c-Myc is essential for vasculogenesis and angiogenesis during development and tumor progression - Wikidata - wikimedia - TriplyDB

c-Myc is essential for vasculogenesis and angiogenesis during development and tumor progression

c-Myc is essential for vasculogenesis and angiogenesis during development and tumor progression

http://www.wikidata.org/entity/Q35804203

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c-Myc augments gamma irradiation-induced apoptosis by suppressing Bcl-XL Novel c-MYC target genes mediate differential effects on cell proliferation and migration Hypoxia-inducible factor 1 and dysregulated c-Myc cooperatively induce vascular endothelial growth factor and metabolic switches hexokinase 2 and pyruvate dehydrogenase kinase 1 Transient in utero knockout (TIUKO) of C-MYC affects late lung and intestinal development in the mouse Interactions between Myc and Mediators of Inflammation in Chronic Liver Diseases Tumor endothelial cells The cell cycle and pluripotency Small-molecule inhibitors of the Myc oncoprotein Deficiency for the cysteine protease cathepsin L impairs Myc-induced tumorigenesis in a mouse model of pancreatic neuroendocrine cancer c-Myc recruits P-TEFb for transcription, cellular proliferation and apoptosis Formation and differentiation of multiple mesenchymal lineages during lung development is regulated by beta-catenin signaling The ubiquitin ligase Nedd4-1 is required for heart development and is a suppressor of thrombospondin-1 Myeloid leukemia factor 1 associates with a novel heterogeneous nuclear ribonucleoprotein U-like molecule Concurrent HDAC and mTORC1 inhibition attenuate androgen receptor and hypoxia signaling associated with alterations in microRNA expression MYC on the path to cancer Augmentation of tumor angiogenesis by a Myc-activated microRNA cluster c-myc in the hematopoietic lineage is crucial for its angiogenic function in the mouse embryo.Combinational delivery of c-myc siRNA and nucleoside analogs in a single, synthetic nanocarrier for targeted cancer therapy.ToP: a trend-of-disease-progression procedure works well for identifying cancer genes from multi-state cohort gene expression data for human colorectal cancer.In Situ Pluripotency Factor Expression Promotes Functional Recovery From Cerebral Ischemia Correlating global gene regulation to angiogenesis in the developing chick extra-embryonic vascular system.tRNA synthetase counteracts c-Myc to develop functional vasculature.SLEPR: a sample-level enrichment-based pathway ranking method -- seeking biological themes through pathway-level consistency RNA interference-mediated c-MYC inhibition prevents cell growth and decreases sensitivity to radio- and chemotherapy in childhood medulloblastoma cells.Nursing some sense out of Myc.The CASZ1/Egfl7 transcriptional pathway is required for RhoA expression in vascular endothelial cells.HIF-1alpha induces cell cycle arrest by functionally counteracting Myc.c-MYC independent nuclear reprogramming favors cardiogenic potential of induced pluripotent stem cells Increased stem cell marker expressions during the peri-implantation period in the rat endometrium: constructive role of exogenous zinc and/or progesterone.p130Rb2 and p27kip1 cooperate to control mobilization of angiogenic progenitors from the bone marrow.Comparison of selected gene expression profiles in sensitive and resistant cancer cells treated with doxorubicin and Selol Oncogene withdrawal engages the immune system to induce sustained cancer regression.Aurora kinases A and B are up-regulated by Myc and are essential for maintenance of the malignant state.Tridermal tumorigenesis of induced pluripotent stem cells transplanted in ischemic brain.The miR-17-92 cluster and its target THBS1 are differentially expressed in angiosarcomas dependent on MYC amplification Myc represses primitive endoderm differentiation in pluripotent stem cells.Mouse genetics suggests cell-context dependency for Myc-regulated metabolic enzymes during tumorigenesis An essential role for the immune system in the mechanism of tumor regression following targeted oncogene inactivation.In vivo inhibition of c-MYC in myeloid cells impairs tumor-associated macrophage maturation and pro-tumoral activities.PIWIL2 induces c-Myc expression by interacting with NME2 and regulates c-Myc-mediated tumor cell proliferation.

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c-Myc is essential for vasculogenesis and angiogenesis during development and tumor progression

http://www.wikidata.org/entity/Q35804203

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2002 nî lūn-bûn

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2002年の論文

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c-Myc is essential for vasculo ...... elopment and tumor progression

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c-Myc is essential for vasculo ...... elopment and tumor progression

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c-Myc is essential for vasculo ...... elopment and tumor progression

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c-Myc is essential for vasculo ...... elopment and tumor progression

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c-Myc is essential for vasculo ...... elopment and tumor progression

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c-Myc is essential for vasculo ...... elopment and tumor progression

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c-Myc is essential for vasculo ...... elopment and tumor progression

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Ann C Davis

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Catriona McKay

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Elsie L White

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Helene Pendeville-Samain

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James N Ihle

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John L Cleveland

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Jonas A Nilsson

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Kirsteen H Maclean

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Troy A Baudino

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P2860

The ornithine decarboxylase gene is a transcriptional target of c-Myc

Analysis of C-MYC function in normal cells via conditional gene-targeted mutation

Cellular and developmental control of O2 homeostasis by hypoxia-inducible factor 1 alpha

Role of HIF-1alpha in hypoxia-mediated apoptosis, cell proliferation and tumour angiogenesis

Expression of vascular endothelial growth factor during embryonic angiogenesis and endothelial cell differentiation

Role of the Flt-1 receptor tyrosine kinase in regulating the assembly of vascular endothelium

HIF-1 alpha is required for solid tumor formation and embryonic vascularization.

Failure of blood-island formation and vasculogenesis in Flk-1-deficient mice

A common precursor for primitive erythropoiesis and definitive haematopoiesis

Patterns and emerging mechanisms of the angiogenic switch during tumorigenesis

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2530-2543

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10.1101/GAD.1024602

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19

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16

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11091721

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12368264

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187450

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