Experimental diabetes causes breakdown of the blood-retina barrier by a mechanism involving tyrosine nitration and increases in expression of vascular endothelial growth factor and urokinase plasminogen activator receptor.
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Nitric oxide and peroxynitrite in health and diseaseRole of nitrosative stress and peroxynitrite in the pathogenesis of diabetic complications. Emerging new therapeutical strategiesFunctions of Müller cell-derived vascular endothelial growth factor in diabetic retinopathyContribution of microglia-mediated neuroinflammation to retinal degenerative diseasesMolecular mechanisms of diabetic retinopathy, general preventive strategies, and novel therapeutic targetsPathophysiological Role of Peroxynitrite Induced DNA Damage in Human Diseases: A Special Focus on Poly(ADP-ribose) Polymerase (PARP)Pulmonary exposure to carbon black nanoparticles and vascular effectsCalcium dobesilate inhibits the alterations in tight junction proteins and leukocyte adhesion to retinal endothelial cells induced by diabetesDifferential roles of uPAR in peritoneal ovarian carcinomatosisCannabidiol As a Putative Novel Therapy for Diabetic Retinopathy: A Postulated Mechanism of Action as an Entry Point for Biomarker-Guided Clinical Development.Endoplasmic reticulum stress and diabetic retinopathy.Diabetic retinopathy: current management and experimental therapeutic targets.Adaptive cerebral neovascularization in a model of type 2 diabetes: relevance to focal cerebral ischemia.Vascular dysfunction in retinopathy-an emerging role for arginaseRole of NADPH oxidase and Stat3 in statin-mediated protection against diabetic retinopathy.NAD(P)H oxidase-dependent regulation of CCL2 production during retinal inflammation.STAT1-mediated Bim expression promotes the apoptosis of retinal pericytes under high glucose conditions.Antipermeability function of PEDF involves blockade of the MAP kinase/GSK/beta-catenin signaling pathway and uPAR expression.Cannabidiol protects retinal neurons by preserving glutamine synthetase activity in diabetes.The pathogenesis of early retinal changes of diabetic retinopathy.Vascular endothelial growth factor and diabetic retinopathy: pathophysiological mechanisms and treatment perspectives.Protection of blood retinal barrier and systemic vasculature by insulin-like growth factor binding protein-3.Effects of asymmetric dimethylarginine on bovine retinal capillary endothelial cell proliferation, reactive oxygen species production, permeability, intercellular adhesion molecule-1, and occludin expression.Arginase in retinopathy.CD40 promotes the development of early diabetic retinopathy in miceDiabetes-induced superoxide anion and breakdown of the blood-retinal barrier: role of the VEGF/uPAR pathway.Neuroprotective and blood-retinal barrier-preserving effects of cannabidiol in experimental diabetesNeurovascular interaction and the pathophysiology of diabetic retinopathyRequirement of NOX2 expression in both retina and bone marrow for diabetes-induced retinal vascular injury.Inhibition of NAD(P)H oxidase activity blocks vascular endothelial growth factor overexpression and neovascularization during ischemic retinopathy.The role of N-methyl-D-aspartate receptor activation in homocysteine-induced death of retinal ganglion cellsMetformin treatment in the period after stroke prevents nitrative stress and restores angiogenic signaling in the brain in diabetes.Regression of early diabetic macular oedema is associated with prevention of dark adaptationNeuroprotective effect of (-)Delta9-tetrahydrocannabinol and cannabidiol in N-methyl-D-aspartate-induced retinal neurotoxicity: involvement of peroxynitritePeroxynitrite upregulates angiogenic factors VEGF-A, BFGF, and HIF-1α in human corneal limbal epithelial cells.Tetramethylpyrazine Protects Retinal Capillary Endothelial Cells (TR-iBRB2) against IL-1β-Induced Nitrative/Oxidative StressRole of peroxynitrite in the pathogenesis of cardiovascular complications of diabetes.Nitrosative stress plays an important role in Wnt pathway activation in diabetic retinopathyIntravitreal administration of multipotent mesenchymal stromal cells triggers a cytoprotective microenvironment in the retina of diabetic mice.Potential therapeutic effects of pigment epithelium-derived factor for treatment of diabetic retinopathy
P2860
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P2860
Experimental diabetes causes breakdown of the blood-retina barrier by a mechanism involving tyrosine nitration and increases in expression of vascular endothelial growth factor and urokinase plasminogen activator receptor.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Experimental diabetes causes b ...... lasminogen activator receptor.
@ast
Experimental diabetes causes b ...... lasminogen activator receptor.
@en
type
label
Experimental diabetes causes b ...... lasminogen activator receptor.
@ast
Experimental diabetes causes b ...... lasminogen activator receptor.
@en
prefLabel
Experimental diabetes causes b ...... lasminogen activator receptor.
@ast
Experimental diabetes causes b ...... lasminogen activator receptor.
@en
P2093
P2860
P1476
Experimental diabetes causes b ...... lasminogen activator receptor.
@en
P2093
Azza B El-Remessy
Gamal Abou-Mohamed
M Ali Behzadian
Robert W Caldwell
Ruth B Caldwell
Telina Franklin
P2860
P304
P356
10.1016/S0002-9440(10)64332-5
P407
P577
2003-06-01T00:00:00Z