DTNBP1 genotype influences cognitive decline in schizophrenia. - Wikidata - wikimedia - TriplyDB

DTNBP1 genotype influences cognitive decline in schizophrenia.

DTNBP1 genotype influences cognitive decline in schizophrenia.

http://www.wikidata.org/entity/Q35688111

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Association between the DTNBP1 gene and intelligence: a case-control study in young patients with schizophrenia and related disorders and unaffected siblings TRIM32 is an E3 ubiquitin ligase for dysbindin RGS4 polymorphisms associated with variability of cognitive performance in a family-based schizophrenia sample Circuit-based framework for understanding neurotransmitter and risk gene interactions in schizophrenia Gene × Environment Interactions in Schizophrenia: Evidence from Genetic Mouse Models Impaired long-term memory retention and working memory in sdy mutant mice with a deletion in Dtnbp1, a susceptibility gene for schizophrenia Cognitive control deficits in schizophrenia: mechanisms and meaning Neurobehavioral abnormalities in the dysbindin-1 mutant, sandy, on a C57BL/6J genetic background.Exome Sequence Data From Multigenerational Families Implicate AMPA Receptor Trafficking in Neurocognitive Impairment and Schizophrenia Risk.Cognitive heterogeneity in schizophrenia.Dual constraints on synapse formation and regression in schizophrenia: neuregulin, neuroligin, dysbindin, DISC1, MuSK and agrin.Developmental disruptions in neural connectivity in the pathophysiology of schizophrenia.A reappraisal of the association between Dysbindin (DTNBP1) and schizophrenia in a large combined case-control and family-based sample of German ancestry No association of dysbindin with symptom factors of schizophrenia in an Irish case-control sample Synaptic dysbindin-1 reductions in schizophrenia occur in an isoform-specific manner indicating their subsynaptic location.The DTNBP1 (dysbindin-1) gene variant rs2619522 is associated with variation of hippocampal and prefrontal grey matter volumes in humans.DISC1 in schizophrenia: genetic mouse models and human genomic imaging.Differential effects of common variants in SCN2A on general cognitive ability, brain physiology, and messenger RNA expression in schizophrenia cases and control individuals.The effects of a DTNBP1 gene variant on attention networks: an fMRI study Reduced dysbindin expression mediates N-methyl-D-aspartate receptor hypofunction and impaired working memory performance.Neuroplasticity signaling pathways linked to the pathophysiology of schizophrenia.DTNBP1 is associated with imaging phenotypes in schizophrenia.Can genomics help usher schizophrenia into the age of RDoC and DSM-6?The Schizophrenia-Related Protein Dysbindin-1A Is Degraded and Facilitates NF-Kappa B Activity in the Nucleus Dissecting the Syndrome of Schizophrenia: Progress toward Clinically Useful Biomarkers.Increased dysbindin-1B isoform expression in schizophrenia and its propensity in aggresome formation.Dysbindin-1 in dorsolateral prefrontal cortex of schizophrenia cases is reduced in an isoform-specific manner unrelated to dysbindin-1 mRNA expression.Realising stratified psychiatry using multidimensional signatures and trajectories.What have the genomics ever done for the psychoses?Controversies surrounding the diagnosis of schizophrenia and other psychoses.Genetic influence on the working memory circuitry: behavior, structure, function and extensions to illness.Can genetics inform the management of cognitive deficits in schizophrenia?Translating advances in the molecular basis of schizophrenia into novel cognitive treatment strategies.Dysregulation of Specialized Delay/Interference-Dependent Working Memory Following Loss of Dysbindin-1A in Schizophrenia-Related Phenotypes.Antipsychotic drugs attenuate aberrant DNA methylation of DTNBP1 (dysbindin) promoter in saliva and post-mortem brain of patients with schizophrenia and Psychotic bipolar disorder.COMT, BDNF, and DTNBP1 polymorphisms and cognitive functions in patients with brain tumors.Loss of dysbindin-1, a risk gene for schizophrenia, leads to impaired group 1 metabotropic glutamate receptor function in mice.Meta-analysis of genetic variation in DTNBP1 and general cognitive ability.Dysbindin modulates prefrontal cortical glutamatergic circuits and working memory function in mice.The effect of COMT, BDNF, 5-HTT, NRG1 and DTNBP1 genes on hippocampal and lateral ventricular volume in psychosis.

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DTNBP1 genotype influences cognitive decline in schizophrenia.

http://www.wikidata.org/entity/Q35688111

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DTNBP1 genotype influences cognitive decline in schizophrenia.

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DTNBP1 genotype influences cognitive decline in schizophrenia.

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DTNBP1 genotype influences cognitive decline in schizophrenia.

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DTNBP1 genotype influences cognitive decline in schizophrenia.

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Schizophrenia Research

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DTNBP1 genotype influences cognitive decline in schizophrenia.

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Anil K Malhotra

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Birgit Funke

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John A Bates

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Katherine E Burdick

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Raju Kucherlapati

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Terry E Goldberg

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P2860

Genetic variation in the 6p22.3 gene DTNBP1, the human ortholog of the mouse dysbindin gene, is associated with schizophrenia

Evidence of novel neuronal functions of dysbindin, a susceptibility gene for schizophrenia

Association of the DTNBP1 locus with schizophrenia in a U.S. population

Identification in 2 independent samples of a novel schizophrenia risk haplotype of the dystrobrevin binding protein gene (DTNBP1).

Genetic variation in DTNBP1 influences general cognitive ability.

An update on the genetics of schizophrenia.

Stability and course of neuropsychological deficits in schizophrenia.

Intelligence quotient and neuropsychological profiles in patients with schizophrenia and in normal volunteers.

Human dysbindin (DTNBP1) gene expression in normal brain and in schizophrenic prefrontal cortex and midbrain.

A population-based cohort study of premorbid intellectual, language, and behavioral functioning in patients with schizophrenia, schizoaffective disorder, and nonpsychotic bipolar disorder.

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