Modeling sporadic loss of heterozygosity in mice by using mosaic analysis with double markers (MADM)
about
A genetic strategy for stochastic gene activation with regulated sparseness (STARS)Timing neurogenesis and differentiation: insights from quantitative clonal analyses of cerebellar granule cellsStudies of Tumor Suppressor Genes via Chromosome EngineeringDevelopmental origins of brain tumorsExtensions of MADM (mosaic analysis with double markers) in miceClonal dynamics following p53 loss of heterozygosity in Kras-driven cancers.Cell of origin for malignant gliomas and its implication in therapeutic developmentCortical representations of olfactory input by trans-synaptic tracing.The expanding role of mouse genetics for understanding human biology and diseaseAccelerated turnover of taste bud cells in mice deficient for the cyclin-dependent kinase inhibitor p27Kip1.Generation and analysis of partially haploid cells with Cre-mediated chromosome deletion in the lymphoid system.MADM-ML, a mouse genetic mosaic system with increased clonal efficiencyNew mouse models of cancer: single-cell knockouts.Fly MARCM and mouse MADM: genetic methods of labeling and manipulating single neurons.Generation of Dhx9-deficient clones in T-cell development with a mitotic recombination technique.The effort to make mosaic analysis a household tool.Genetically engineered mouse models in cancer researchA mitotic recombination system for mouse chromosome 17.Mosaic analysis with double markers reveals cell-type-specific paternal growth dominance.Cutaneous melanoma in genetically modified animals.Applications of the site-specific recombinase Cre to the study of genomic imprintingNew approaches for modelling sporadic genetic disease in the mouseUncoupling dendrite growth and patterning: single-cell knockout analysis of NMDA receptor 2B.Lineage-tracing methods and the kidney.LOHAS: loss-of-heterozygosity analysis suite.MASTR: a technique for mosaic mutant analysis with spatial and temporal control of recombination using conditional floxed alleles in mice.Distinct cell-autonomous functions of RETINOBLASTOMA-RELATED in Arabidopsis stem cells revealed by the Brother of Brainbow clonal analysis system.Linking neuronal lineage and wiring specificity.
P2860
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P2860
Modeling sporadic loss of heterozygosity in mice by using mosaic analysis with double markers (MADM)
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Modeling sporadic loss of hete ...... sis with double markers (MADM)
@ast
Modeling sporadic loss of hete ...... sis with double markers (MADM)
@en
type
label
Modeling sporadic loss of hete ...... sis with double markers (MADM)
@ast
Modeling sporadic loss of hete ...... sis with double markers (MADM)
@en
prefLabel
Modeling sporadic loss of hete ...... sis with double markers (MADM)
@ast
Modeling sporadic loss of hete ...... sis with double markers (MADM)
@en
P2093
P2860
P356
P1476
Modeling sporadic loss of hete ...... sis with double markers (MADM)
@en
P2093
Mandar Deepak Muzumdar
P2860
P304
P356
10.1073/PNAS.0606491104
P407
P577
2007-03-05T00:00:00Z