about
Overexpression of human and fly frataxins in Drosophila provokes deleterious effects at biochemical, physiological and developmental levelsEarly Social Isolation Stress and Perinatal NMDA Receptor Antagonist Treatment Induce Changes in the Structure and Neurochemistry of Inhibitory Neurons of the Adult Amygdala and Prefrontal Cortex.TORC1 Inhibition by Rapamycin Promotes Antioxidant Defences in a Drosophila Model of Friedreich's AtaxiaMetal Homeostasis Regulators Suppress FRDA Phenotypes in a Drosophila Model of the DiseaseFriedreich ataxia: an update on animal models, frataxin function and therapies.Drosophila melanogaster Models of Metal-Related Human Diseases and Metal Toxicity.Causative role of oxidative stress in a Drosophila model of Friedreich ataxia.Mitochondrial iron supply is required for the developmental pulse of ecdysone biosynthesis that initiates metamorphosis in Drosophila melanogaster.Drosophila melanogaster Models of Friedreich's Ataxia.Gypsy endogenous retrovirus maintains potential infectivity in several species of DrosophilidsImpact of Models in the Study and Treatment of Friedreich's Ataxia
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
José Vicente Llorens
@ast
José Vicente Llorens
@en
José Vicente Llorens
@es
José Vicente Llorens
@nl
type
label
José Vicente Llorens
@ast
José Vicente Llorens
@en
José Vicente Llorens
@es
José Vicente Llorens
@nl
prefLabel
José Vicente Llorens
@ast
José Vicente Llorens
@en
José Vicente Llorens
@es
José Vicente Llorens
@nl
P106
P1153
15839898100
P21
P31
P496
0000-0001-9823-6246