Activation of Wnt signaling bypasses the requirement for RTK/Ras signaling during C. elegans vulval induction.
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Antagonism of LIN-17/Frizzled and LIN-18/Ryk in nematode vulva induction reveals evolutionary alterations in core developmental pathwaysEGF signal propagation during C. elegans vulval development mediated by ROM-1 rhomboidReproductive Aging Drives Protein Accumulation in the Uterus and Limits Lifespan in C. elegansNeurons refine the Caenorhabditis elegans body plan by directing axial patterning by WntsConserved nutrient sensor O-GlcNAc transferase is integral to C. elegans pathogen-specific immunityThe Mediator Kinase Module Restrains Epidermal Growth Factor Receptor Signaling and Represses Vulval Cell Fate Specification in Caenorhabditis elegansWnt signal from multiple tissues and lin-3/EGF signal from the gonad maintain vulval precursor cell competence in Caenorhabditis elegans.Loss-of-function of β-catenin bar-1 slows development and activates the Wnt pathway in Caenorhabditis elegansUse of an activated beta-catenin to identify Wnt pathway target genes in caenorhabditis elegans, including a subset of collagen genes expressed in late larval development.The Caenorhabditis elegans GATA factor ELT-1 works through the cell proliferation regulator BRO-1 and the Fusogen EFF-1 to maintain the seam stem-like fate.A gain-of-function allele of cbp-1, the Caenorhabditis elegans ortholog of the mammalian CBP/p300 gene, causes an increase in histone acetyltransferase activity and antagonism of activated Ras.Repression of Wnt signaling by a Fer-type nonreceptor tyrosine kinase.Wnt signaling controls the stem cell-like asymmetric division of the epithelial seam cells during C. elegans larval development.The CRAL/TRIO and GOLD domain protein CGR-1 promotes induction of vulval cell fates in Caenorhabditis elegans and interacts genetically with the Ras signaling pathway.lin-35/Rb cooperates with the SWI/SNF complex to control Caenorhabditis elegans larval developmentCanonical and non-canonical Wnt signaling pathways in Caenorhabditis elegans: variations on a common signaling theme.Distinct DNA binding sites contribute to the TCF transcriptional switch in C. elegans and Drosophila.sli-3 negatively regulates the LET-23/epidermal growth factor receptor-mediated vulval induction pathway in Caenorhabditis elegans.C. elegans EOR-1/PLZF and EOR-2 positively regulate Ras and Wnt signaling and function redundantly with LIN-25 and the SUR-2 Mediator componentLIN-39/Hox triggers cell division and represses EFF-1/fusogen-dependent vulval cell fusion.LIN-61, one of two Caenorhabditis elegans malignant-brain-tumor-repeat-containing proteins, acts with the DRM and NuRD-like protein complexes in vulval development but not in certain other biological processes.Re-programming of C. elegans male epidermal precursor fates by Wnt, Hox, and LIN-12/Notch activities.Identification of Wnt Pathway Target Genes Regulating the Division and Differentiation of Larval Seam Cells and Vulval Precursor Cells in Caenorhabditis elegansUNC-4 antagonizes Wnt signaling to regulate synaptic choice in the C. elegans motor circuit.The C. elegans embryonic fate specification factor EGL-18 (GATA) is reutilized downstream of Wnt signaling to maintain a population of larval progenitor cells.β-catenin-dependent Wnt signaling in C. elegans: teaching an old dog a new trickFoxm1 mediates cross talk between Kras/mitogen-activated protein kinase and canonical Wnt pathways during development of respiratory epitheliumGenetic control of vulval development in Caenorhabditis briggsaeC. elegans GATA factors EGL-18 and ELT-6 function downstream of Wnt signaling to maintain the progenitor fate during larval asymmetric divisions of the seam cellsSmall-molecule inhibition of Wnt signaling through activation of casein kinase 1αThe Axin-like protein PRY-1 is a negative regulator of a canonical Wnt pathway in C. elegans.The C. elegans homolog of the mammalian tumor suppressor Dep-1/Scc1 inhibits EGFR signaling to regulate binary cell fate decisions.A network model for the specification of vulval precursor cells and cell fusion control in Caenorhabditis elegansCryptic genetic variation uncovers evolution of environmentally sensitive parameters in Caenorhabditis vulval development.Huwe1-mediated ubiquitylation of dishevelled defines a negative feedback loop in the Wnt signaling pathway.A systems toxicology approach reveals the Wnt-MAPK crosstalk pathway mediated reproductive failure in Caenorhabditis elegans exposed to graphene oxide (GO) but not to reduced graphene oxide (rGO).
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P2860
Activation of Wnt signaling bypasses the requirement for RTK/Ras signaling during C. elegans vulval induction.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
2002年论文
@zh
2002年论文
@zh-cn
name
Activation of Wnt signaling by ...... g C. elegans vulval induction.
@ast
Activation of Wnt signaling by ...... g C. elegans vulval induction.
@en
type
label
Activation of Wnt signaling by ...... g C. elegans vulval induction.
@ast
Activation of Wnt signaling by ...... g C. elegans vulval induction.
@en
prefLabel
Activation of Wnt signaling by ...... g C. elegans vulval induction.
@ast
Activation of Wnt signaling by ...... g C. elegans vulval induction.
@en
P2860
P356
P1433
P1476
Activation of Wnt signaling by ...... g C. elegans vulval induction.
@en
P2093
Hendrik C Korswagen
Julie E Gleason
P2860
P304
P356
10.1101/GAD.981602
P577
2002-05-01T00:00:00Z