Inhibition of glycogen synthase kinase-3β prevents NSAID-induced acute kidney injury.
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Programmed necrosis in acute kidney injuryTherapeutic targeting of aldosterone: a novel approach to the treatment of glomerular diseaseIdentification of prostaglandin receptors in human ureters.Regulation of necrotic cell death: p53, PARP1 and cyclophilin D-overlapping pathways of regulated necrosis?Glycogen synthase kinase 3 regulates cell death and survival signaling in tumor cells under redox stress.Pharmacological targeting of GSK3β confers protection against podocytopathy and proteinuria by desensitizing mitochondrial permeability transition.Glycogen synthase kinase-3 inhibition attenuates fibroblast activation and development of fibrosis following renal ischemia-reperfusion in mice.Remote ischemic preconditioning for kidney protection: GSK3β-centric insights into the mechanism of action.Adrenocorticotropic hormone ameliorates acute kidney injury by steroidogenic-dependent and -independent mechanismsMitochondrial permeability transition and cell death: the role of cyclophilin dThe β isoform of GSK3 mediates podocyte autonomous injury in proteinuric glomerulopathy.Redox-sensitive glycogen synthase kinase 3β-directed control of mitochondrial permeability transition: rheostatic regulation of acute kidney injury.Inhibition of GSK-3β increases trabecular bone volume but not cortical bone volume in adenine-induced uremic mice with severe hyperparathyroidism.Delayed administration of a single dose of lithium promotes recovery from AKI.Molecular determinants of acute kidney injury.Mitochondrial Ca2+ and regulation of the permeability transition pore.Mitochondrial permeability transition in cardiac ischemia-reperfusion: whether cyclophilin D is a viable target for cardioprotection?The Emerging Role of Mitochondrial Targeting in Kidney Disease.Plant Natural Product Formononetin Protects Rat Cardiomyocyte H9c2 Cells against Oxygen Glucose Deprivation and Reoxygenation via Inhibiting ROS Formation and Promoting GSK-3β Phosphorylation.Effects of chronic alcohol exposure on ischemia-reperfusion-induced acute kidney injury in mice: the role of β-arrestin 2 and glycogen synthase kinase 3.Glycogen synthase kinase-3β inhibitor ameliorates imbalance of connexin 43 in an acute kidney injury model.Epoxide metabolites of arachidonate and docosahexaenoate function conversely in acute kidney injury involved in GSK3β signaling.
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P2860
Inhibition of glycogen synthase kinase-3β prevents NSAID-induced acute kidney injury.
description
2012 nî lūn-bûn
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2012年の論文
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2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
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2012年论文
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2012年论文
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name
Inhibition of glycogen synthase kinase-3β prevents NSAID-induced acute kidney injury.
@ast
Inhibition of glycogen synthase kinase-3β prevents NSAID-induced acute kidney injury.
@en
type
label
Inhibition of glycogen synthase kinase-3β prevents NSAID-induced acute kidney injury.
@ast
Inhibition of glycogen synthase kinase-3β prevents NSAID-induced acute kidney injury.
@en
prefLabel
Inhibition of glycogen synthase kinase-3β prevents NSAID-induced acute kidney injury.
@ast
Inhibition of glycogen synthase kinase-3β prevents NSAID-induced acute kidney injury.
@en
P2093
P2860
P356
P1433
P1476
Inhibition of glycogen synthase kinase-3β prevents NSAID-induced acute kidney injury
@en
P2093
Lance D Dworkin
Rujun Gong
Shougang Zhuang
P2860
P2888
P304
P356
10.1038/KI.2011.443
P407
P50
P577
2012-01-18T00:00:00Z