Shear stress-induced changes in atherosclerotic plaque composition are modulated by chemokines. - Wikidata - wikimedia - TriplyDB

Shear stress-induced changes in atherosclerotic plaque composition are modulated by chemokines.

Shear stress-induced changes in atherosclerotic plaque composition are modulated by chemokines.

http://www.wikidata.org/entity/Q35629011

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High shear stress induces atherosclerotic vulnerable plaque formation through angiogenesis The multifaceted functions of CXCL10 in cardiovascular disease Chemokines as effector and target molecules in vascular biology Disturbed Flow Induces Autophagy, but Impairs Autophagic Flux to Perturb Mitochondrial Homeostasis.Biomechanical factors and macrophages in plaque stability The CXCL10/CXCR3 Axis and Cardiac Inflammation: Implications for Immunotherapy to Treat Infectious and Noninfectious Diseases of the Heart Influence of shear stress magnitude and direction on atherosclerotic plaque composition Low intensity shear stress increases endothelial ELR+ CXC chemokine production via a focal adhesion kinase-p38{beta} MAPK-NF-{kappa}B pathway.The role of cellular adaptation to mechanical forces in atherosclerosis.Atherosclerosis-prone hemodynamics differentially regulates endothelial and smooth muscle cell phenotypes and promotes pro-inflammatory priming.Host-derived smooth muscle cells accumulate in cardiac allografts: role of inflammation and monocyte chemoattractant protein 1.Innate and adaptive immunity in atherosclerosis.Anti-inflammatory treatment with pentoxifylline improves HIV-related endothelial dysfunction: a pilot study.Effects of disturbed flow on vascular endothelium: pathophysiological basis and clinical perspectives PEG-albumin plasma expansion increases expression of MCP-1 evidencing increased circulatory wall shear stress: an experimental study.Differential endothelial cell gene expression by African Americans versus Caucasian Americans: a possible contribution to health disparity in vascular disease and cancer.Coronary computed tomography angiography based assessment of endothelial shear stress and its association with atherosclerotic plaque distribution in-vivo.Elevated uptake of plasma macromolecules by regions of arterial wall predisposed to plaque instability in a mouse model.Atherosclerotic Plaque Stability Is Affected by the Chemokine CXCL10 in Both Mice and Humans.Role of PECAM-1 in arteriogenesis and specification of preexisting collaterals The Features of Inflammation Factors Concentrations in Aqueous Humor of Polypoidal Choroidal Vasculopathy Hck/Fgr Kinase Deficiency Reduces Plaque Growth and Stability by Blunting Monocyte Recruitment and Intraplaque Motility.Blood flow modulation of vascular dynamics.Elevated electrochemical impedance in the endoluminal regions with high shear stress: implication for assessing lipid-rich atherosclerotic lesions.Treatment with Evasin-3 reduces atherosclerotic vulnerability for ischemic stroke, but not brain injury in mice Immune and inflammatory mechanisms of atherosclerosis (*)Chemokines as therapeutic targets for atherosclerotic plaque destabilization and rupture.The Role of Immunogenicity in Cardiovascular Disease.Macrophage heterogeneity in atherosclerotic plaques.Chemokines in the vascular inflammatory response of atherosclerosis.Cervical Rotatory Manipulation Decreases Uniaxial Tensile Properties of Rabbit Atherosclerotic Internal Carotid Artery.Animal, in vitro, and ex vivo models of flow-dependent atherosclerosis: role of oxidative stress.Endothelial shear stress in the evolution of coronary atherosclerotic plaque and vascular remodelling: current understanding and remaining questions.Inflammation and immune system interactions in atherosclerosis.Systems biology of the functional and dysfunctional endothelium.How do we prevent the vulnerable atherosclerotic plaque from rupturing? Insights from in vivo assessments of plaque, vascular remodeling, and local endothelial shear stress.Role of biomechanical forces in the natural history of coronary atherosclerosis.Laminar shear stress regulates endothelial kinin B1 receptor expression and function: potential implication in atherogenesis.Shear Stress Counteracts Endothelial CX3CL1 Induction and Monocytic Cell Adhesion.Treatment of hypertriglyceridemia and HIV: fenofibrate-induced changes in the expression of chemokine genes in circulating leukocytes

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P2860

Shear stress-induced changes in atherosclerotic plaque composition are modulated by chemokines.

http://www.wikidata.org/entity/Q35629011

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Shear stress-induced changes i ...... n are modulated by chemokines.

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Shear stress-induced changes i ...... n are modulated by chemokines.

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Shear stress-induced changes i ...... n are modulated by chemokines.

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Shear stress-induced changes i ...... n are modulated by chemokines.

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Journal of Clinical Investigation

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Shear stress-induced changes i ...... n are modulated by chemokines.

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P2093

Anton van der Steen

http://www.w3.org/2001/XMLSchema#string

Caroline Cheng

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Dennie Tempel

http://www.w3.org/2001/XMLSchema#string

Dolf Segers

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Hetty C de Boer

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Martin Huisman

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Pieter J M Leenen

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Rien van Haperen

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Rini de Crom

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Rob Krams

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P2860

Solution structure and dynamics of the CX3C chemokine domain of fractalkine and its interaction with an N-terminal fragment of CX3CR1

Fractalkine is expressed by smooth muscle cells in response to IFN-gamma and TNF-alpha and is modulated by metalloproteinase activity

Human interferon-inducible protein-10 induces mononuclear cell infiltration in mice and promotes the migration of human T lymphocytes into the peripheral tissues and human peripheral blood lymphocytes-SCID mice

Chemokine receptor specific for IP10 and mig: structure, function, and expression in activated T-lymphocytes

Mast cells migrate, but do not degranulate, in response to fractalkine, a membrane-bound chemokine expressed constitutively in diverse cells of the skin

Mechanisms that regulate the function of the selectins and their ligands

Novel role of the membrane-bound chemokine fractalkine in platelet activation and adhesion

Optimized RNA amplification using T7-RNA-polymerase based in vitro transcription.

Major reduction of atherosclerosis in fractalkine (CX3CL1)-deficient mice is at the brachiocephalic artery, not the aortic root.

Mast cells and basophils.

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616-626

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10.1172/JCI28180

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3

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117

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Anton Jan van Zonneveld

Patrick W Serruys

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2007-02-15T00:00:00Z

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6501860

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17304353

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atherosclerosis

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1794116

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