Twist contributes to hormone resistance in breast cancer by downregulating estrogen receptor-α. - Wikidata - wikimedia - TriplyDB

Twist contributes to hormone resistance in breast cancer by downregulating estrogen receptor-α.

Twist contributes to hormone resistance in breast cancer by downregulating estrogen receptor-α.

http://www.wikidata.org/entity/Q35748639

about

The relationship between vasculogenic mimicry and epithelial-mesenchymal transitions Novel Molecular Markers for Breast Cancer Epithelial-Mesenchymal Transition (EMT) and Regulation of EMT Factors by Steroid Nuclear Receptors in Breast Cancer: A Review and in Silico Investigation Role of EMT in Metastasis and Therapy Resistance The network of pluripotency, epithelial-mesenchymal transition, and prognosis of breast cancer Thymoquinone inhibits cancer metastasis by downregulating TWIST1 expression to reduce epithelial to mesenchymal transition Twist: a molecular target in cancer therapeutics Fulvestrant induces resistance by modulating GPER and CDK6 expression: implication of methyltransferases, deacetylases and the hSWI/SNF chromatin remodelling complex WP1130 increases cisplatin sensitivity through inhibition of usp9x in estrogen receptor-negative breast cancer cells.Z-ligustilide restores tamoxifen sensitivity of ERa negative breast cancer cells by reversing MTA1/IFI16/HDACs complex mediated epigenetic repression of ERa ZEB1 induces ER-α promoter hypermethylation and confers antiestrogen resistance in breast cancer.Co-expression of putative stemness and epithelial-to-mesenchymal transition markers on single circulating tumour cells from patients with early and metastatic breast cancer δEF1 down-regulates ER-α expression and confers tamoxifen resistance in breast cancer Reduced expression of miR-200 family members contributes to antiestrogen resistance in LY2 human breast cancer cells TRIM29 suppresses TWIST1 and invasive breast cancer behavior.RNA-based TWIST1 inhibition via dendrimer complex to reduce breast cancer cell metastasis.Molecular characterization and targeted therapeutic approaches in breast cancer Over-Expressed Twist Associates with Markers of Epithelial Mesenchymal Transition and Predicts Poor Prognosis in Breast Cancers via ERK and Akt Activation.Targeting DDX3 with a small molecule inhibitor for lung cancer therapy.TWIST represses estrogen receptor-alpha expression by recruiting the NuRD protein complex in breast cancer cells.MDC1 Enhances Estrogen Receptor-mediated Transactivation and Contributes to Breast Cancer Suppression.An 8-gene mRNA expression profile in circulating tumor cells predicts response to aromatase inhibitors in metastatic breast cancer patients Mesoporous silica nanoparticle delivery of chemically modified siRNA against TWIST1 leads to reduced tumor burden.Altered death receptor signaling promotes epithelial-to-mesenchymal transition and acquired chemoresistance.Genomic pathways modulated by Twist in breast cancer.NF-kappaB Is Involved in the Regulation of EMT Genes in Breast Cancer Cells.Combination Twist1 and CA15-3 in axillary lymph nodes for breast cancer prognosis.DLX4 upregulates TWIST and enhances tumor migration, invasion and metastasis TWIST-1 promotes cell growth, drug resistance and progenitor clonogenic capacities in myeloid leukemia and is a novel poor prognostic factor in acute myeloid leukemia A poxviral-based cancer vaccine the transcription factor twist inhibits primary tumor growth and metastases in a model of metastatic breast cancer and improves survival in a spontaneous prostate cancer model Identification of the DEAD box RNA helicase DDX3 as a therapeutic target in colorectal cancer Vaccine-mediated immunotherapy directed against a transcription factor driving the metastatic process.Metformin represses cancer cells via alternate pathways in N-cadherin expressing vs. N-cadherin deficient cells NZ51, a ring-expanded nucleoside analog, inhibits motility and viability of breast cancer cells by targeting the RNA helicase DDX3.Ibandronate increases the expression of the pro-apoptotic gene FAS by epigenetic mechanisms in tumor cells Endocrine resistance in breast cancer: from cellular signaling pathways to epigenetic mechanisms.Prognostic Value of EMT-inducing Transcription Factors (EMT-TFs) in Metastatic Breast Cancer: A Systematic Review and Meta-analysis.Twist promotes angiogenesis in pancreatic cancer by targeting miR-497/VEGFA axis.Twist1 promotes breast cancer invasion and metastasis by silencing Foxa1 expression.Luteolin suppresses gastric cancer progression by reversing epithelial-mesenchymal transition via suppression of the Notch signaling pathway.

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Twist contributes to hormone resistance in breast cancer by downregulating estrogen receptor-α.

http://www.wikidata.org/entity/Q35748639

description

2011 nî lūn-bûn

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Twist contributes to hormone r ...... egulating estrogen receptor-α.

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Twist contributes to hormone r ...... egulating estrogen receptor-α.

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Twist contributes to hormone r ...... egulating estrogen receptor-α.

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D Artemov

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P2860

Saethre-Chotzen mutations cause TWIST protein degradation or impaired nuclear location

A new DNA binding and dimerization motif in immunoglobulin enhancer binding, daughterless, MyoD, and myc proteins

HOXA5-twist interaction alters p53 homeostasis in breast cancer cells

Twist modulates breast cancer stem cells by transcriptional regulation of CD24 expression

Phosphatidylinositol 3-kinase/AKT-mediated activation of estrogen receptor alpha: a new model for anti-estrogen resistance

Repression of muscle-specific gene activation by the murine Twist protein

A twist code determines the onset of osteoblast differentiation

Synergy of demethylation and histone deacetylase inhibition in the re-expression of genes silenced in cancer

Selective estrogen receptor modulators: structure, function, and clinical use.

Steroid hormone receptors in breast cancer management.

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