Tacking toward reconciliation on Tacr3/TACR3 mutations.

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Interactions between kisspeptins and neurokinin B Neurokinin B receptor antagonism decreases luteinising hormone pulse frequency and amplitude and delays puberty onset in the female rat.

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Tacking toward reconciliation on Tacr3/TACR3 mutations.

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2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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Tacking toward reconciliation on Tacr3/TACR3 mutations.

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Tacking toward reconciliation on Tacr3/TACR3 mutations.

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Tacking toward reconciliation on Tacr3/TACR3 mutations.

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Tacking toward reconciliation on Tacr3/TACR3 mutations.

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Tacking toward reconciliation on Tacr3/TACR3 mutations.

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Tacking toward reconciliation on Tacr3/TACR3 mutations.

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Tacking toward reconciliation on Tacr3/TACR3 mutations.

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Robert A Steiner

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Víctor M Navarro

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Hypogonadotropic hypogonadism due to loss of function of the KiSS1-derived peptide receptor GPR54

The GPR54 gene as a regulator of puberty

Kisspeptin signaling in the brain

Neurokinin B stimulates GnRH release in the male monkey (Macaca mulatta) and is colocalized with kisspeptin in the arcuate nucleus

Uncovering novel reproductive defects in neurokinin B receptor null mice: closing the gap between mice and men.

Regulation of NKB pathways and their roles in the control of Kiss1 neurons in the arcuate nucleus of the male mouse.

Neurobiological mechanisms of the onset of puberty in primates.

TAC3/TACR3 mutations reveal preferential activation of gonadotropin-releasing hormone release by neurokinin B in neonatal life followed by reversal in adulthood

Normosmic congenital hypogonadotropic hypogonadism due to TAC3/TACR3 mutations: characterization of neuroendocrine phenotypes and novel mutations

Neurokinin B acts via the neurokinin-3 receptor in the retrochiasmatic area to stimulate luteinizing hormone secretion in sheep.

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1578-1581

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scholarly article

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10.1210/EN.2012-1032

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English

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153

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2012-03-09T00:00:00Z

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22408179

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3320245

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Tacking toward reconciliation on Tacr3/TACR3 mutations.

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Tacking toward reconciliation on Tacr3/TACR3 mutations.

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