A coding variant in CR1 interacts with APOE-ε4 to influence cognitive decline. - Wikidata - wikimedia - TriplyDB

A coding variant in CR1 interacts with APOE-ε4 to influence cognitive decline.

A coding variant in CR1 interacts with APOE-ε4 to influence cognitive decline.

http://www.wikidata.org/entity/Q35909452

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Genetics ignite focus on microglial inflammation in Alzheimer's disease The complotype: dictating risk for inflammation and infection The Alzheimer's Disease Neuroimaging Initiative: a review of papers published since its inception Genetic analysis of quantitative phenotypes in AD and MCI: imaging, cognition and biomarkers GWAS of cerebrospinal fluid tau levels identifies risk variants for Alzheimer's disease.2014 Update of the Alzheimer's Disease Neuroimaging Initiative: A review of papers published since its inception.The PSEN1, p.E318G variant increases the risk of Alzheimer's disease in APOE-ε4 carriers Geographical distribution of complement receptor type 1 variants and their associated disease risk.Building a pipeline to discover and validate novel therapeutic targets and lead compounds for Alzheimer's disease.Apolipoprotein E4 produced in GABAergic interneurons causes learning and memory deficits in mice Late-onset Alzheimer's risk variants in memory decline, incident mild cognitive impairment, and Alzheimer's disease A TREM1 variant alters the accumulation of Alzheimer-related amyloid pathology.Alzheimer's loci: epigenetic associations and interaction with genetic factors.Analysis of the Putative Role of CR1 in Alzheimer's Disease: Genetic Association, Expression and Function.Late-onset Alzheimer disease risk variants mark brain regulatory loci An inflection point in gene discovery efforts for neurodegenerative diseases: from syndromic diagnoses toward endophenotypes and the epigenome.Using an Alzheimer Disease Polygenic Risk Score to Predict Memory Decline in Black and White Americans Over 14 Years of Follow-up.Interaction between two cholesterol metabolism genes influences memory: findings from the Wisconsin Registry for Alzheimer's Prevention Genetic susceptibility for Alzheimer disease neuritic plaque pathology.Where genotype is not predictive of phenotype: towards an understanding of the molecular basis of reduced penetrance in human inherited disease.Pathways to neurodegeneration: mechanistic insights from GWAS in Alzheimer's disease, Parkinson's disease, and related disorders Evaluation of memory endophenotypes for association with CLU, CR1, and PICALM variants in black and white subjects.Alternative Approaches in Gene Discovery and Characterization in Alzheimer's Disease.CR1 in Alzheimer's disease.Alzheimer's disease genetics: from the bench to the clinic.Late Onset Alzheimer's Disease Risk Variants in Cognitive Decline: The PATH Through Life Study.The Genetic Variability of UCP4 Affects the Individual Susceptibility to Late-Onset Alzheimer's Disease and Modifies the Disease's Risk in APOE-ɛ4 Carriers.Association of Alzheimer's related genotypes with cognitive decline in multiple domains: results from the Three-City Dijon study.De-regulation of gene expression and alternative splicing affects distinct cellular pathways in the aging hippocampus.A Dopamine Receptor genetic variant enhances perceptual speed in cognitive healthy subjects Effect of CR1 Genetic Variants on Cerebrospinal Fluid and Neuroimaging Biomarkers in Healthy, Mild Cognitive Impairment and Alzheimer's Disease Cohorts.Peripheral complement interactions with amyloid β peptide in Alzheimer's disease: 2. Relationship to Aβ immunotherapy.Potential contribution of the Alzheimer's disease risk locus BIN1 to episodic memory performance in cognitively normal Type 2 diabetes elderly.Genetic Biomarkers on Age-Related Cognitive Decline.Alzheimer disease: the quest for Alzheimer disease genes--focus on CSF tau.Association of GWAS Top Genes With Late-Onset Alzheimer's Disease in Colombian Population.Complement receptor 1 gene (CR1) intragenic duplication and risk of Alzheimer's disease.

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P2860

A coding variant in CR1 interacts with APOE-ε4 to influence cognitive decline.

http://www.wikidata.org/entity/Q35909452

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

A coding variant in CR1 interacts with APOE-ε4 to influence cognitive decline.

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A coding variant in CR1 interacts with APOE-ε4 to influence cognitive decline.

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type

label

A coding variant in CR1 interacts with APOE-ε4 to influence cognitive decline.

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A coding variant in CR1 interacts with APOE-ε4 to influence cognitive decline.

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prefLabel

A coding variant in CR1 interacts with APOE-ε4 to influence cognitive decline.

@ast

A coding variant in CR1 interacts with APOE-ε4 to influence cognitive decline.

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Human Molecular Genetics

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A coding variant in CR1 interacts with APOE-ε4 to influence cognitive decline.

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Amanda J Myers

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Anne Nicholson-Weller

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April N Allen

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Brendan T Keenan

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Cynthia A Lemere

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Denis A Evans

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Dong Tran

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Jason J Corneveaux

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Joshua M Shulman

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Lori B Chibnik

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P2860

Alzheimer's Disease

Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease

A map of human genome variation from population-scale sequencing

Clinical diagnosis of Alzheimer's disease: report of the NINCDS-ADRDA Work Group under the auspices of Department of Health and Human Services Task Force on Alzheimer's Disease

Principal components analysis corrects for stratification in genome-wide association studies

Common variants at MS4A4/MS4A6E, CD2AP, CD33 and EPHA1 are associated with late-onset Alzheimer's disease.

METAL: fast and efficient meta-analysis of genomewide association scans

Common variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer's disease

Genome-wide association study identifies variants at CLU and CR1 associated with Alzheimer's disease

C1q and C4b bind simultaneously to CR1 and additively support erythrocyte adhesion.

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2377-2388

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10.1093/HMG/DDS054

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10

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21

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Alzheimer's Disease Neuroimaging Initiative

Mert R. Sabuncu

John Anthony Hardy

David A Bennett

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2012-02-17T00:00:00Z

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221840965

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22343410

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