Varicella-zoster virus Fc receptor gE glycoprotein: serine/threonine and tyrosine phosphorylation of monomeric and dimeric forms
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Exocytosis of Varicella-Zoster Virus Virions Involves a Convergence of Endosomal and Autophagy PathwaysThe herpes virus Fc receptor gE-gI mediates antibody bipolar bridging to clear viral antigens from the cell surfaceIntracellular traffic of herpes simplex virus glycoprotein gE: characterization of the sorting signals required for its trans-Golgi network localization.Varicella-zoster virus Fc receptor component gI is phosphorylated on its endodomain by a cyclin-dependent kinaseMutation of the YXXL endocytosis motif in the cytoplasmic tail of pseudorabies virus gERole of envelope protein gE endocytosis in the pseudorabies virus life cycle.The extracellular domain of herpes simplex virus gE is sufficient for accumulation at cell junctions but not for cell-to-cell spread.Tyrosine phosphorylation of bovine herpesvirus 1 tegument protein VP22 correlates with the incorporation of VP22 into virions.Cytoplasmic domain of herpes simplex virus gE causes accumulation in the trans-Golgi network, a site of virus envelopment and sorting of virions to cell junctions.Cholesterol dependence of varicella-zoster virion entry into target cellsPhosphorylation by the varicella-zoster virus ORF47 protein serine kinase determines whether endocytosed viral gE traffics to the trans-Golgi network or recycles to the cell membrane.Molecular characterization and functions of fatty acid and retinoid binding protein gene (Ab-far-1) in Aphelenchoides besseyi.Mutagenesis of varicella-zoster virus glycoprotein I (gI) identifies a cysteine residue critical for gE/gI heterodimer formation, gI structure, and virulence in skin cells.Essential functions of the unique N-terminal region of the varicella-zoster virus glycoprotein E ectodomain in viral replication and in the pathogenesis of skin infectionAberrant infection and persistence of varicella-zoster virus in human dorsal root ganglia in vivo in the absence of glycoprotein I.Deletion of the first cysteine-rich region of the varicella-zoster virus glycoprotein E ectodomain abolishes the gE and gI interaction and differentially affects cell-cell spread and viral entry.Variable Effects of Autophagy Induction by Trehalose on Herpesviruses Depending on Conditions of Infection.Varicella-Zoster Virus Glycoproteins: Entry, Replication, and Pathogenesis.Bovine herpesvirus 5 glycoprotein E is important for neuroinvasiveness and neurovirulence in the olfactory pathway of the rabbit.Trafficking of varicella-zoster virus glycoprotein gI: T(338)-dependent retention in the trans-Golgi network, secretion, and mannose 6-phosphate-inhibitable uptake of the ectodomain.Essential role played by the C-terminal domain of glycoprotein I in envelopment of varicella-zoster virus in the trans-Golgi network: interactions of glycoproteins with tegument.Varicella-zoster virus ORF47 protein serine kinase: characterization of a cloned, biologically active phosphotransferase and two viral substrates, ORF62 and ORF63Complex formation facilitates endocytosis of the varicella-zoster virus gE:gI Fc receptor.A functional YNKI motif in the short cytoplasmic tail of varicella-zoster virus glycoprotein gH mediates clathrin-dependent and antibody-independent endocytosis.Endocytosis and recycling of varicella-zoster virus Fc receptor glycoprotein gE: internalization mediated by a YXXL motif in the cytoplasmic tail.Characterization of pseudorabies virus mutants expressing carboxy-terminal truncations of gE: evidence for envelope incorporation, virulence, and neurotropism domains.Antibody-induced and cytoskeleton-mediated redistribution and shedding of viral glycoproteins, expressed on pseudorabies virus-infected cells.Mutational analysis of the role of glycoprotein I in varicella-zoster virus replication and its effects on glycoprotein E conformation and trafficking.Quantitative measurement of varicella-zoster virus infection by semiautomated flow cytometry.Egress of light particles among filopodia on the surface of Varicella-Zoster virus-infected cells.The Fc Domain of Immunoglobulin Is Sufficient to Bridge NK Cells with Virally Infected Cells.Incorporation of three endocytosed varicella-zoster virus glycoproteins, gE, gH, and gB, into the virion envelopeGlycoprotein E of varicella-zoster virus enhances cell-cell contact in polarized epithelial cells.Pseudorabies virus triggers glycoprotein gE-mediated ERK1/2 activation and ERK1/2-dependent migratory behavior in T cells.Autophagy and the effects of its inhibition on varicella-zoster virus glycoprotein biosynthesis and infectivity.Molecular Basis for the Recognition of Herpes Simplex Virus Type 1 Infection by Human Natural Killer Cells.Expression of variable viruses as herpes simplex glycoprotein D and varicella zoster gE glycoprotein using a novel plasmid based expression system in insect cell
P2860
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P2860
Varicella-zoster virus Fc receptor gE glycoprotein: serine/threonine and tyrosine phosphorylation of monomeric and dimeric forms
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年学术文章
@wuu
1997年学术文章
@zh-cn
1997年学术文章
@zh-hans
1997年学术文章
@zh-my
1997年学术文章
@zh-sg
1997年學術文章
@yue
1997年學術文章
@zh
1997年學術文章
@zh-hant
name
Varicella-zoster virus Fc rece ...... of monomeric and dimeric forms
@ast
Varicella-zoster virus Fc rece ...... of monomeric and dimeric forms
@en
type
label
Varicella-zoster virus Fc rece ...... of monomeric and dimeric forms
@ast
Varicella-zoster virus Fc rece ...... of monomeric and dimeric forms
@en
prefLabel
Varicella-zoster virus Fc rece ...... of monomeric and dimeric forms
@ast
Varicella-zoster virus Fc rece ...... of monomeric and dimeric forms
@en
P2093
P2860
P1433
P1476
Varicella-zoster virus Fc rece ...... of monomeric and dimeric forms
@en
P2093
P2860
P304
P407
P577
1997-01-01T00:00:00Z