Deranged neuronal calcium signaling and Huntington disease.
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Evaluation of Dimebon in cellular model of Huntington's diseaseSTIM2 is a feedback regulator that stabilizes basal cytosolic and endoplasmic reticulum Ca2+ levelsThe excitatory neurotransmitter glutamate stimulates DNA repair to increase neuronal resiliencyTissue transglutaminase overexpression does not modify the disease phenotype of the R6/2 mouse model of Huntington's diseaseMitochondria, calcium and cell death: a deadly triad in neurodegenerationNeuroprotective effects of inositol 1,4,5-trisphosphate receptor C-terminal fragment in a Huntington's disease mouse modelMitochondria-associated membranes as hubs for neurodegenerationFrom mechanosensitivity to inflammatory responses: new players in the pathology of glaucomaReactive Oxygen Species: Physiological and Physiopathological Effects on Synaptic PlasticityCalpain and STriatal-Enriched protein tyrosine phosphatase (STEP) activation contribute to extrasynaptic NMDA receptor localization in a Huntington's disease mouse modelAssessment of motor function, sensory motor gating and recognition memory in a novel BACHD transgenic rat model for huntington disease.Characterization of the Kallikrein-Kinin System Post Chemical Neuronal Injury: An In Vitro Biochemical and Neuroproteomics AssessmentDeranged calcium signaling and neurodegeneration in spinocerebellar ataxia type 3Genome-wide loss of 5-hmC is a novel epigenetic feature of Huntington's diseaseAmyloid oligomer neurotoxicity, calcium dysregulation, and lipid raftsElucidating a normal function of huntingtin by functional and microarray analysis of huntingtin-null mouse embryonic fibroblasts.Wild-type huntingtin ameliorates striatal neuronal atrophy but does not prevent other abnormalities in the YAC128 mouse model of Huntington disease.GM1 and NGF modulate Ca2+ homeostasis and GAP43 mRNA expression in cultured dorsal root ganglion neurons with excitotoxicity induced by glutamate.CalDAG-GEFI down-regulation in the striatum as a neuroprotective change in Huntington's disease.Striatal Vulnerability in Huntington's Disease: Neuroprotection Versus Neurotoxicity.Disturbed Ca2+ signaling and apoptosis of medium spiny neurons in Huntington's disease.Neuregulin-1β prevents Ca(2+) overloading and apoptosis through PI3K/Akt activation in cultured dorsal root ganglion neurons with excitotoxicity induced by glutamate.Proteostasis in striatal cells and selective neurodegeneration in Huntington's disease.Computational analysis of calcium signaling and membrane electrophysiology in cerebellar Purkinje neurons associated with ataxia.Altered Ca(2+) signaling in skeletal muscle fibers of the R6/2 mouse, a model of Huntington's diseaseInvolvement of mitochondrial complex II defects in neuronal death produced by N-terminus fragment of mutated huntingtinIP3 receptor/Ca2+ channel: from discovery to new signaling concepts.Type 2 transglutaminase in Huntington's disease: a double-edged sword with clinical potential.Role of inositol 1,4,5-trisphosphate receptors in pathogenesis of Huntington's disease and spinocerebellar ataxiasComputational neurobiology is a useful tool in translational neurology: the example of ataxiaHuntington's Disease: From Mutant Huntingtin Protein to Neurotrophic Factor Therapy.Basal Ganglia disorders associated with imbalances in the striatal striosome and matrix compartments.Evaluating the SERCA2 and VEGF mRNAs as Potential Molecular Biomarkers of the Onset and Progression in Huntington's DiseaseDantrolene is neuroprotective in Huntington's disease transgenic mouse model.Pathological cell-cell interactions are necessary for striatal pathogenesis in a conditional mouse model of Huntington's diseaseDopaminergic signaling and striatal neurodegeneration in Huntington's disease.Ca(2+) handling in isolated brain mitochondria and cultured neurons derived from the YAC128 mouse model of Huntington's diseaseCalcium Handling by Endoplasmic Reticulum and Mitochondria in a Cell Model of Huntington's Disease.Alleviating neurodegeneration in Drosophila models of PolyQ diseases.Fast-to-Slow Transition of Skeletal Muscle Contractile Function and Corresponding Changes in Myosin Heavy and Light Chain Formation in the R6/2 Mouse Model of Huntington's Disease
P2860
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P2860
Deranged neuronal calcium signaling and Huntington disease.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Deranged neuronal calcium signaling and Huntington disease.
@ast
Deranged neuronal calcium signaling and Huntington disease.
@en
type
label
Deranged neuronal calcium signaling and Huntington disease.
@ast
Deranged neuronal calcium signaling and Huntington disease.
@en
prefLabel
Deranged neuronal calcium signaling and Huntington disease.
@ast
Deranged neuronal calcium signaling and Huntington disease.
@en
P1476
Deranged neuronal calcium signaling and Huntington disease
@en
P2093
Ilya Bezprozvanny
P304
P356
10.1016/J.BBRC.2004.08.035
P407
P577
2004-10-01T00:00:00Z