VEGFR-3 controls tip to stalk conversion at vessel fusion sites by reinforcing Notch signalling. - Wikidata - wikimedia - TriplyDB

VEGFR-3 controls tip to stalk conversion at vessel fusion sites by reinforcing Notch signalling.

VEGFR-3 controls tip to stalk conversion at vessel fusion sites by reinforcing Notch signalling.

http://www.wikidata.org/entity/Q35682284

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"Decoding" Angiogenesis: New Facets Controlling Endothelial Cell Behavior Lymphatic Vessels, Inflammation, and Immunity in Skin Cancer Biomarkers of Angiogenesis in Colorectal Cancer Vascularisation of the central nervous system Angiogenesis in metastatic colorectal cancer and the benefits of targeted therapy Endothelial tip cells in ocular angiogenesis: potential target for anti-angiogenesis therapy Receptor tyrosine kinase-mediated angiogenesis Improved Angiogenesis in Response to Localized Delivery of Macrophage-Recruiting Molecules Canonical and noncanonical vascular endothelial growth factor pathways: new developments in biology and signal transduction.Structural and mechanistic insights into VEGF receptor 3 ligand binding and activation VEGF signaling inside vascular endothelial cells and beyond.Schlemm's canal is a unique vessel with a combination of blood vascular and lymphatic phenotypes that forms by a novel developmental process Notch-dependent VEGFR3 upregulation allows angiogenesis without VEGF-VEGFR2 signalling Macrophage biology in development, homeostasis and disease A novel mechanism for sevoflurane preconditioning-induced neuroprotection.Multiscale mechanisms of cell migration during development: theory and experiment.A truncation allele in vascular endothelial growth factor c reveals distinct modes of signaling during lymphatic and vascular development.Macrophages regulate corpus luteum development during embryo implantation in mice.The embryonic mouse hindbrain as a qualitative and quantitative model for studying the molecular and cellular mechanisms of angiogenesis Lymphangiogenesis and Inflammation-Looking for the "Missing Pieces" of the Puzzle.Efficacy of pEgr-1-endostatin combined with ionizing radiation on hypoxic conditions in nude mice bearing SKOV3 ovarian carcinoma.A ligand-independent VEGFR2 signaling pathway limits angiogenic responses in diabetes.VEGFR2 but not VEGFR3 governs integrity and remodeling of thyroid angiofollicular unit in normal state and during goitrogenesis.Critical role of aberrant angiogenesis in the development of tumor hypoxia and associated radioresistance Hif-1α regulates macrophage-endothelial interactions during blood vessel development in zebrafish.Small RNA combination therapy for lung cancer In vivo endothelial siRNA delivery using polymeric nanoparticles with low molecular weight The impact of microglial activation on blood-brain barrier in brain diseases.Leukotriene B4 antagonism ameliorates experimental lymphedema.Involvement of lysosomal degradation in VEGF-C-induced down-regulation of VEGFR-3.Biomarkers for anti-angiogenic therapy in cancer.The sinus venosus contributes to coronary vasculature through VEGFC-stimulated angiogenesis.VEGFR3 does not sustain retinal angiogenesis without VEGFR2 NOTCH decoys that selectively block DLL/NOTCH or JAG/NOTCH disrupt angiogenesis by unique mechanisms to inhibit tumor growth.Autotaxin overexpression causes embryonic lethality and vascular defects Endothelial Snail Regulates Capillary Branching Morphogenesis via Vascular Endothelial Growth Factor Receptor 3 Expression The matricellular protein CCN1 controls retinal angiogenesis by targeting VEGF, Src homology 2 domain phosphatase-1 and Notch signaling.In vivo imaging of lymphatic vessels in development, wound healing, inflammation, and tumor metastasis.Notch signals in the endothelium and cancer "stem-like" cells: opportunities for cancer therapy.Alk1 and Alk5 inhibition by Nrp1 controls vascular sprouting downstream of Notch.

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P2860

VEGFR-3 controls tip to stalk conversion at vessel fusion sites by reinforcing Notch signalling.

http://www.wikidata.org/entity/Q35682284

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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name

VEGFR-3 controls tip to stalk ...... reinforcing Notch signalling.

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VEGFR-3 controls tip to stalk ...... reinforcing Notch signalling.

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type

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VEGFR-3 controls tip to stalk ...... reinforcing Notch signalling.

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VEGFR-3 controls tip to stalk ...... reinforcing Notch signalling.

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prefLabel

VEGFR-3 controls tip to stalk ...... reinforcing Notch signalling.

@ast

VEGFR-3 controls tip to stalk ...... reinforcing Notch signalling.

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P1433

Nature Cell Biology

P1476

VEGFR-3 controls tip to stalk ...... y reinforcing Notch signalling

@en

P2093

Aino Murtomäki

http://www.w3.org/2001/XMLSchema#string

Anne Eichmann

http://www.w3.org/2001/XMLSchema#string

Denis Tvorogov

http://www.w3.org/2001/XMLSchema#string

Evelyn Aranda

http://www.w3.org/2001/XMLSchema#string

Georgia Zarkada

http://www.w3.org/2001/XMLSchema#string

Jeffrey W Pollard

http://www.w3.org/2001/XMLSchema#string

Kari Alitalo

http://www.w3.org/2001/XMLSchema#string

Krista Heinolainen

http://www.w3.org/2001/XMLSchema#string

Naoyuki Miura

http://www.w3.org/2001/XMLSchema#string

Taija Mäkinen

http://www.w3.org/2001/XMLSchema#string

P2860

Isolated lymphatic endothelial cells transduce growth, survival and migratory signals via the VEGF-C/D receptor VEGFR-3

VEGF receptor 2/-3 heterodimers detected in situ by proximity ligation on angiogenic sprouts

VEGF-C is a trophic factor for neural progenitors in the vertebrate embryonic brain

Effective suppression of vascular network formation by combination of antibodies blocking VEGFR ligand binding and receptor dimerization

FOXC2 controls formation and maturation of lymphatic collecting vessels through cooperation with NFATc1

Delta-like ligand 4 (Dll4) is induced by VEGF as a negative regulator of angiogenic sprouting

Generalized lacZ expression with the ROSA26 Cre reporter strain

Targeted deficiency or cytosolic truncation of the VE-cadherin gene in mice impairs VEGF-mediated endothelial survival and angiogenesis

Vascular endothelial growth factor C is required for sprouting of the first lymphatic vessels from embryonic veins

Vascular endothelial growth factor receptor-2 and neuropilin-1 form a receptor complex that is responsible for the differential signaling potency of VEGF(165) and VEGF(121)

P2888

P304

1202-1213

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scholarly article

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10.1038/NCB2331

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10

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13

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Seppo Ylä-Herttuala

Holger Gerhardt

Claudio A. Franco

Marcus Fruttiger

Holger Gerhardt

P577

2011-09-11T00:00:00Z

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51640505

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21909098

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3261765

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