Long-term exposure of proximal tubular epithelial cells to glucose induces transforming growth factor-beta 1 synthesis via an autocrine PDGF loop.
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Post-transcriptional regulation of Transforming Growth Factor Beta-1 by microRNA-744The AMPK agonist AICAR inhibits TGF-β1 induced activation of kidney myofibroblastsDysregulation of TGFβ1 Activity in Cancer and Its Influence on the Quality of Anti-Tumor ImmunityHigh glucose down-regulates miR-29a to increase collagen IV production in HK-2 cellsPolarity of response to transforming growth factor-beta1 in proximal tubular epithelial cells is regulated by beta-cateninA conserved stem loop motif in the 5'untranslated region regulates transforming growth factor-β(1) translation.Tribbles 3 regulates the fibrosis cytokine TGF- β 1 through ERK1/2-MAPK signaling pathway in diabetic nephropathy.Regulation of mRNA translation in renal physiology and disease.Can we target tubular damage to prevent renal function decline in diabetes?Targeting pericyte differentiation as a strategy to modulate kidney fibrosis in diabetic nephropathy.Novel mechanisms of protein synthesis in diabetic nephropathy--role of mRNA translation.Tubulointerstitial disease in diabetic nephropathy.MicroRNAs, transforming growth factor beta-1, and tissue fibrosis.Renal proximal tubular epithelial cell transforming growth factor-beta1 generation and monocyte bindingBone morphogenetic protein-7 inhibits proximal tubular epithelial cell Smad3 signaling via increased SnoN expression.Cation-independent mannose 6-phosphate receptor inhibitor (PXS25) inhibits fibrosis in human proximal tubular cells by inhibiting conversion of latent to active TGF-beta1.Triptolide inhibits extracellular matrix protein synthesis by suppressing the Smad2 but not the MAPK pathway in TGF-beta1-stimulated NRK-49F cells.Interleukin-1 beta regulates proximal tubular cell transforming growth factor beta-1 signalling.Hyaluronan orchestrates transforming growth factor-beta1-dependent maintenance of myofibroblast phenotype.C-peptide reverses TGF-beta1-induced changes in renal proximal tubular cells: implications for treatment of diabetic nephropathyInhibition of transglutaminase activity reduces extracellular matrix accumulation induced by high glucose levels in proximal tubular epithelial cells.ERK, p38, and Smad signaling pathways differentially regulate transforming growth factor-beta1 autoinduction in proximal tubular epithelial cells.A combination of biomolecules enhances expression of E-cadherin and peroxisome proliferator-activated receptor gene leading to increased cell proliferation in primary human meniscal cells: an in vitro studyEffect of activin A on tubulointerstitial fibrosis in diabetic nephropathy.PDGF receptor-β uses Akt/mTORC1 signaling node to promote high glucose-induced renal proximal tubular cell collagen I (α2) expression.Acute Kidney Injury and Progression of Diabetic Kidney Disease.Fluorofenidone attenuates tubulointerstitial fibrosis by inhibiting TGF-β(1)-induced fibroblast activation.Osteomeles schwerinae Extract Prevents Diabetes-Induced Renal Injury in Spontaneously Diabetic Torii Rats.
P2860
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P2860
Long-term exposure of proximal tubular epithelial cells to glucose induces transforming growth factor-beta 1 synthesis via an autocrine PDGF loop.
description
2003 nî lūn-bûn
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2003年の論文
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2003年学术文章
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name
Long-term exposure of proximal ...... is via an autocrine PDGF loop.
@ast
Long-term exposure of proximal ...... is via an autocrine PDGF loop.
@en
type
label
Long-term exposure of proximal ...... is via an autocrine PDGF loop.
@ast
Long-term exposure of proximal ...... is via an autocrine PDGF loop.
@en
prefLabel
Long-term exposure of proximal ...... is via an autocrine PDGF loop.
@ast
Long-term exposure of proximal ...... is via an autocrine PDGF loop.
@en
P2093
P2860
P1476
Long-term exposure of proximal ...... is via an autocrine PDGF loop.
@en
P2093
Aled Phillips
Nigel Brunskill
Takafumi Ito
P2860
P304
P356
10.1016/S0002-9440(10)63611-5
P407
P577
2003-12-01T00:00:00Z