Oxidative capacity, lipotoxicity, and mitochondrial damage in type 2 diabetes.
about
Mitochondrial uncoupling proteins and energy metabolismMitochondrial dysfunction in the type 2 diabetic heart is associated with alterations in spatially distinct mitochondrial proteomesReactive oxygen species in inflammation and tissue injuryMitochondrial targeted coenzyme Q, superoxide, and fuel selectivity in endothelial cellsTetramethylpyrazine ameliorates high glucose-induced endothelial dysfunction by increasing mitochondrial biogenesis.UCP3 in muscle wasting, a role in modulating lipotoxicity?Reduced heart size and increased myocardial fuel substrate oxidation in ACC2 mutant miceHDAC4 regulates muscle fiber type-specific gene expression programs.S-glutathionylation: from molecular mechanisms to health outcomesRestoration of muscle mitochondrial function and metabolic flexibility in type 2 diabetes by exercise training is paralleled by increased myocellular fat storage and improved insulin sensitivityHigh-throughput screening for fatty acid uptake inhibitors in humanized yeast identifies atypical antipsychotic drugs that cause dyslipidemias.UCP-2 and UCP-3 proteins are differentially regulated in pancreatic beta-cells.Alterations in fatty acid utilization and an impaired antioxidant defense mechanism are early events in podocyte injury: a proteomic analysis.Mitochondrial dysfunction in diabetes: from molecular mechanisms to functional significance and therapeutic opportunities.Acute and chronic changes in rat soleus muscle after high-fat high-sucrose diet.Acylcarnitines activate proinflammatory signaling pathways.Therapeutic interventions and oxidative stress in diabetesCardiorespiratory fitness and insulin sensitivity in overweight or obese subjects may be linked through intrahepatic lipid content.p53: exercise capacity and metabolism.Improvement of mechanical heart function by trimetazidine in db/db mice.Modulation of renal-specific oxidoreductase/myo-inositol oxygenase by high-glucose ambience.Different effects of oleate vs. palmitate on mitochondrial function, apoptosis, and insulin signaling in L6 skeletal muscle cells: role of oxidative stressEndurance training increases skeletal muscle LKB1 and PGC-1alpha protein abundance: effects of time and intensity.FTO is increased in muscle during type 2 diabetes, and its overexpression in myotubes alters insulin signaling, enhances lipogenesis and ROS production, and induces mitochondrial dysfunction.Palmitoleic acid reduces intramuscular lipid and restores insulin sensitivity in obese sheep.Astaxanthin supplementation delays physical exhaustion and prevents redox imbalances in plasma and soleus muscles of Wistar ratsLow intrinsic running capacity is associated with reduced skeletal muscle substrate oxidation and lower mitochondrial content in white skeletal muscle.Hepatic mitochondrial and ER stress induced by defective PPARα signaling in the pathogenesis of hepatic steatosis.Adipose tissue content, muscle performance and physical function in obese adults with type 2 diabetes mellitus and peripheral neuropathyMyocyte enhancer factor 2C function in skeletal muscle is required for normal growth and glucose metabolism in mice.Intramuscular triacylglycerol and insulin resistance: guilty as charged or wrongly accused?Long-chain acylcarnitines activate cell stress and myokine release in C2C12 myotubes: calcium-dependent and -independent effects.Increased oxidative stress is associated with balanced increases in hepatocyte apoptosis and proliferation in glycerol-3-phosphate acyltransferase-1 deficient mice.Mechanisms underlying skeletal muscle insulin resistance induced by fatty acids: importance of the mitochondrial functionHigh-fat diet accelerates progression of osteoarthritis after meniscal/ligamentous injuryThe Subtle Balance between Lipolysis and Lipogenesis: A Critical Point in Metabolic Homeostasis.A review of thiazolidinediones and metformin in the treatment of type 2 diabetes with focus on cardiovascular complications.Skeletal intramyocellular lipid metabolism and insulin resistance.myo-Inositol Oxygenase Overexpression Accentuates Generation of Reactive Oxygen Species and Exacerbates Cellular Injury following High Glucose Ambience: A NEW MECHANISM RELEVANT TO THE PATHOGENESIS OF DIABETIC NEPHROPATHYMitochondrial function, fibre types and ageing: new insights from human muscle in vivo.
P2860
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P2860
Oxidative capacity, lipotoxicity, and mitochondrial damage in type 2 diabetes.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Oxidative capacity, lipotoxicity, and mitochondrial damage in type 2 diabetes.
@ast
Oxidative capacity, lipotoxicity, and mitochondrial damage in type 2 diabetes.
@en
type
label
Oxidative capacity, lipotoxicity, and mitochondrial damage in type 2 diabetes.
@ast
Oxidative capacity, lipotoxicity, and mitochondrial damage in type 2 diabetes.
@en
prefLabel
Oxidative capacity, lipotoxicity, and mitochondrial damage in type 2 diabetes.
@ast
Oxidative capacity, lipotoxicity, and mitochondrial damage in type 2 diabetes.
@en
P1433
P1476
Oxidative capacity, lipotoxicity, and mitochondrial damage in type 2 diabetes.
@en
P304
P356
10.2337/DIABETES.53.6.1412
P407
P577
2004-06-01T00:00:00Z