TORC1 Inhibition by Rapamycin Promotes Antioxidant Defences in a Drosophila Model of Friedreich's Ataxia - Wikidata - wikimedia - TriplyDB

TORC1 Inhibition by Rapamycin Promotes Antioxidant Defences in a Drosophila Model of Friedreich's Ataxia

TORC1 Inhibition by Rapamycin Promotes Antioxidant Defences in a Drosophila Model of Friedreich's Ataxia

http://www.wikidata.org/entity/Q35687064

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Circulating miR-323-3p is a biomarker for cardiomyopathy and an indicator of phenotypic variability in Friedreich's ataxia patients.Metal Homeostasis Regulators Suppress FRDA Phenotypes in a Drosophila Model of the Disease Rapamycin alleviates oxidative stress-induced damage in rat erythrocytes.Whey Protein Concentrate Renders MDA-MB-231 Cells Sensitive to Rapamycin by Altering Cellular Redox State and Activating GSK3β/mTOR Signaling.Rapamycin protects testes against germ cell apoptosis and oxidative stress induced by testicular ischemia-reperfusion.The role of oxidative stress in Friedreich's ataxia.Rapamycin mitigates erythrocyte membrane transport functions and oxidative stress during aging in rats.Ketamine Exhibits Different Neuroanatomical Profile After Mammalian Target of Rapamycin Inhibition in the Prefrontal Cortex: the Role of Inflammation and Oxidative Stress.Mitofusin-Dependent ER Stress Triggers Glial Dysfunction and Nervous System Degeneration in a Drosophila Model of Friedreich's Ataxia.Iron Sulfur and Molybdenum Cofactor Enzymes Regulate the Drosophila Life Cycle by Controlling Cell Metabolism.Drosophila melanogaster Models of Friedreich's Ataxia.

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TORC1 Inhibition by Rapamycin Promotes Antioxidant Defences in a Drosophila Model of Friedreich's Ataxia

http://www.wikidata.org/entity/Q35687064

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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TORC1 Inhibition by Rapamycin ...... a Model of Friedreich's Ataxia

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TORC1 Inhibition by Rapamycin ...... a Model of Friedreich's Ataxia

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TORC1 Inhibition by Rapamycin ...... a Model of Friedreich's Ataxia

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TORC1 Inhibition by Rapamycin ...... a Model of Friedreich's Ataxia

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TORC1 Inhibition by Rapamycin ...... a Model of Friedreich's Ataxia

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TORC1 Inhibition by Rapamycin ...... a Model of Friedreich's Ataxia

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JOURNAL.PONE.0132376

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TORC1 Inhibition by Rapamycin ...... a Model of Friedreich's Ataxia

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Ismael Al-Ramahi

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Juan Botas

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María Dolores Moltó

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María José Martínez-Sebastián

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Pablo Calap-Quintana

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Sirena Soriano

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P2860

Antioxidants and other pharmacological treatments for Friedreich ataxia

Reduced TOR signaling extends chronological life span via increased respiration and upregulation of mitochondrial gene expression

TOR signaling in growth and metabolism

Glutathione-dependent redox status of frataxin-deficient cells in a yeast model of Friedreich's ataxia.

TOR signaling and rapamycin influence longevity by regulating SKN-1/Nrf and DAF-16/FoxO

Overexpression of frataxin in the mitochondria increases resistance to oxidative stress and extends lifespan in Drosophila

Friedreich's ataxia: autosomal recessive disease caused by an intronic GAA triplet repeat expansion

Impaired nuclear Nrf2 translocation undermines the oxidative stress response in Friedreich ataxia

Overexpression of human and fly frataxins in Drosophila provokes deleterious effects at biochemical, physiological and developmental levels

mTOR inhibition induces upstream receptor tyrosine kinase signaling and activates Akt

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10.1371/JOURNAL.PONE.0132376

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7

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José Vicente Llorens

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2015-07-09T00:00:00Z

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