Roles of the transcription factors snail and slug during mammary morphogenesis and breast carcinoma progression. - Wikidata - wikimedia - TriplyDB

Roles of the transcription factors snail and slug during mammary morphogenesis and breast carcinoma progression.

Roles of the transcription factors snail and slug during mammary morphogenesis and breast carcinoma progression.

http://www.wikidata.org/entity/Q35858258

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Jagged1-mediated Notch activation induces epithelial-to-mesenchymal transition through Slug-induced repression of E-cadherin.Regulation of BRCA2 gene expression by the SLUG repressor protein in human breast cells HER-2 overexpression differentially alters transforming growth factor-beta responses in luminal versus mesenchymal human breast cancer cells The impact of low-dose carcinogens and environmental disruptors on tissue invasion and metastasis Wilms' tumor 1 (Wt1) regulates pleural mesothelial cell plasticity and transition into myofibroblasts in idiopathic pulmonary fibrosis Drug development against metastasis-related genes and their pathways: A rationale for cancer therapy GSK3β controls epithelial-mesenchymal transition and tumor metastasis by CHIP-mediated degradation of Slug An NF-kappaB and slug regulatory loop active in early vertebrate mesoderm.Staurosporine augments EGF-mediated EMT in PMC42-LA cells through actin depolymerisation, focal contact size reduction and Snail1 induction - a model for cross-modulation Protein kinase D1 maintains the epithelial phenotype by inducing a DNA-bound, inactive SNAI1 transcriptional repressor complex.In vivo tumorigenesis was observed after injection of in vitro expanded neural crest stem cells isolated from adult bone marrow GATA1 induces epithelial-mesenchymal transition in breast cancer cells through PAK5 oncogenic signaling.Direct and indirect contribution of bone marrow-derived cells to cancer.Oseltamivir phosphate monotherapy ablates tumor neovascularization, growth, and metastasis in mouse model of human triple-negative breast adenocarcinoma Snail family members unequally trigger EMT and thereby differ in their ability to promote the neoplastic transformation of mammary epithelial cells.MCF-7 Human Breast Cancer Cells Form Differentiated Microtissues in Scaffold-Free Hydrogels.Singleminded-2s (Sim2s) promotes delayed involution of the mouse mammary gland through suppression of Stat3 and NFκB An in vitro-in vivo model of epithelial mesenchymal transition in triple negative breast cancer.The skinny on Slug.Polyethylene glycol-mediated colorectal cancer chemoprevention: roles of epidermal growth factor receptor and Snail.The transcription factors Slug (SNAI2) and Snail (SNAI1) regulate phospholipase D (PLD) promoter in opposite ways towards cancer cell invasion.Transient SNAIL1 expression is necessary for metastatic competence in breast cancer Proteomic analysis of tumor necrosis factor-alpha resistant human breast cancer cells reveals a MEK5/Erk5-mediated epithelial-mesenchymal transition phenotype The transcription factor snail mediates epithelial to mesenchymal transitions by repression of estrogen receptor-alpha.Autocrine epiregulin activates EGFR pathway for lung metastasis via EMT in salivary adenoid cystic carcinoma.PAK signaling in oncogenesis.Molecular biology of adenoid cystic carcinoma Snail2 directly represses cadherin6B during epithelial-to-mesenchymal transitions of the neural crest.Cytokeratin 18 is necessary for initiation of TGF-β1-induced epithelial-mesenchymal transition in breast epithelial cells.RNA Interference Targeting Snail Inhibits the Transforming Growth Factor β 2-Induced Epithelial-Mesenchymal Transition in Human Lens Epithelial Cells.Immunohistochemical Study EMT-Related Proteins in HPV-, and EBV-Negative Patients with Sinonasal Tumours.TRPV4 plays a role in breast cancer cell migration via Ca2+-dependent activation of AKT and downregulation of E-cadherin cell cortex protein.Novel insight into the function and regulation of alphaN-catenin by Snail2 during chick neural crest cell migration.Snail1, Snail2, and E47 promote mammary epithelial branching morphogenesis.A PHD12-Snail2 repressive complex epigenetically mediates neural crest epithelial-to-mesenchymal transition.A critical role for Cadherin6B in regulating avian neural crest emigration.Further evidence that E-cadherin is not a tumour suppressor gene in invasive ductal carcinoma of the breast: an immunohistochemical study.Targeted expression of GLI1 in the mammary gland disrupts pregnancy-induced maturation and causes lactation failure.

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Roles of the transcription factors snail and slug during mammary morphogenesis and breast carcinoma progression.

http://www.wikidata.org/entity/Q35858258

description

2004 nî lūn-bûn

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年论文

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2004年论文

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Roles of the transcription fac ...... breast carcinoma progression.

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Roles of the transcription fac ...... breast carcinoma progression.

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Journal of Mammary Gland Biology and Neoplasia

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Roles of the transcription fac ...... breast carcinoma progression.

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Christophe Côme

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Frédéric Bibeau

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Pierre Savagner

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Valérie Arnoux

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P2860

The SLUG zinc-finger protein represses E-cadherin in breast cancer

RB and c-Myc activate expression of the E-cadherin gene in epithelial cells through interaction with transcription factor AP-2.

dCtBP mediates transcriptional repression by Knirps, Krüppel and Snail in the Drosophila embryo.

The E-cadherin/catenin complex: an important gatekeeper in breast cancer tumorigenesis and malignant progression.

E-cadherin expression in primary carcinomas of the breast and its distant metastases

Epithelial-mesenchymal transitions in tumour progression

The transcription factor snail is a repressor of E-cadherin gene expression in epithelial tumour cells

Increased invasion and matrix metalloproteinase-2 expression by Snail-induced mesenchymal transition in squamous cell carcinomas

MTA3, a Mi-2/NuRD complex subunit, regulates an invasive growth pathway in breast cancer

Down-regulation of promoter 1.3 activity of the human aromatase gene in breast tissue by zinc-finger protein, snail (SnaH)

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