Experimental autoimmune encephalomyelitis (EAE) in CCR2(-/-) mice: susceptibility in multiple strains
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Neuroprotection and remyelination after autoimmune demyelination in mice that inducibly overexpress CXCL1Involvement of beta-chemokines in the development of inflammatory demyelinationThe chemokine CCL2 modulates Ca2+ dynamics and electrophysiological properties of cultured cerebellar Purkinje neuronsSelective chemokine receptor usage by central nervous system myeloid cells in CCR2-red fluorescent protein knock-in mice.Differential contribution of immune effector mechanisms to cortical demyelination in multiple sclerosis.Exogenous fms-like tyrosine kinase 3 ligand overrides brain immune privilege and facilitates recognition of a neo-antigen without causing autoimmune neuropathologyRole of CCR2 in inflammatory conditions of the central nervous system.Possible association between expression of chemokine receptor-2 (CCR2) and amyotrophic lateral sclerosis (ALS) patients of North India.T-cell properties determine disease site, clinical presentation, and cellular pathology of experimental autoimmune encephalomyelitisReversible demyelination, blood-brain barrier breakdown, and pronounced neutrophil recruitment induced by chronic IL-1 expression in the brain.T lymphocytes do not directly mediate the protective effect of estrogen on experimental autoimmune encephalomyelitisDeficient CX3CR1 signaling promotes recovery after mouse spinal cord injury by limiting the recruitment and activation of Ly6Clo/iNOS+ macrophages.Chronic pelvic allodynia is mediated by CCL2 through mast cells in an experimental autoimmune cystitis modelSubstrain differences reveal novel disease-modifying gene candidates that alter the clinical course of a rodent model of multiple sclerosis.CCR2-dependent dendritic cell accumulation in the central nervous system during early effector experimental autoimmune encephalomyelitis is essential for effector T cell restimulation in situ and disease progressionDiscovery, optimization, and pharmacological characterization of novel heteroaroylphenylureas antagonists of C-C chemokine ligand 2 functionBioenergetics of the spinal cord in experimental autoimmune encephalitis of rats.The Role and Potential Therapeutic Application of Myeloid-Derived Suppressor Cells in Allo- and AutoimmunityCC chemokine receptor 4 is required for experimental autoimmune encephalomyelitis by regulating GM-CSF and IL-23 production in dendritic cells.The complex role of the chemokine receptor CCR2 in collagen-induced arthritis: implications for therapeutic targeting of CCR2 in rheumatoid arthritis.Chemokine receptors: attractive targets for drug discovery.CCR2 defines in vivo development and homing of IL-23-driven GM-CSF-producing Th17 cellsEndogenous adaptation to low oxygen modulates T-cell regulatory pathways in EAE.Chemokines and chemokine receptors in multiple sclerosis. Potential targets for new therapies.Multiple sclerosis therapy: an update on recently finished trials.Peripheral blood mononuclear cell infiltration and neuroinflammation in the HexB-/- mouse model of neurodegeneration.G protein-coupled receptors as therapeutic targets for multiple sclerosis.On the occurrence of hypomyelination in a transgenic mouse model: a consequence of the myelin basic protein promoter?Chemokines and chemokine receptors: standing at the crossroads of immunobiology and neurobiology.Mice lacking natural killer T cells are more susceptible to metabolic alterations following high fat diet feeding.High-resolution intravital imaging reveals that blood-derived macrophages but not resident microglia facilitate secondary axonal dieback in traumatic spinal cord injury.GM-CSF is not essential for experimental autoimmune encephalomyelitis but promotes brain-targeted disease.Therapeutic approaches to multiple sclerosis: an update on failed, interrupted, or inconclusive trials of immunomodulatory treatment strategies.The blood-brain barrier, chemokines and multiple sclerosisMDSC in autoimmunity.Insights into Myeloid-Derived Suppressor Cells in Inflammatory Diseases.Microglia Versus Myeloid Cell Nomenclature during Brain InflammationRegulator of G-protein Signaling (RGS)1 and RGS10 Proteins as Potential Drug Targets for Neuroinflammatory and Neurodegenerative Diseases.Cytokine networks in neuroinflammation.CC chemokine receptor (CCR)-2 prevents arthritis development following infection by Mycobacterium avium.
P2860
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P2860
Experimental autoimmune encephalomyelitis (EAE) in CCR2(-/-) mice: susceptibility in multiple strains
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Experimental autoimmune enceph ...... eptibility in multiple strains
@ast
Experimental autoimmune enceph ...... eptibility in multiple strains
@en
type
label
Experimental autoimmune enceph ...... eptibility in multiple strains
@ast
Experimental autoimmune enceph ...... eptibility in multiple strains
@en
prefLabel
Experimental autoimmune enceph ...... eptibility in multiple strains
@ast
Experimental autoimmune enceph ...... eptibility in multiple strains
@en
P2093
P2860
P1476
Experimental autoimmune enceph ...... eptibility in multiple strains
@en
P2093
Barbara Cannella
Cedric S Raine
David Pitt
Stefanie Gaupp
William A Kuziel
P2860
P304
P356
10.1016/S0002-9440(10)63805-9
P407
P577
2003-01-01T00:00:00Z