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Calsequestrin 2 and arrhythmiasRegulation of intracellular Na(+) in health and disease: pathophysiological mechanisms and implications for treatmentStochastic spontaneous calcium release events trigger premature ventricular complexes by overcoming electrotonic loadLate Na(+) current and protracted electrical recovery are critical determinants of the aging myopathyCirculating Pneumolysin Is a Potent Inducer of Cardiac Injury during Pneumococcal InfectionArrhythmogenic mechanisms in a mouse model of catecholaminergic polymorphic ventricular tachycardia.Aberrant cell-to-cell coupling in Ca2+-overloaded guinea pig ventricular muscles.Reduced Ca2+ transport across sarcolemma but enhanced spontaneous activity in cardiomyocytes isolated from left atrium-pulmonary veins tissue of myopathic hamster.Role of calcium desensitization in the treatment of myocardial dysfunction after deep hypothermic circulatory arrestPurkinje cells from RyR2 mutant mice are highly arrhythmogenic but responsive to targeted therapySirt3 protects cortical neurons against oxidative stress via regulating mitochondrial Ca2+ and mitochondrial biogenesis.Novel oscillatory mechanisms in the cholinergic control of Guinea pig sino-atrial node discharge.Overexpression of adenylyl cyclase type 5 (AC5) confers a proarrhythmic substrate to the heartIntercalated disc-associated protein, mXin-alpha, influences surface expression of ITO currents in ventricular myocytes.Ca2+- and mitochondrial-dependent cardiomyocyte necrosis as a primary mediator of heart failureKetamine attenuates the Na+-dependent Ca2+ overload in rabbit ventricular myocytes in vitro by inhibiting late Na+ and L-type Ca2+ currents.Focal but reversible diastolic sheet dysfunction reflects regional calcium mishandling in dystrophic mdx mouse hearts.Myocyte repolarization modulates myocardial function in aging dogsConstitutive Intracellular Na+ Excess in Purkinje Cells Promotes Arrhythmogenesis at Lower Levels of Stress Than Ventricular Myocytes From Mice With Catecholaminergic Polymorphic Ventricular TachycardiaOn the mechanisms of arrhythmias in the myocardium of mXinalpha-deficient murine left atrial-pulmonary veins.Hypothermia/rewarming disrupts excitation-contraction coupling in cardiomyocytes.Association of a congenital long QT syndrome type 1 with Takotsubo cardiomyopathyEssential role of diastolic oscillatory potentials in adrenergic control of guinea pig sino-atrial node discharge.Role of the excessive amounts of circulating catecholamines and glucocorticoids in stress-induced heart disease.Aging is a primary risk factor for cardiac arrhythmias: disruption of intracellular Ca2+ regulation as a key suspect.Ranolazine: clinical applications and therapeutic basis.Implantable cardioverter defibrillators in diabetics: efficacy and safety in patients at risk of sudden cardiac death.The Significance of the Late Na+ Current for Arrhythmia Induction and the Therapeutic Antiarrhythmic Potential of Ranolazine.Murine Electrophysiological Models of Cardiac Arrhythmogenesis.How does cholinergic activation slow down sinus node automaticity?"diastolic voltage oscillations" vs. "calcium clock" mechanisms.PCP4 regulates Purkinje cell excitability and cardiac rhythmicity.Mechanisms responsible for beneficial and adverse effects of rosiglitazone in a rat model of acute cardiac ischaemia-reperfusion.Load-dependent effects of apelin on murine cardiomyocytes.Heat Failure Phenotypes Induced by Knockdown of DAPIT in Zebrafish: A New Insight into Mechanism of Dilated Cardiomyopathy.Myocardial calcium overload during graded hypothermia and after rewarming in an in vivo rat model.Wenxin Keli diminishes Ca2+ overload induced by hypoxia/reoxygenation in cardiomyocytes through inhibiting INaL and ICaL.Effects of phosphodiestrase type 5 inhibitors in epinephrine-induced arrhythmia in rats: Involvement of lactate dehydrogenase and creatine kinase downregulation and adiponectin expression.Phospholamban phosphorylation increases the passive calcium leak from cardiac sarcoplasmic reticulum.Mechanical stimuli modulate intracellular calcium oscillations: a pathological model without chemical cues.Intermittent peripheral tissue ischemia during coronary ischemia reduces myocardial infarction through a KATP-dependent mechanism: first demonstration of remote ischemic perconditioning.
P2860
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P2860
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Calcium overload and cardiac function.
@ast
Calcium overload and cardiac function.
@en
type
label
Calcium overload and cardiac function.
@ast
Calcium overload and cardiac function.
@en
prefLabel
Calcium overload and cardiac function.
@ast
Calcium overload and cardiac function.
@en
P2860
P356
P1476
Calcium overload and cardiac function.
@en
P2093
Cheng-I Lin
Mario Vassalle
P2860
P2888
P304
P356
10.1007/BF02256119
P577
2004-09-01T00:00:00Z
P6179
1043510427