GSK3beta promotes apoptosis after renal ischemic injury. - Wikidata - wikimedia - TriplyDB

GSK3beta promotes apoptosis after renal ischemic injury.

GSK3beta promotes apoptosis after renal ischemic injury.

http://www.wikidata.org/entity/Q33711281

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Megalin in acute kidney injury: foe and friend Molecular mechanisms of ischemic preconditioning in the kidney Role of mitofusin 2 in the renal stress response Glycogen synthase kinase 3β dictates podocyte motility and focal adhesion turnover by modulating paxillin activity: implications for the protective effect of low-dose lithium in podocytopathy.Cyclophilin D and the mitochondrial permeability transition in kidney proximal tubules after hypoxic and ischemic injury Glycogen synthase kinase 3β orchestrates microtubule remodeling in compensatory glomerular adaptation to podocyte depletion.Inhibition of glycogen synthase kinase 3 beta ameliorates liver ischemia reperfusion injury by way of an interleukin-10-mediated immune regulatory mechanism.A nonerythropoietic peptide that mimics the 3D structure of erythropoietin reduces organ injury/dysfunction and inflammation in experimental hemorrhagic shock.Enhanced glycogen synthase kinase-3β activity mediates podocyte apoptosis under diabetic conditions.Endoplasmic reticulum stress-induced IRE1α activation mediates cross-talk of GSK-3β and XBP-1 to regulate inflammatory cytokine production.Conditional knockout of proximal tubule mitofusin 2 accelerates recovery and improves survival after renal ischemia Impaired wound healing in hypoxic renal tubular cells: roles of hypoxia-inducible factor-1 and glycogen synthase kinase 3β/β-catenin signaling Inhibition of glycogen synthase kinase-3β prevents NSAID-induced acute kidney injury.Glycogen synthase kinase-3 inhibition attenuates fibroblast activation and development of fibrosis following renal ischemia-reperfusion in mice.Comparing the Bbs10 complete knockout phenotype with a specific renal epithelial knockout one highlights the link between renal defects and systemic inactivation in mice Hexokinase regulates Bax-mediated mitochondrial membrane injury following ischemic stress Delayed administration of pyroglutamate helix B surface peptide (pHBSP), a novel nonerythropoietic analog of erythropoietin, attenuates acute kidney injury.Apoptosis and acute kidney injury Specific deletion of glycogen synthase kinase-3β in the renal proximal tubule protects against acute nephrotoxic injury in mice Muc1 enhances the β-catenin protective pathway during ischemia-reperfusion injury Nucleophosmin, a critical Bax cofactor in ischemia-induced cell death Glycogen synthase kinase-3 regulation of urinary concentrating ability AKT/GSK3β signaling pathway is critically involved in human pluripotent stem cell survival.Redox-sensitive glycogen synthase kinase 3β-directed control of mitochondrial permeability transition: rheostatic regulation of acute kidney injury.Inhibition of GSK-3β increases trabecular bone volume but not cortical bone volume in adenine-induced uremic mice with severe hyperparathyroidism.Renoprotective approaches and strategies in acute kidney injury A cAMP and CREB-mediated feed-forward mechanism regulates GSK3β in polycystic kidney disease.Delayed administration of a single dose of lithium promotes recovery from AKI.Lithium in the Kidney: Friend and Foe?Proteinuria causes dysfunctional autophagy in the proximal tubule.Molecular mechanisms in lithium-associated renal disease: a systematic review.Ser9-phosphorylated GSK3β induced by 14-3-3ζ actively antagonizes cell apoptosis in a NF-κB dependent manner.Effects of chronic alcohol exposure on ischemia-reperfusion-induced acute kidney injury in mice: the role of β-arrestin 2 and glycogen synthase kinase 3.Glycogen synthase kinase-3β inhibitor ameliorates imbalance of connexin 43 in an acute kidney injury model.Lithium in kidney diseases: big roles for the smallest metal Glycogen synthase kinase-3β is involved in electroacupuncture pretreatment via the cannabinoid CB1 receptor in ischemic stroke.Epoxide metabolites of arachidonate and docosahexaenoate function conversely in acute kidney injury involved in GSK3β signaling.Learning ability is a key outcome determinant of GSK-3 inhibition on visuospatial memory in rats.Renal Tubule Repair: Is Wnt/β-Catenin a Friend or Foe?UCP2 attenuates apoptosis of tubular epithelial cells in renal ischemia-reperfusion injury.

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P2860

GSK3beta promotes apoptosis after renal ischemic injury.

http://www.wikidata.org/entity/Q33711281

description

2010 nî lūn-bûn

@nan

2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունվարին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

GSK3beta promotes apoptosis after renal ischemic injury.

@ast

GSK3beta promotes apoptosis after renal ischemic injury.

@en

type

label

GSK3beta promotes apoptosis after renal ischemic injury.

@ast

GSK3beta promotes apoptosis after renal ischemic injury.

@en

prefLabel

GSK3beta promotes apoptosis after renal ischemic injury.

@ast

GSK3beta promotes apoptosis after renal ischemic injury.

@en

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P1433

Journal of the American Society of Nephrology

P1476

GSK3beta promotes apoptosis after renal ischemic injury

@en

P2093

Haiping Mao

http://www.w3.org/2001/XMLSchema#string

Jonathan Gall

http://www.w3.org/2001/XMLSchema#string

Zhijian Li

http://www.w3.org/2001/XMLSchema#string

Zhiyong Wang

http://www.w3.org/2001/XMLSchema#string

P2860

Glycogen synthase kinase-3 regulates mitochondrial outer membrane permeabilization and apoptosis by destabilization of MCL-1

GSK-3: tricks of the trade for a multi-tasking kinase

Cytosol-to-membrane redistribution of Bax and Bcl-X(L) during apoptosis

BH4 domain of antiapoptotic Bcl-2 family members closes voltage-dependent anion channel and inhibits apoptotic mitochondrial changes and cell death

Structure of Bax: coregulation of dimer formation and intracellular localization

A ubiquitin ligase complex essential for the NF-kappaB, Wnt/Wingless, and Hedgehog signaling pathways

Essential role of voltage-dependent anion channel in various forms of apoptosis in mammalian cells

Glycogen synthase kinase-3beta regulates cyclin D1 proteolysis and subcellular localization

Voltage-dependent anion channels are dispensable for mitochondrial-dependent cell death

Cell adhesion and the integrin-linked kinase regulate the LEF-1 and beta-catenin signaling pathways

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284-294

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scholarly article

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10.1681/ASN.2009080828

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2

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21

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John H Schwartz

Ramon G Bonegio

Steven C Borkan

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2010-01-21T00:00:00Z

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20093356

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2834548

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