Actinonin, a meprin A inhibitor, protects the renal microcirculation during sepsis
about
Systemic and renal oxidative stress in the pathogenesis of hypertension: modulation of long-term control of arterial blood pressure by resveratrolADAM10 is the major sheddase responsible for the release of membrane-associated meprin A.Discovery of novel inhibitors of a disintegrin and metalloprotease 17 (ADAM17) using glycosylated and non-glycosylated substrates.Suppression of mitochondrial biogenesis through toll-like receptor 4-dependent mitogen-activated protein kinase kinase/extracellular signal-regulated kinase signaling in endotoxin-induced acute kidney injury.Pharmacologic targeting of sphingosine-1-phosphate receptor 1 improves the renal microcirculation during sepsis in the mouse.Hemodynamic changes in the kidney in a pediatric rat model of sepsis-induced acute kidney injury.Peptide deformylase inhibitor actinonin reduces celastrol's HSP70 induction while synergizing proliferation inhibition in tumor cellsRole of mitochondrial oxidants in an in vitro model of sepsis-induced renal injury.Resveratrol improves renal microcirculation, protects the tubular epithelium, and prolongs survival in a mouse model of sepsis-induced acute kidney injury.THE ENDOTHELIUM IN SEPSIS.Development of oxidative stress in the peritubular capillary microenvironment mediates sepsis-induced renal microcirculatory failure and acute kidney injury.The metalloproteases meprin α and meprin β: unique enzymes in inflammation, neurodegeneration, cancer and fibrosis.Adrenomedullin binding improves catecholamine responsiveness and kidney function in resuscitated murine septic shock.Meprin A metalloproteinase and its role in acute kidney injury.Targeted fibrillar nanocarbon RNAi treatment of acute kidney injury.Rolipram improves renal perfusion and function during sepsis in the mouse.Inactivation of renal mitochondrial respiratory complexes and manganese superoxide dismutase during sepsis: mitochondria-targeted antioxidant mitigates injuryExtracellular superoxide dismutase is necessary to maintain renal blood flow during sepsis development.Pharmacological targets in the renal peritubular microenvironment: implications for therapy for sepsis-induced acute kidney injury.MEP1A contributes to tumor progression and predicts poor clinical outcome in human hepatocellular carcinoma.Metalloproteinase meprin α regulates migration and invasion of human hepatocarcinoma cells and is a mediator of the oncoprotein Reptin.The metalloprotease meprin β generates amino terminal-truncated amyloid β peptide species.Renal ADAM10 and 17: Their Physiological and Medical Meanings
P2860
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P2860
Actinonin, a meprin A inhibitor, protects the renal microcirculation during sepsis
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Actinonin, a meprin A inhibitor, protects the renal microcirculation during sepsis
@ast
Actinonin, a meprin A inhibitor, protects the renal microcirculation during sepsis
@en
type
label
Actinonin, a meprin A inhibitor, protects the renal microcirculation during sepsis
@ast
Actinonin, a meprin A inhibitor, protects the renal microcirculation during sepsis
@en
prefLabel
Actinonin, a meprin A inhibitor, protects the renal microcirculation during sepsis
@ast
Actinonin, a meprin A inhibitor, protects the renal microcirculation during sepsis
@en
P2093
P2860
P1433
P1476
Actinonin, a meprin A inhibitor, protects the renal microcirculation during sepsis
@en
P2093
Christian Herzog
Gur P Kaushal
Neriman Gokden
Philip R Mayeux
P2860
P304
P356
10.1097/SHK.0B013E3181EC39CC
P577
2011-02-01T00:00:00Z