Recombinant human tumor necrosis factor alpha suppresses autoimmune diabetes in nonobese diabetic mice.
about
Proof-of-concept, randomized, controlled clinical trial of Bacillus-Calmette-Guerin for treatment of long-term type 1 diabetesSelective death of autoreactive T cells in human diabetes by TNF or TNF receptor 2 agonismInhibition of autoimmune type 1 diabetes by gastrointestinal helminth infectionThe central role of calcium in the effects of cytokines on beta-cell function: implications for type 1 and type 2 diabetesEvidence that low-grade systemic inflammation can induce islet dysfunction as measured by impaired calcium handling.Microbiota in T-cell homeostasis and inflammatory diseasesAnalysis of tumor necrosis factor promoter responses to ultraviolet light.Post-thymectomy organ-specific autoimmunity: enhancement by cyclosporine A and inhibition by IL-2.The dual role of scavenger receptor class A in development of diabetes in autoimmune NOD miceStudies of T-cell activation in chronic inflammation.Identification of a serum-induced transcriptional signature associated with type 1 diabetes in the BioBreeding rat.Identification of a new susceptibility locus for insulin-dependent diabetes mellitus by ancestral haplotype congenic mappingPrevention of diabetes in nonobese diabetic mice by dendritic cell transfer.Interleukin 4 reverses T cell proliferative unresponsiveness and prevents the onset of diabetes in nonobese diabetic miceEffect of tumor necrosis factor alpha on insulin-dependent diabetes mellitus in NOD mice. I. The early development of autoimmunity and the diabetogenic process.IL-10 is necessary and sufficient for autoimmune diabetes in conjunction with NOD MHC homozygosityLocal expression of transgene encoded TNF alpha in islets prevents autoimmune diabetes in nonobese diabetic (NOD) mice by preventing the development of auto-reactive islet-specific T cellsIn autoimmune diabetes the transition from benign to pernicious insulitis requires an islet cell response to tumor necrosis factor alpha.Beta cells in type 2 diabetes - a crucial contribution to pathogenesis.Regulation of autoimmune inflammation by pro-inflammatory cytokinesBiochemical and molecular actions of interleukin-1 on pancreatic beta-cells.A T-cell dormant state in the autoimmune process of nonobese diabetic mice treated with complete Freund's adjuvant.Extensive genetic polymorphism in the human tumor necrosis factor region and relation to extended HLA haplotypes.Pleiotropic targets: the problem of shared signaling circuitry in rheumatoid arthritis disease progression and protection.CD4 T cell differentiation in type 1 diabetes.Neutralization Versus Reinforcement of Proinflammatory Cytokines to Arrest Autoimmunity in Type 1 Diabetes.Exogenous administration of IL-1 alpha inhibits active and adoptive transfer autoimmune diabetes in NOD mice.Endogenous TNF production differs between high and low diabetes incidence non-obese diabetic (NOD) mice.Enhanced tumor necrosis factor in anti-CD3 antibody stimulated diabetic NOD mice: modulation by PGE1 and dietary lipid.Expression of a tumor necrosis factor alpha transgene in murine pancreatic beta cells results in severe and permanent insulitis without evolution towards diabetes.Response to Treatment with TNFα Inhibitors in Rheumatoid Arthritis Is Associated with High Levels of GM-CSF and GM-CSF+ T Lymphocytes.Fas/Fas-Ligand Interaction As a Mechanism of Immune Homeostasis and β-Cell Cytotoxicity: Enforcement Rather Than Neutralization for Treatment of Type 1 Diabetes.mRNA of the pro-apoptotic gene BBC3 serves as a molecular marker for TNF-α-induced islet damage in humans.Interleukin-1 promotes hyperglycemia and insulitis in mice normally resistant to streptozotocin-induced diabetes.Unmasking of a protective tumor necrosis factor receptor I-mediated signal in the collagen-induced arthritis model.TNFR1 and TNFR2 differentially mediate TNF-α-induced inflammatory responses in rheumatoid arthritis fibroblast-like synoviocytes.A New Venue of TNF Targeting.What Have We Learned about the Pathogenesis of Rheumatoid Arthritis from TNF-Targeted Therapy?
P2860
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P2860
Recombinant human tumor necrosis factor alpha suppresses autoimmune diabetes in nonobese diabetic mice.
description
1989 nî lūn-bûn
@nan
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
1989年论文
@zh
1989年论文
@zh-cn
name
Recombinant human tumor necros ...... tes in nonobese diabetic mice.
@ast
Recombinant human tumor necros ...... tes in nonobese diabetic mice.
@en
type
label
Recombinant human tumor necros ...... tes in nonobese diabetic mice.
@ast
Recombinant human tumor necros ...... tes in nonobese diabetic mice.
@en
prefLabel
Recombinant human tumor necros ...... tes in nonobese diabetic mice.
@ast
Recombinant human tumor necros ...... tes in nonobese diabetic mice.
@en
P2093
P2860
P356
P1476
Recombinant human tumor necros ...... tes in nonobese diabetic mice.
@en
P2093
Nobunaga T
Shintani S
P2860
P304
P356
10.1172/JCI114304
P407
P577
1989-10-01T00:00:00Z