Suppression of spontaneous genome rearrangements in yeast DNA helicase mutants
about
An N-terminal acidic region of Sgs1 interacts with Rpa70 and recruits Rad53 kinase to stalled forks.Rad52 sumoylation prevents the toxicity of unproductive Rad51 filaments independently of the anti-recombinase Srs2Sgs1 and exo1 redundantly inhibit break-induced replication and de novo telomere addition at broken chromosome ends.PIF1 family DNA helicases suppress R-loop mediated genome instability at tRNA genes.Formation of complex and unstable chromosomal translocations in yeastSrs2: the "Odd-Job Man" in DNA repair.Whole-Genome Sequence and Variant Analysis of W303, a Widely-Used Strain of Saccharomyces cerevisiae.A genetic and structural study of genome rearrangements mediated by high copy repeat Ty1 elementsAn mre11 mutation that promotes telomere recombination and an efficient bypass of senescence.Differential genetic interactions between Sgs1, DNA-damage checkpoint components and DNA repair factors in the maintenance of chromosome stability.Yeast Assay Highlights the Intrinsic Genomic Instability of Human PML Intron 6 over Intron 3 and the Role of Replication Fork ProteinsUvrD controls the access of recombination proteins to blocked replication forksA Novel Rrm3 Function in Restricting DNA Replication via an Orc5-Binding Domain Is Genetically Separable from Rrm3 Function as an ATPase/Helicase in Facilitating Fork Progression.A new role for Rrm3 in repair of replication-born DNA breakage by sister chromatid recombination.Mapping genomic hotspots of DNA damage by a single-strand-DNA-compatible and strand-specific ChIP-seq method.Rrm3 protects the Saccharomyces cerevisiae genome from instability at nascent sites of retrotranspositionThe Saccharomyces cerevisiae Rad6 postreplication repair and Siz1/Srs2 homologous recombination-inhibiting pathways process DNA damage that arises in asf1 mutants.A saccharomyces cerevisiae RNase H2 interaction network functions to suppress genome instabilityThe Balance between Recombination Enzymes and Accessory Replicative Helicases in Facilitating Genome Duplication.A genetic network that suppresses genome rearrangements in Saccharomyces cerevisiae and contains defects in cancers.dNTP pools determine fork progression and origin usage under replication stress.RRM3 regulates epigenetic conversions in Saccharomyces cerevisiae in conjunction with Chromatin Assembly Factor I.Defects in DNA lesion bypass lead to spontaneous chromosomal rearrangements and increased cell death.Histone dosage regulates DNA damage sensitivity in a checkpoint-independent manner by the homologous recombination pathway.Chromosomal translocations caused by either pol32-dependent or pol32-independent triparental break-induced replication.
P2860
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P2860
Suppression of spontaneous genome rearrangements in yeast DNA helicase mutants
description
2006 nî lūn-bûn
@nan
2006年の論文
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2006年論文
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2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
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2006年論文
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2006年论文
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2006年论文
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2006年论文
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name
Suppression of spontaneous genome rearrangements in yeast DNA helicase mutants
@ast
Suppression of spontaneous genome rearrangements in yeast DNA helicase mutants
@en
type
label
Suppression of spontaneous genome rearrangements in yeast DNA helicase mutants
@ast
Suppression of spontaneous genome rearrangements in yeast DNA helicase mutants
@en
prefLabel
Suppression of spontaneous genome rearrangements in yeast DNA helicase mutants
@ast
Suppression of spontaneous genome rearrangements in yeast DNA helicase mutants
@en
P2860
P356
P1476
Suppression of spontaneous genome rearrangements in yeast DNA helicase mutants
@en
P2093
Kristina H Schmidt
Richard D Kolodner
P2860
P304
18196-18201
P356
10.1073/PNAS.0608566103
P407
P577
2006-11-17T00:00:00Z