ALS.Sporadic amyotrophic lateral sclerosis (SALS) - skeletal muscle response to cerebrospinal fluid from SALS patients in a rat model.Amyotrophic Lateral Sclerosis (ALS) and Adenosine Receptors.Synaptic Failure: Focus in an Integrative View of ALS.

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Gender-Specific Mechanism of Synaptic Impairment and Its Prevention by GCSF in a Mouse Model of ALS.

http://www.wikidata.org/entity/Q35613992

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2011 nî lūn-bûn

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2011年の論文

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2011年論文

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Gender-Specific Mechanism of S ...... GCSF in a Mouse Model of ALS.

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Gender-Specific Mechanism of S ...... GCSF in a Mouse Model of ALS.

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Gender-Specific Mechanism of S ...... GCSF in a Mouse Model of ALS.

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Gender-Specific Mechanism of S ...... GCSF in a Mouse Model of ALS.

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Gender-Specific Mechanism of S ...... GCSF in a Mouse Model of ALS.

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Gender-Specific Mechanism of S ...... GCSF in a Mouse Model of ALS.

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FNCEL.2011.00026

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Frontiers in Cellular Neuroscience

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Gender-Specific Mechanism of S ...... GCSF in a Mouse Model of ALS.

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Anastasia Shakirzyanova

http://www.w3.org/2001/XMLSchema#string

Antti Kurronen

http://www.w3.org/2001/XMLSchema#string

Eveliina Pollari

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Jari Koistinaho

http://www.w3.org/2001/XMLSchema#string

Johanna Magga

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Nikolay Naumenko

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Raisa Giniatullina

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Rashid Giniatullin

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P2860

The hematopoietic factor G-CSF is a neuronal ligand that counteracts programmed cell death and drives neurogenesis

Granulocyte-colony stimulating factor improves outcome in a mouse model of amyotrophic lateral sclerosis

Early and selective loss of neuromuscular synapse subtypes with low sprouting competence in motoneuron diseases

Astrocytes expressing ALS-linked mutated SOD1 release factors selectively toxic to motor neurons

Hypertonic enhancement of transmitter release from frog motor nerve terminals: Ca2+ independence and role of integrins

ALS: a disease of motor neurons and their nonneuronal neighbors

Nerve terminal degeneration is independent of muscle fiber genotype in SOD1 mice

Skeletal muscle-restricted expression of human SOD1 causes motor neuron degeneration in transgenic mice

Transgenic mouse model for familial amyotrophic lateral sclerosis with superoxide dismutase-1 mutation.

Current hypotheses for the underlying biology of amyotrophic lateral sclerosis.

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10.3389/FNCEL.2011.00026

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5

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2011-12-12T00:00:00Z

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22180738

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amyotrophic lateral sclerosis

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3238042

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