New therapies for muscular dystrophy: cautious optimism.
about
Injectable polyethylene glycol-fibrinogen hydrogel adjuvant improves survival and differentiation of transplanted mesoangioblasts in acute and chronic skeletal-muscle degeneration.Utrophin up-regulation by an artificial transcription factor in transgenic miceTerm Amniotic membrane is a high throughput source for multipotent Mesenchymal Stem Cells with the ability to differentiate into endothelial cells in vitro.Corticortophin releasing factor 2 receptor agonist treatment significantly slows disease progression in mdx miceMagic-factor 1, a partial agonist of Met, induces muscle hypertrophy by protecting myogenic progenitors from apoptosis.Repairing skeletal muscle: regenerative potential of skeletal muscle stem cells.Co-administration of ibuprofen and nitric oxide is an effective experimental therapy for muscular dystrophy, with immediate applicability to humans.Bone marrow mesenchymal stromal cells stimulate skeletal myoblast proliferation through the paracrine release of VEGFInterleukin-15 administration improves diaphragm muscle pathology and function in dystrophic mdx mice.Nitric oxide release combined with nonsteroidal antiinflammatory activity prevents muscular dystrophy pathology and enhances stem cell therapy.The potential of stem cells in the treatment of skeletal muscle injury and diseaseThe role of myostatin and bone morphogenetic proteins in muscular disorders.Stressed out: the skeletal muscle ryanodine receptor as a target of stressNitric oxide: emerging concepts about its use in cell-based therapies.Skeletal muscle as a paradigm for regenerative biology and medicine.Trophic actions of bone marrow-derived mesenchymal stromal cells for muscle repair/regeneration.Fusion of bone marrow-derived stem cells with striated muscle may not be sufficient to activate muscle genesMesoangioblasts of inclusion-body myositis: a twofold tool to study pathogenic mechanisms and enhance defective muscle regenerationGlobular adiponectin as a complete mesoangioblast regulator: role in proliferation, survival, motility, and skeletal muscle differentiation.Stem cells from umbilical cord blood do have myogenic potential, with and without differentiation induction in vitroSources for skeletal muscle repair: from satellite cells to reprogrammingAdaptive Immune Response Impairs the Efficacy of Autologous Transplantation of Engineered Stem Cells in Dystrophic Dogs.Human mesenchymal stem cells ectopically expressing full-length dystrophin can complement Duchenne muscular dystrophy myotubes by cell fusion.Stem cells from umbilical cord blood differentiate into myotubes and express dystrophin in vitro only after exposure to in vivo muscle environment.Transplantation of human skeletal muscle-derived progenitor cells ameliorates knee osteoarthritis in streptozotocin-induced diabetic mice.
P2860
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P2860
New therapies for muscular dystrophy: cautious optimism.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
New therapies for muscular dystrophy: cautious optimism.
@ast
New therapies for muscular dystrophy: cautious optimism.
@en
type
label
New therapies for muscular dystrophy: cautious optimism.
@ast
New therapies for muscular dystrophy: cautious optimism.
@en
prefLabel
New therapies for muscular dystrophy: cautious optimism.
@ast
New therapies for muscular dystrophy: cautious optimism.
@en
P1476
New therapies for muscular dystrophy: cautious optimism.
@en
P2093
Giulio Cossu
Maurilio Sampaolesi
P304
P356
10.1016/J.MOLMED.2004.08.007
P577
2004-10-01T00:00:00Z