Comprehensive quantification of herpes simplex virus latency at the single-cell level
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The probability of in vivo reactivation of herpes simplex virus type 1 increases with the number of latently infected neurons in the ganglia.Bovine Herpes Virus 1 (BHV-1) and Herpes Simplex Virus Type 1 (HSV-1) Promote Survival of Latently Infected Sensory Neurons, in Part by Inhibiting ApoptosisA comparison of herpes simplex virus type 1 and varicella-zoster virus latency and reactivationThe molecular basis of herpes simplex virus latencyOcular herpes simplex virus: how are latency, reactivation, recurrent disease and therapy interrelated?The challenges and opportunities for the development of a T-cell epitope-based herpes simplex vaccineHerpes simplex virus and the lexicon of latency and reactivation: a call for defining terms and building an integrated collective frameworkLaser-capture microdissection: refining estimates of the quantity and distribution of latent herpes simplex virus 1 and varicella-zoster virus DNA in human trigeminal Ganglia at the single-cell level.Role of chromatin during herpesvirus infections.De novo synthesis of VP16 coordinates the exit from HSV latency in vivo.Role for gamma interferon in control of herpes simplex virus type 1 reactivation.Towards an understanding of the herpes simplex virus type 1 latency-reactivation cycleEpigenetic regulation of latent HSV-1 gene expression.The latent herpes simplex virus type 1 genome copy number in individual neurons is virus strain specific and correlates with reactivationReplication of herpes simplex virus type 1 within trigeminal ganglia is required for high frequency but not high viral genome copy number latency.Granzyme A, a noncytolytic component of CD8(+) cell granules, restricts the spread of herpes simplex virus in the peripheral nervous systems of experimentally infected mice.The latency-associated transcript gene enhances establishment of herpes simplex virus type 1 latency in rabbits.Optimized viral dose and transient immunosuppression enable herpes simplex virus ICP0-null mutants To establish wild-type levels of latency in vivo.Prevalence of varicella-zoster virus DNA in dissociated human trigeminal ganglion neurons and nonneuronal cellsQuantitation of latent varicella-zoster virus and herpes simplex virus genomes in human trigeminal ganglia.Detection of the genome and transcripts of a persistent DNA virus in neuronal tissues by fluorescent in situ hybridization combined with immunostaining.A historical analysis of herpes simplex virus promoter activation in vivo reveals distinct populations of latently infected neuronesLytic gene expression is frequent in HSV-1 latent infection and correlates with the engagement of a cell-intrinsic transcriptional responseHSV-1-based vectors for gene therapy of neurological diseases and brain tumors: part I. HSV-1 structure, replication and pathogenesis.Herpes simplex virus DNA synthesis is not a decisive regulatory event in the initiation of lytic viral protein expression in neurons in vivo during primary infection or reactivation from latency.Neither LAT nor open reading frame P mutations increase expression of spliced or intron-containing ICP0 transcripts in mouse ganglia latently infected with herpes simplex virusA mutation in the latency-related gene of bovine herpesvirus 1 disrupts the latency reactivation cycle in calves.Ostreid herpesvirus type 1 replication and host response in adult Pacific oysters, Crassostrea gigas.HSV-1 genome subnuclear positioning and associations with host-cell PML-NBs and centromeres regulate LAT locus transcription during latency in neurons.MicroRNAs expressed by herpes simplex virus 1 during latent infection regulate viral mRNAsOcular herpes simplex virus type 1: is the cornea a reservoir for viral latency or a fast pit stop?Tegument protein control of latent herpesvirus establishment and animation.PrP(c) expression influences the establishment of herpes simplex virus type 1 latency.Transcription of the herpes simplex virus latency-associated transcript promotes the formation of facultative heterochromatin on lytic promoters.Herpes simplex virus type 1 and bovine herpesvirus 1 latency.The checkpoints of viral gene expression in productive and latent infection: the role of the HDAC/CoREST/LSD1/REST repressor complexEstablishment and maintenance of gammaherpesvirus latency are independent of infective dose and route of infectionEstablishment of HSV1 latency in immunodeficient mice facilitates efficient in vivo reactivation.Of mice and not humans: how reliable are animal models for evaluation of herpes CD8(+)-T cell-epitopes-based immunotherapeutic vaccine candidates?Herpes simplex virus type 2 (HSV-2) establishes latent infection in a different population of ganglionic neurons than HSV-1: role of latency-associated transcripts.
P2860
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P2860
Comprehensive quantification of herpes simplex virus latency at the single-cell level
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Comprehensive quantification of herpes simplex virus latency at the single-cell level
@ast
Comprehensive quantification of herpes simplex virus latency at the single-cell level
@en
type
label
Comprehensive quantification of herpes simplex virus latency at the single-cell level
@ast
Comprehensive quantification of herpes simplex virus latency at the single-cell level
@en
prefLabel
Comprehensive quantification of herpes simplex virus latency at the single-cell level
@ast
Comprehensive quantification of herpes simplex virus latency at the single-cell level
@en
P2860
P1433
P1476
Comprehensive quantification of herpes simplex virus latency at the single-cell level
@en
P2093
N M Sawtell
P2860
P304
P407
P577
1997-07-01T00:00:00Z