Dual-Specificity Phosphatase 1 and Tristetraprolin Cooperate To Regulate Macrophage Responses to Lipopolysaccharide
about
Gene Expression Control by Glucocorticoid Receptors during Innate Immune ResponsesMitogen-Activated Protein Kinases and Mitogen Kinase Phosphatase 1: A Critical Interplay in Macrophage BiologyThe control of inflammation via the phosphorylation and dephosphorylation of tristetraprolin: a tale of two phosphatasesTreatment of inflammatory arthritis via targeting of tristetraprolin, a master regulator of pro-inflammatory gene expression.Macrophage responses to lipopolysaccharide are modulated by a feedback loop involving prostaglandin E2, dual specificity phosphatase 1 and tristetraprolin.Dominant Suppression of Inflammation via Targeted Mutation of the mRNA Destabilizing Protein Tristetraprolin.Negative Feed-forward Control of Tumor Necrosis Factor (TNF) by Tristetraprolin (ZFP36) Is Limited by the Mitogen-activated Protein Kinase Phosphatase, Dual-specificity Phosphatase 1 (DUSP1): IMPLICATIONS FOR REGULATION BY GLUCOCORTICOIDS.The RNA-binding protein TTP is a global post-transcriptional regulator of feedback control in inflammationRNA binding proteins as regulators of immune cell biology.An Ancient Family of RNA-Binding Proteins: Still Important!Glucocorticoid and cytokine crosstalk: Feedback, feedforward, and co-regulatory interactions determine repression or resistance.Mitogen-activated protein kinase phosphatase 1 (MKP-1) in macrophage biology and cardiovascular disease. A redox-regulated master controller of monocyte function and macrophage phenotype.DUSP1 Maintains IRF1 and Leads to Increased Expression of IRF1-dependent Genes: A MECHANISM PROMOTING GLUCOCORTICOID INSENSITIVITY.Beta Interferon Production Is Regulated by p38 Mitogen-Activated Protein Kinase in Macrophages via both MSK1/2- and Tristetraprolin-Dependent Pathways.Role and regulation of MKP-1 in airway inflammation.Gain-of-Function Mutation of Tristetraprolin Impairs Negative Feedback Control of Macrophages In Vitro yet Has Overwhelmingly Anti-Inflammatory Consequences In Vivo.DUSP1 regulates apoptosis and cell migration, but not the JIP1-protected cytokine response, during Respiratory Syncytial Virus and Sendai Virus infection.The role of RNA-binding protein tristetraprolin in cancer and immunity.Roflumilast N-Oxide in Combination with Formoterol Enhances the Antiinflammatory Effect of Dexamethasone in Airway Smooth Muscle Cells.A Time- and Compartment-Specific Activation of Lung Macrophages in Hypoxic Pulmonary Hypertension.Corticosteroid-Induced MKP-1 Represses Pro-Inflammatory Cytokine Secretion by Enhancing Activity of Tristetraprolin (TTP) in ASM Cells.
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Dual-Specificity Phosphatase 1 and Tristetraprolin Cooperate To Regulate Macrophage Responses to Lipopolysaccharide
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Dual-Specificity Phosphatase 1 ...... esponses to Lipopolysaccharide
@ast
Dual-Specificity Phosphatase 1 ...... esponses to Lipopolysaccharide
@en
type
label
Dual-Specificity Phosphatase 1 ...... esponses to Lipopolysaccharide
@ast
Dual-Specificity Phosphatase 1 ...... esponses to Lipopolysaccharide
@en
prefLabel
Dual-Specificity Phosphatase 1 ...... esponses to Lipopolysaccharide
@ast
Dual-Specificity Phosphatase 1 ...... esponses to Lipopolysaccharide
@en
P2093
P2860
P50
P356
P1476
Dual-Specificity Phosphatase 1 ...... esponses to Lipopolysaccharide
@en
P2093
Christopher D Buckley
Dalya R Rosner
Helen E Cunliffe
Jeremy Saklatvala
Jonathan L Dean
Tim Smallie
P2860
P304
P356
10.4049/JIMMUNOL.1402830
P407
P577
2015-05-27T00:00:00Z