Revisiting the ionic mechanisms of early afterdepolarizations in cardiomyocytes: predominant by Ca waves or Ca currents?
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Mechanisms of ventricular arrhythmias: from molecular fluctuations to electrical turbulenceA Comparative Study of Early Afterdepolarization-Mediated Fibrillation in Two Mathematical Models for Human Ventricular CellsPerspective: a dynamics-based classification of ventricular arrhythmiasNonlinear and Stochastic Dynamics in the HeartMechanisms and determinants of ultralong action potential duration and slow rate-dependence in cardiac myocytes.Nonequilibrium reactivation of Na+ current drives early afterdepolarizations in mouse ventricleOverexpression of adenylyl cyclase type 5 (AC5) confers a proarrhythmic substrate to the heartRole of sarcoplasmic reticulum calcium in development of secondary calcium rise and early afterdepolarizations in long QT syndrome rabbit model.Hyperphosphorylation of RyRs underlies triggered activity in transgenic rabbit model of LQT2 syndrome.Calcium-voltage coupling in the genesis of early and delayed afterdepolarizations in cardiac myocytesMechanistic Investigation of the Arrhythmogenic Role of Oxidized CaMKII in the HeartBeta-Adrenoceptor Stimulation Reveals Ca2+ Waves and Sarcoplasmic Reticulum Ca2+ Depletion in Left Ventricular Cardiomyocytes from Post-Infarction Rats with and without Heart Failure.Criticality in intracellular calcium signaling in cardiac myocytesRole of the transient outward potassium current in the genesis of early afterdepolarizations in cardiac cells.Synchronization of early afterdepolarizations and arrhythmogenesis in heterogeneous cardiac tissue models.Docosahexaenoic Acid reduces the incidence of early afterdepolarizations caused by oxidative stress in rabbit ventricular myocytes.Prolonged Action Potential and After depolarizations Are Not due to Changes in Potassium Currents in NOS3 Knockout Ventricular Myocytes.Phospholamban Ablation Using CRISPR/Cas9 System Improves Mortality in a Murine Heart Failure Model.The link between abnormal calcium handling and electrical instability in acquired long QT syndrome--Does calcium precipitate arrhythmic storms?Early afterdepolarizations in cardiac myocytes: beyond reduced repolarization reserveImaging ROS signaling in cells and animals.Dynamics of the late Na(+) current during cardiac action potential and its contribution to afterdepolarizations.SERCA2a gene therapy in heart failure: an anti-arrhythmic positive inotrope.Dependence of phase-2 reentry and repolarization dispersion on epicardial and transmural ionic heterogeneity: a simulation study.Electrotonic suppression of early afterdepolarizations in the neonatal rat ventricular myocyte monolayer.The electromechanical window is no better than QT prolongation to assess risk of Torsade de Pointes in the complete atrioventricular block model in dogs.Modeling CaMKII-mediated regulation of L-type Ca(2+) channels and ryanodine receptors in the heart.Electrical storm: recent pathophysiological insights and therapeutic consequences.Do t-tubules play a role in arrhythmogenesis in cardiac ventricular myocytes?The promise of CaMKII inhibition for heart disease: preventing heart failure and arrhythmias.New antiarrhythmic targets to control intracellular calcium handling.Role of abnormal repolarization in the mechanism of cardiac arrhythmia.The Effects of SEA0400 on Ca2+ Transient Amplitude and Proarrhythmia Depend on the Na+/Ca2+ Exchanger Expression Level in Murine Models.Atrial-selective targeting of arrhythmogenic phase-3 early afterdepolarizations in human myocytesOxidative stress, fibrosis, and early afterdepolarization-mediated cardiac arrhythmias.Spontaneous initiation of premature ventricular complexes and arrhythmias in type 2 long QT syndrome.A model of cardiac ryanodine receptor gating predicts experimental Ca2+-dynamics and Ca2+-triggered arrhythmia in the long QT syndrome.Role of Apamin-Sensitive Calcium-Activated Small-Conductance Potassium Currents on the Mechanisms of Ventricular Fibrillation in Pacing-Induced Failing Rabbit Hearts.Mechanisms linking T-wave alternans to spontaneous initiation of ventricular arrhythmias in rabbit models of long QT syndrome.Dynamical mechanisms of phase-2 early afterdepolarizations in human ventricular myocytes: insights from bifurcation analyses of two mathematical models.
P2860
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P2860
Revisiting the ionic mechanisms of early afterdepolarizations in cardiomyocytes: predominant by Ca waves or Ca currents?
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Revisiting the ionic mechanism ...... nt by Ca waves or Ca currents?
@ast
Revisiting the ionic mechanism ...... nt by Ca waves or Ca currents?
@en
type
label
Revisiting the ionic mechanism ...... nt by Ca waves or Ca currents?
@ast
Revisiting the ionic mechanism ...... nt by Ca waves or Ca currents?
@en
prefLabel
Revisiting the ionic mechanism ...... nt by Ca waves or Ca currents?
@ast
Revisiting the ionic mechanism ...... nt by Ca waves or Ca currents?
@en
P2093
P2860
P1476
Revisiting the ionic mechanism ...... nt by Ca waves or Ca currents?
@en
P2093
Akemichi Baba
Charelle Allen
Hairuo Wen
Lai-Hua Xie
Nadezhda Fefelova
Toshio Matsuda
Zhenghang Zhao
P2860
P304
P356
10.1152/AJPHEART.00742.2011
P577
2012-02-03T00:00:00Z