Elevated synaptic activity preconditions neurons against an in vitro model of ischemia
about
Altered network communication following a neuroprotective drug treatmentActivity-Dependent Degradation of Synaptic Vesicle Proteins Requires Rab35 and the ESCRT PathwayOligodendrocyte precursor cells modulate the neuronal network by activity-dependent ectodomain cleavage of glial NG2.Force spectroscopy measurements show that cortical neurons exposed to excitotoxic agonists stiffen before showing evidence of bleb damage.Excitation-transcription coupling via calcium/calmodulin-dependent protein kinase/ERK1/2 signaling mediates the coordinate induction of VGLUT2 and Narp triggered by a prolonged increase in glutamatergic synaptic activity.Essential involvement of the NMDA receptor in ethanol preconditioning-dependent neuroprotection from amyloid-betain vitro.Increased BDNF protein expression after ischemic or PKC epsilon preconditioning promotes electrophysiologic changes that lead to neuroprotection.Synaptic and extrasynaptic NMDA receptors differentially modulate neuronal cyclooxygenase-2 function, lipid peroxidation, and neuroprotection.Extracting functionally feedforward networks from a population of spiking neurons.Burst predicting neurons survive an in vitro glutamate injury model of cerebral ischemiaPituitary adenylate cyclase-activating peptide induces long-lasting neuroprotection through the induction of activity-dependent signaling via the cyclic AMP response element-binding protein-regulated transcription co-activator 1.Statistical Evaluation of Waveform Collapse Reveals Scale-Free Properties of Neuronal Avalanches.The influence of synaptic activity on neuronal health.Global cerebral ischemia: synaptic and cognitive dysfunction.Mitochondrial calcium uniporter Mcu controls excitotoxicity and is transcriptionally repressed by neuroprotective nuclear calcium signals.Cerebral ischemic preconditioning reduces glutamate excitotoxicity by up-regulating the uptake activity of GLT-1 in rats.NR2D-containing NMDA receptors mediate tissue plasminogen activator-promoted neuronal excitotoxicity.Knocking down of the KCC2 in rat hippocampal neurons increases intracellular chloride concentration and compromises neuronal survival.BrainPhys® increases neurofilament levels in CNS cultures, and facilitates investigation of axonal damage after a mechanical stretch-injury in vitro.Glycine bidirectionally regulates ischemic tolerance via different mechanisms including NR2A-dependent CREB phosphorylation.N-methyl-D-aspartate preconditioning prevents quinolinic acid-induced deregulation of glutamate and calcium homeostasis in mice hippocampus.Scale-free and economical features of functional connectivity in neuronal networks.
P2860
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P2860
Elevated synaptic activity preconditions neurons against an in vitro model of ischemia
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Elevated synaptic activity preconditions neurons against an in vitro model of ischemia
@ast
Elevated synaptic activity preconditions neurons against an in vitro model of ischemia
@en
type
label
Elevated synaptic activity preconditions neurons against an in vitro model of ischemia
@ast
Elevated synaptic activity preconditions neurons against an in vitro model of ischemia
@en
prefLabel
Elevated synaptic activity preconditions neurons against an in vitro model of ischemia
@ast
Elevated synaptic activity preconditions neurons against an in vitro model of ischemia
@en
P2093
P2860
P356
P1476
Elevated synaptic activity preconditions neurons against an in vitro model of ischemia
@en
P2093
Eric Brunette
Geoffrey A R Mealing
Jean-Philippe Thivierge
Joseph S Tauskela
Melissa Hewitt
Tanya Comas
Tarun Ahuja
P2860
P304
34667-34676
P356
10.1074/JBC.M805624200
P407
P577
2008-10-09T00:00:00Z