dNTP pools determine fork progression and origin usage under replication stress.
about
The FHIT gene product: tumor suppressor and genome "caretaker"Rescuing stalled or damaged replication forksRad53-Mediated Regulation of Rrm3 and Pif1 DNA Helicases Contributes to Prevention of Aberrant Fork Transitions under Replication Stress.Sen1p contributes to genomic integrity by regulating expression of ribonucleotide reductase 1 (RNR1) in Saccharomyces cerevisiae.ATR-like kinase Mec1 facilitates both chromatin accessibility at DNA replication forks and replication fork progression during replication stress.Replication fork instability and the consequences of fork collisions from rereplication(Ubi)quitin' the h2bit: recent insights into the roles of H2B ubiquitylation in DNA replication and transcriptionReplication stress in Mammalian cells and its consequences for mitosisGenetic instability in budding and fission yeast-sources and mechanismsAPOBEC3A and APOBEC3B Preferentially Deaminate the Lagging Strand Template during DNA ReplicationDepletion of cellular iron by curcumin leads to alteration in histone acetylation and degradation of Sml1p in Saccharomyces cerevisiaeThe mutation spectrum in genomic late replication domains shapes mammalian GC contentYeast DNA polymerase ζ maintains consistent activity and mutagenicity across a wide range of physiological dNTP concentrationsDomain within the helicase subunit Mcm4 integrates multiple kinase signals to control DNA replication initiation and fork progression.MYC and the control of DNA replication.DNA replication and transcription programs respond to the same chromatin cues.A mammalian-like DNA damage response of fission yeast to nucleoside analogsCauses and consequences of replication stress.SAMHD1 prevents autoimmunity by maintaining genome stability.Continued DNA synthesis in replication checkpoint mutants leads to fork collapse.Rescue from replication stress during mitosis.Single molecule analysis of Trypanosoma brucei DNA replication dynamics.Replication fork integrity and intra-S phase checkpoint suppress gene amplification.Whole genome RNAi screens reveal a critical role of REV3 in coping with replication stress.Increased Rrm2 gene dosage reduces fragile site breakage and prolongs survival of ATR mutant mice.H2B mono-ubiquitylation facilitates fork stalling and recovery during replication stress by coordinating Rad53 activation and chromatin assembly.Endogenous DNA replication stress results in expansion of dNTP pools and a mutator phenotypeSinglet Oxygen-Mediated Oxidation during UVA Radiation Alters the Dynamic of Genomic DNA Replication.Slow Replication Fork Velocity of Homologous Recombination-Defective Cells Results from Endogenous Oxidative StressA Checkpoint-Related Function of the MCM Replicative Helicase Is Required to Avert Accumulation of RNA:DNA Hybrids during S-phase and Ensuing DSBs during G2/MRtt107 Is a Multi-functional Scaffold Supporting Replication Progression with Partner SUMO and Ubiquitin Ligases.A Novel Rrm3 Function in Restricting DNA Replication via an Orc5-Binding Domain Is Genetically Separable from Rrm3 Function as an ATPase/Helicase in Facilitating Fork Progression.Mutations in the Non-Catalytic Subunit Dpb2 of DNA Polymerase Epsilon Affect the Nrm1 Branch of the DNA Replication Checkpoint.The human lagging strand DNA polymerase δ holoenzyme is distributive.Nucleoside salvage pathway kinases regulate hematopoiesis by linking nucleotide metabolism with replication stress.S phase block following MEC1ATR inactivation occurs without severe dNTP depletion.Mec1, INO80, and the PAF1 complex cooperate to limit transcription replication conflicts through RNAPII removal during replication stressConcerted activities of Mcm4, Sld3, and Dbf4 in control of origin activation and DNA replication fork progressionMammalian RAD51 paralogs protect nascent DNA at stalled forks and mediate replication restart.Single-molecule analysis of DNA replication reveals novel features in the divergent eukaryotes Leishmania and Trypanosoma brucei versus mammalian cells.
P2860
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P2860
dNTP pools determine fork progression and origin usage under replication stress.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
dNTP pools determine fork progression and origin usage under replication stress.
@en
type
label
dNTP pools determine fork progression and origin usage under replication stress.
@en
prefLabel
dNTP pools determine fork progression and origin usage under replication stress.
@en
P2093
P2860
P356
P1433
P1476
dNTP pools determine fork progression and origin usage under replication stress.
@en
P2093
Andrea Keszthelyi
Armelle Lengronne
Jérôme Poli
Laure Crabbé
Olga Tsaponina
Véronique Pantesco
P2860
P304
P356
10.1038/EMBOJ.2011.470
P407
P577
2012-01-10T00:00:00Z