Bid-independent mitochondrial activation in tumor necrosis factor alpha-induced apoptosis and liver injury - Wikidata - wikimedia - TriplyDB

Bid-independent mitochondrial activation in tumor necrosis factor alpha-induced apoptosis and liver injury

Bid-independent mitochondrial activation in tumor necrosis factor alpha-induced apoptosis and liver injury

http://www.wikidata.org/entity/Q35642101

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A nanoconjugate Apaf-1 inhibitor protects mesothelial cells from cytokine-induced injury Ectodomain shedding of EGFR ligands and TNFR1 dictates hepatocyte apoptosis during fulminant hepatitis in mice VDAC2 is required for truncated BID-induced mitochondrial apoptosis by recruiting BAK to the mitochondria Modeling the TNFα-induced apoptosis pathway in hepatocytes.The strength of the Fas ligand signal determines whether hepatocytes act as type 1 or type 2 cells in murine livers.Deficiency of Bid protein reduces sepsis-induced apoptosis and inflammation, while improving septic survival.Fatal hepatitis mediated by tumor necrosis factor TNFalpha requires caspase-8 and involves the BH3-only proteins Bid and Bim.TNF-alpha signaling in glaucomatous neurodegeneration Mitochondrial permeability transition in rat hepatocytes after anoxia/reoxygenation: role of Ca2+-dependent mitochondrial formation of reactive oxygen species.Protein kinase C-β inhibitor treatment attenuates hepatic ischemia and reperfusion injury in diabetic rats Differential roles of JNK in ConA/GalN and ConA-induced liver injury in mice.Neuroinflammatory mechanisms in Parkinson's disease: potential environmental triggers, pathways, and targets for early therapeutic intervention Genetic delineation of the pathways mediated by bid and JNK in tumor necrosis factor-alpha-induced liver injury in adult and embryonic mice.Tumor necrosis factor-alpha induces intrinsic apoptotic signaling during renal obstruction through truncated bid activation.Murine cytomegalovirus infection and apoptosis in organotypic retinal cultures.Rejuvenating Bi(d)ology.A novel scoring system based on common laboratory tests predicts the efficacy of TNF-inhibitor and IL-6 targeted therapy in patients with rheumatoid arthritis: a retrospective, multicenter observational study JNK1/2 regulate Bid by direct phosphorylation at Thr59 in response to ALDH1L1.Caspase Inhibition Prevents Tumor Necrosis Factor-α-Induced Apoptosis and Promotes Necrotic Cell Death in Mouse Hepatocytes in Vivo and in Vitro.

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Bid-independent mitochondrial activation in tumor necrosis factor alpha-induced apoptosis and liver injury

http://www.wikidata.org/entity/Q35642101

description

2006 nî lūn-bûn

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

@zh-tw

2006年论文

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2006年论文

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2006年论文

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name

Bid-independent mitochondrial ...... ced apoptosis and liver injury

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Bid-independent mitochondrial ...... ced apoptosis and liver injury

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type

label

Bid-independent mitochondrial ...... ced apoptosis and liver injury

@ast

Bid-independent mitochondrial ...... ced apoptosis and liver injury

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prefLabel

Bid-independent mitochondrial ...... ced apoptosis and liver injury

@ast

Bid-independent mitochondrial ...... ced apoptosis and liver injury

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Molecular and Cellular Biology

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Bid-independent mitochondrial ...... ced apoptosis and liver injury

@en

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Amer A Beg

http://www.w3.org/2001/XMLSchema#string

Andrea A Gambotto

http://www.w3.org/2001/XMLSchema#string

Hong-Min Ni

http://www.w3.org/2001/XMLSchema#string

Jia Fan

http://www.w3.org/2001/XMLSchema#string

Wen-Xing Ding

http://www.w3.org/2001/XMLSchema#string

Wentao Gao

http://www.w3.org/2001/XMLSchema#string

Xiao-Ming Yin

http://www.w3.org/2001/XMLSchema#string

Xiaoyun Chen

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Ying-Hong Shi

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tBID, a membrane-targeted death ligand, oligomerizes BAK to release cytochrome c

In vitro activation of CPP32 and Mch3 by Mch4, a novel human apoptotic cysteine protease containing two FADD-like domains

Inhibition of death receptor signals by cellular FLIP

BID: a novel BH3 domain-only death agonist

Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

Signal transduction by the JNK group of MAP kinases

To be, or not to be: NF-kappaB is the answer--role of Rel/NF-kappaB in the regulation of apoptosis

Bcl-2 and Bcl-xL inhibit CD95-mediated apoptosis by preventing mitochondrial release of Smac/DIABLO and subsequent inactivation of X-linked inhibitor-of-apoptosis protein

Reactive oxygen species promote TNFalpha-induced death and sustained JNK activation by inhibiting MAP kinase phosphatases

Cell death: critical control points

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541-553

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scholarly article

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10.1128/MCB.01166-06

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2

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27

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2006-11-13T00:00:00Z

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6695409

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17101783

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1800794

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