TXNIP links innate host defense mechanisms to oxidative stress and inflammation in retinal Muller glia under chronic hyperglycemia: implications for diabetic retinopathy. - Wikidata - wikimedia - TriplyDB

TXNIP links innate host defense mechanisms to oxidative stress and inflammation in retinal Muller glia under chronic hyperglycemia: implications for diabetic retinopathy.

TXNIP links innate host defense mechanisms to oxidative stress and inflammation in retinal Muller glia under chronic hyperglycemia: implications for diabetic retinopathy.

http://www.wikidata.org/entity/Q35855616

about

Adipose tissue macrophages: going off track during obesity The unfolded protein response and diabetic retinopathy Thioredoxin/Txnip: redoxisome, as a redox switch for the pathogenesis of diseases Thioredoxin Interacting Protein (TXNIP) and Pathogenesis of Diabetic Retinopathy Targeting the Nrf2 Signaling Pathway in the Retina With a Gene-Delivered Secretable and Cell-Penetrating Peptide Resolvin D1 inhibits inflammatory response in STZ-induced diabetic retinopathy rats: Possible involvement of NLRP3 inflammasome and NF-κB signaling pathway.Inflammasomes: molecular regulation and implications for metabolic and cognitive diseases Bone morphogenetic protein 7 regulates reactive gliosis in retinal astrocytes and Müller glia.NLRP3 gene silencing ameliorates diabetic cardiomyopathy in a type 2 diabetes rat model.Modulating Molecular Chaperones Improves Mitochondrial Bioenergetics and Decreases the Inflammatory Transcriptome in Diabetic Sensory Neurons Retinal Ganglion Cell Loss is Delayed Following Optic Nerve Crush in NLRP3 Knockout Mice.KCa3.1 mediates dysfunction of tubular autophagy in diabetic kidneys via PI3k/Akt/mTOR signaling pathways Promoting Neuronal Tolerance of Diabetic Stress: Modulating Molecular Chaperones.Thioredoxin interacting protein (TXNIP) regulates tubular autophagy and mitophagy in diabetic nephropathy through the mTOR signaling pathway.Animal models of diabetic retinopathy: summary and comparison.Deletion of thioredoxin-interacting protein preserves retinal neuronal function by preventing inflammation and vascular injury.Autophagy regulates inflammation following oxidative injury in diabetes.Small-interfering RNAs (siRNAs) as a promising tool for ocular therapy.Epigenetic modifications and potential new treatment targets in diabetic retinopathy Role of inflammasome activation in the pathophysiology of vascular diseases of the neurovascular unit.Molecular mechanisms of diabetic retinopathy: potential therapeutic targets.TXNIP regulates mitophagy in retinal Müller cells under high-glucose conditions: implications for diabetic retinopathy.High fat diet dysregulates microRNA-17-5p and triggers retinal inflammation: Role of endoplasmic-reticulum-stress.miR-146a Attenuates Inflammatory Pathways Mediated by TLR4/NF-κB and TNFα to Protect Primary Human Retinal Microvascular Endothelial Cells Grown in High Glucose.Therapeutic targeting of diabetic retinal neuropathy as a strategy in preventing diabetic retinopathy.Cerebrovascular complications of diabetes: focus on cognitive dysfunction PI3K-mediated glioprotective effect of epidermal growth factor under oxidative stress conditions.Quercetin and allopurinol reduce liver thioredoxin-interacting protein to alleviate inflammation and lipid accumulation in diabetic rats.Critical role of TXNIP in oxidative stress, DNA damage and retinal pericyte apoptosis under high glucose: implications for diabetic retinopathy Mitochondrial Reactive Oxygen Species and Kidney Hypoxia in the Development of Diabetic Nephropathy Regulatory Role of Redox Balance in Determination of Neural Precursor Cell Fate.Diabetic retinopathy: reversibility of epigenetic modifications and new therapeutic targets.Thioredoxin-interacting protein: a novel target for neuroprotection in experimental thromboembolic stroke in mice.High Glucose and Lipopolysaccharide Prime NLRP3 Inflammasome via ROS/TXNIP Pathway in Mesangial Cells.Hyperreflective intraretinal spots in diabetics without and with nonproliferative diabetic retinopathy: an in vivo study using spectral domain OCT.Thioredoxin-interacting protein is required for endothelial NLRP3 inflammasome activation and cell death in a rat model of high-fat diet NLRP3 Inflammasome Activation-Mediated Pyroptosis Aggravates Myocardial Ischemia/Reperfusion Injury in Diabetic Rats.Inflammasomes, the eye and anti-inflammasome therapy.miR-15a/16 inhibits TGF-beta3/VEGF signaling and increases retinal endothelial cell barrier proteins.NLRP3 inflammasome activation is associated with proliferative diabetic retinopathy.

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P2860

TXNIP links innate host defense mechanisms to oxidative stress and inflammation in retinal Muller glia under chronic hyperglycemia: implications for diabetic retinopathy.

http://www.wikidata.org/entity/Q35855616

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年论文

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name

TXNIP links innate host defens ...... ions for diabetic retinopathy.

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TXNIP links innate host defens ...... ions for diabetic retinopathy.

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TXNIP links innate host defens ...... ions for diabetic retinopathy.

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TXNIP links innate host defens ...... ions for diabetic retinopathy.

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TXNIP links innate host defens ...... ions for diabetic retinopathy.

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TXNIP links innate host defens ...... ions for diabetic retinopathy.

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Experimental Diabetes Research

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TXNIP links innate host defens ...... ions for diabetic retinopathy.

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Ashok Kumar

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Icksoo Lee

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Kwaku D Nantwi

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Lalit P Singh

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Takhellambam S Devi

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P2860

cAMP controls oxygen metabolism in mammalian cells

Endogenous HMGB1 regulates autophagy

Reactive oxygen species-independent activation of the IL-1beta inflammasome in cells from patients with chronic granulomatous disease

Resistance of retinal inflammatory mediators to suppress after reinstitution of good glycemic control: novel mechanism for metabolic memory

Interaction between NO and COX pathways in retinal cells exposed to elevated glucose and retina of diabetic rats

Hyperglycemia promotes oxidative stress through inhibition of thioredoxin function by thioredoxin-interacting protein

HMGB1 and RAGE in inflammation and cancer

Induction of ER stress in response to oxygen-glucose deprivation of cortical cultures involves the activation of the PERK and IRE-1 pathways and of caspase-12

RAGE-TXNIP axis is required for S100B-promoted Schwann cell migration, fibronectin expression and cytokine secretion

Regulation of glucose homeostasis through a XBP-1-FoxO1 interaction

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