Established cell lines used in cystic fibrosis research.
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Airway Gland Structure and FunctionA multi-omics approach identifies key hubs associated with cell type-specific responses of airway epithelial cells to staphylococcal alpha-toxin.Barriers to inhaled gene therapy of obstructive lung diseases: A reviewDefective CFTR increases synthesis and mass of sphingolipids that modulate membrane composition and lipid signaling.Staphylococcus aureus alpha-toxin mediates general and cell type-specific changes in metabolite concentrations of immortalized human airway epithelial cells.Aspergillus fumigatus forms biofilms with reduced antifungal drug susceptibility on bronchial epithelial cells.TLR-4-mediated innate immunity is reduced in cystic fibrosis airway cells.Toll-like receptor 4 is not targeted to the lysosome in cystic fibrosis airway epithelial cells.Correction of defective CFTR/ENaC function and tightness of cystic fibrosis airway epithelium by amniotic mesenchymal stromal (stem) cells.Potential of anti-inflammatory treatment for cystic fibrosis lung disease.Nanoparticles that deliver triplex-forming peptide nucleic acid molecules correct F508del CFTR in airway epitheliumRhinovirus Load Is High despite Preserved Interferon-β Response in Cystic Fibrosis Bronchial Epithelial Cells.Role of Interaction and Nucleoside Diphosphate Kinase B in Regulation of the Cystic Fibrosis Transmembrane Conductance Regulator Function by cAMP-Dependent Protein Kinase A.Junctional abnormalities in human airway epithelial cells expressing F508del CFTR.Targeted Integration of a Super-Exon into the CFTR Locus Leads to Functional Correction of a Cystic Fibrosis Cell Line Model.Caspases shutdown nonsense-mediated mRNA decay during apoptosis.Transcomplementation by a truncation mutant of cystic fibrosis transmembrane conductance regulator (CFTR) enhances ΔF508 processing through a biomolecular interactionSurface modified poly(β amino ester)-containing nanoparticles for plasmid DNA delivery.Proteomic biomarker discovery for the monogenic disease cystic fibrosis.New use for an old drug: COX-independent anti-inflammatory effects of sulindac in models of cystic fibrosis.Role of oxygen availability in CFTR expression and function.Novel human bronchial epithelial cell lines for cystic fibrosis research.TALENs Facilitate Single-step Seamless SDF Correction of F508del CFTR in Airway Epithelial Submucosal Gland Cell-derived CF-iPSCs.Generation of SV40-transformed rabbit tracheal-epithelial-cell-derived blastocyst by somatic cell nuclear transfer.Pseudomonas aeruginosa infection of airway epithelial cells modulates expression of Kruppel-like factors 2 and 6 via RsmA-mediated regulation of type III exoenzymes S and Y.Cystic Fibrosis from Laboratory to Bedside: The Role of A20 in NF-κB-Mediated Inflammation.Oxidative stress modulates the expression of genes involved in cell survival in ΔF508 cystic fibrosis airway epithelial cells.Personalized medicine for cystic fibrosis: establishing human model systems.In Vitro Models to Study Human Lung Development, Disease and Homeostasis.Immortalized bovine mammary epithelial cells express stem cell markers and differentiate in vitro.Cystic fibrosis bronchial epithelial cells have impaired ability to activate vitamin D.The tyrosine kinase BceF and the phosphotyrosine phosphatase BceD of Burkholderia contaminans are required for efficient invasion and epithelial disruption of a cystic fibrosis lung epithelial cell lineStaphylococcus aureus hemolysin A disrupts cell-matrix adhesions in human airway epithelial cells.S. aureus haemolysin A-induced IL-8 and IL-6 release from human airway epithelial cells is mediated by activation of p38- and Erk-MAP kinases and additional, cell type-specific signalling mechanisms.Sensitivity of chloride efflux vs. transepithelial measurements in mixed CF and normal airway epithelial cell populations.Comparative transfection of DNA into primary and transformed mammalian cells from different lineages.Efficacy and toxicity of the antimicrobial peptide M33 produced with different counter-ions.Activation of MMP-9 by human lung epithelial cells in response to the cystic fibrosis-associated pathogen Burkholderia cenocepacia reduced wound healing in vitro.Down-regulation of cytokine-induced interleukin-8 requires inhibition of p38 mitogen-activated protein kinase (MAPK) via MAPK phosphatase 1-dependent and -independent mechanisms.A disease-relevant high-content screening assay to identify anti-inflammatory compounds for use in cystic fibrosis.
P2860
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P1343
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P2860
Established cell lines used in cystic fibrosis research.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Established cell lines used in cystic fibrosis research.
@ast
Established cell lines used in cystic fibrosis research.
@en
type
label
Established cell lines used in cystic fibrosis research.
@ast
Established cell lines used in cystic fibrosis research.
@en
prefLabel
Established cell lines used in cystic fibrosis research.
@ast
Established cell lines used in cystic fibrosis research.
@en
P2093
P4510
P1476
Established cell lines used in cystic fibrosis research.
@en
P2093
Cassiman JJ
Frizzell RA
Gruenert DC
P304
P356
10.1016/J.JCF.2004.05.040
P478
P577
2004-08-01T00:00:00Z