Molecular determinants of altered contractility in heart failure.
about
Phosphorylation of the cAMP-dependent protein kinase (PKA) regulatory subunit modulates PKA-AKAP interaction, substrate phosphorylation, and calcium signaling in cardiac cellsRyanodine receptor/calcium release channel PKA phosphorylation: a critical mediator of heart failure progression.The pharmacology of nitroxyl (HNO) and its therapeutic potential: not just the Janus face of NOAKAP-scaffolding proteins and regulation of cardiac physiology.Central-acting therapeutics alleviate respiratory weakness caused by heart failure-induced ventilatory overdrive.Decreased cardiac L-type Ca²⁺ channel activity induces hypertrophy and heart failure in mice.CPU228, a derivative of dofetilide, relieves cardiac dysfunction by normalizing FKBP12.6, NADPH oxidase and protein kinase C epsilon in the myocardium.Effect of the endothelin receptor antagonist CPU0213, and its modulation by rifampin, on cardiac and vascular tissue following chronic isoproterenol treatment.Transcriptome Profiling Reveals PHLDA1 as a Novel Molecular Marker for Ischemic Cardiomyopathy.
P2860
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P2860
Molecular determinants of altered contractility in heart failure.
description
2004 nî lūn-bûn
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2004年の論文
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2004年論文
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2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
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2004年论文
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2004年论文
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name
Molecular determinants of altered contractility in heart failure.
@ast
Molecular determinants of altered contractility in heart failure.
@en
type
label
Molecular determinants of altered contractility in heart failure.
@ast
Molecular determinants of altered contractility in heart failure.
@en
prefLabel
Molecular determinants of altered contractility in heart failure.
@ast
Molecular determinants of altered contractility in heart failure.
@en
P2860
P1433
P1476
Molecular determinants of altered contractility in heart failure.
@en
P2093
Andrew R Marks
P2860
P356
10.1080/17431380410032481
P407
P478
36 Suppl 1
P577
2004-01-01T00:00:00Z