Regulation of HDAC9 gene expression by MEF2 establishes a negative-feedback loop in the transcriptional circuitry of muscle differentiation - Wikidata - wikimedia - TriplyDB

Regulation of HDAC9 gene expression by MEF2 establishes a negative-feedback loop in the transcriptional circuitry of muscle differentiation

Regulation of HDAC9 gene expression by MEF2 establishes a negative-feedback loop in the transcriptional circuitry of muscle differentiation

http://www.wikidata.org/entity/Q35642166

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The many roles of histone deacetylases in development and physiology: implications for disease and therapy Histone deacetylase degradation and MEF2 activation promote the formation of slow-twitch myofibers HDAC4 reduction: a novel therapeutic strategy to target cytoplasmic huntingtin and ameliorate neurodegeneration Regulation of class IIa HDAC activities: it is not only matter of subcellular localization Regulation of myoblast differentiation by the nuclear envelope protein NET39 Polycystin-dependent fluid flow sensing targets histone deacetylase 5 to prevent the development of renal cysts Polyethyleneimine mediated DNA transfection in schistosome parasites and regulation of the WNT signaling pathway by a dominant-negative SmMef2 The Rpd3/Hda1 family of lysine deacetylases: from bacteria and yeast to mice and men Identification of novel transcriptional regulators involved in macrophage differentiation and activation in U937 cells.Effect of DLK1 and RTL1 but not MEG3 or MEG8 on muscle gene expression in Callipyge lambs.The Isl1-Lhx3 Complex Promotes Motor Neuron Specification by Activating Transcriptional Pathways that Enhance Its Own Expression and Formation Predicting targeted drug combinations based on Pareto optimal patterns of coexpression network connectivity A systems biology approach identifies molecular networks defining skeletal muscle abnormalities in chronic obstructive pulmonary disease Genome-wide mapping of Sox6 binding sites in skeletal muscle reveals both direct and indirect regulation of muscle terminal differentiation by Sox6.NCoR1 is a conserved physiological modulator of muscle mass and oxidative function.Building muscle: molecular regulation of myogenesis.Rare copy number variations in adults with tetralogy of Fallot implicate novel risk gene pathways The silencing of the SWI/SNF subunit and anticancer gene BRM in Rhabdoid tumors A rapid one-generation genetic screen in a Drosophila model to capture rhabdomyosarcoma effectors and therapeutic targets Histone deacetylase 9 is a negative regulator of adipogenic differentiation.HDAC9 Inhibits Osteoclastogenesis via Mutual Suppression of PPARγ/RANKL Signaling.A cis-regulatory mutation in troponin-I of Drosophila reveals the importance of proper stoichiometry of structural proteins during muscle assembly.MASTR directs MyoD-dependent satellite cell differentiation during skeletal muscle regeneration Panhistone deacetylase inhibitors inhibit proinflammatory signaling pathways to ameliorate interleukin-18-induced cardiac hypertrophy.Epigenetics and autosomal dominant polycystic kidney disease.Glucocorticoid receptor and myocyte enhancer factor 2 cooperate to regulate the expression of c-JUN in a neuronal context Acetylation of myocardin is required for the activation of cardiac and smooth muscle genes.Bone marrow-induced Mef2c deficiency delays B-cell development and alters the expression of key B-cell regulatory proteins.Control of endothelial cell proliferation and migration by VEGF signaling to histone deacetylase 7.Class IIa histone deacetylases: regulating the regulators.Tissue-specific splicing of a ubiquitously expressed transcription factor is essential for muscle differentiation Genomic Profiling of Adult and Pediatric B-cell Acute Lymphoblastic Leukemia.Ca2+-operated transcriptional networks: molecular mechanisms and in vivo models.Histone deacetylase 9 regulates breast cancer cell proliferation and the response to histone deacetylase inhibitors.SIKs control osteocyte responses to parathyroid hormone Mir-214-dependent regulation of the polycomb protein Ezh2 in skeletal muscle and embryonic stem cells Genomic analyses identify recurrent MEF2D fusions in acute lymphoblastic leukaemia Loss of MEF2D expression inhibits differentiation and contributes to oncogenesis in rhabdomyosarcoma cells Histone H3 Methyltransferase Suv39h1 Prevents Myogenic Terminal Differentiation by Repressing MEF2 Activity in Muscle Cells Deregulated expression of HDAC9 in B cells promotes development of lymphoproliferative disease and lymphoma in mice.

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Regulation of HDAC9 gene expression by MEF2 establishes a negative-feedback loop in the transcriptional circuitry of muscle differentiation

http://www.wikidata.org/entity/Q35642166

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Regulation of HDAC9 gene expre ...... itry of muscle differentiation

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Regulation of HDAC9 gene expre ...... itry of muscle differentiation

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Regulation of HDAC9 gene expre ...... itry of muscle differentiation

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Regulation of HDAC9 gene expre ...... itry of muscle differentiation

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Molecular and Cellular Biology

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Regulation of HDAC9 gene expre ...... itry of muscle differentiation

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Dillon Phan

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Eric N Olson

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John McAnally

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Michael Haberland

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Yuri Kim

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P2860

Regulation of skeletal myogenesis by association of the MEF2 transcription factor with class II histone deacetylases

Signal-dependent nuclear export of a histone deacetylase regulates muscle differentiation

Activation of the myocyte enhancer factor-2 transcription factor by calcium/calmodulin-dependent protein kinase-stimulated binding of 14-3-3 to histone deacetylase 5

Cloning and characterization of a histone deacetylase, HDAC9

CaM kinase II selectively signals to histone deacetylase 4 during cardiomyocyte hypertrophy

An expression screen reveals modulators of class II histone deacetylase phosphorylation

The UCSC Genome Browser Database: update 2006

Identification of a signal-responsive nuclear export sequence in class II histone deacetylases

Requirement for serum response factor for skeletal muscle growth and maturation revealed by tissue-specific gene deletion in mice

A new myocyte-specific enhancer-binding factor that recognizes a conserved element associated with multiple muscle-specific genes

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