Gender modulates cardiac phenotype development in genetically modified mice.
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SIRT2 induces the checkpoint kinase BubR1 to increase lifespanThe OPA1-dependent mitochondrial cristae remodeling pathway controls atrophic, apoptotic, and ischemic tissue damageTissue-specific splicing of an Ndufs6 gene-trap insertion generates a mitochondrial complex I deficiency-specific cardiomyopathyInactivation of Smad5 in endothelial cells and smooth muscle cells demonstrates that Smad5 is required for cardiac homeostasisMechanistic Pathways of Sex Differences in Cardiovascular Disease.Human cardiac-specific cDNA array for idiopathic dilated cardiomyopathy: sex-related differences.Lamina-associated polypeptide 2alpha loss impairs heart function and stress response in mice.Cardiac tissue-specific repression of CELF activity disrupts alternative splicing and causes cardiomyopathy.Cardiac-specific overexpression of dominant-negative CREB leads to increased mortality and mitochondrial dysfunction in female mice.Relaxin: antifibrotic properties and effects in models of diseaseInfluence of genetic background on ex vivo and in vivo cardiac function in several commonly used inbred mouse strainsSirtuin to the rescue: SIRT2 extends life span of BubR1 mice.Sexual dimorphism in the expression of mitochondria-related genes in rat heart at different ages.Gene-Targeted Mice with the Human Troponin T R141W Mutation Develop Dilated Cardiomyopathy with Calcium DesensitizationTherapeutic implications of the gender-specific aspects of cardiovascular disease.The role of sex in cardiac function and disease.Subtle abnormalities in contractile function are an early manifestation of sarcomere mutations in dilated cardiomyopathy.Sex-specific pathways in early cardiac response to pressure overload in mice.Myocardial AKT: the omnipresent nexus.Cardiac tissue factor: roles in physiology and fibrosis.FGF2 modulates cardiac remodeling in an isoform- and sex-specific manner.Cardiac development and physiology are modulated by FGF2 in an isoform- and sex-specific manner.Sex and gender differences in myocardial hypertrophy and heart failure.Protective regulation of the ACE2/ACE gene expression by estrogen in human atrial tissue from elderly men.Sex steroids in relation to cardiac structure and function in men.Interleukin-19 is cardioprotective in dominant negative cyclic adenosine monophosphate response-element binding protein-mediated heart failure in a sex-specific manner.Gender-specific differences in major cardiac events and mortality in lamin A/C mutation carriers.Mouse strain determines the outcome of wound healing after myocardial infarction.Blocking cardiac growth in hypertrophic cardiomyopathy induces cardiac dysfunction and decreased survival only in males.Effect of a low-protein diet during pregnancy on expression of genes involved in cardiac hypertrophy in fetal and adult mouse offspring.Differential PI3K signal transduction in obesity-associated cardiac hypertrophy and response to ischemia.
P2860
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P2860
Gender modulates cardiac phenotype development in genetically modified mice.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Gender modulates cardiac phenotype development in genetically modified mice.
@ast
Gender modulates cardiac phenotype development in genetically modified mice.
@en
type
label
Gender modulates cardiac phenotype development in genetically modified mice.
@ast
Gender modulates cardiac phenotype development in genetically modified mice.
@en
prefLabel
Gender modulates cardiac phenotype development in genetically modified mice.
@ast
Gender modulates cardiac phenotype development in genetically modified mice.
@en
P1476
Gender modulates cardiac phenotype development in genetically modified mice.
@en
P2093
Xiao-Jun Du
P304
P356
10.1016/J.CARDIORES.2004.03.027
P577
2004-08-01T00:00:00Z